• Journal of the Korea Institute of Military Science and Technology
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• v.24 no.6
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• pp.655-663
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• 2021

In this paper, DFT-Based channel estimation with channel response mirroring is proposed and analyzed. In General, pilot symbols for channel estimation in MIMO(Multi-Input Multi-Output) OFDM(Orthogonal Frequency-Division Multiplexing) Systems have a diamond shape in the time-frequency plane. An interpolation technique to estimate the channel response of sub-carriers between reference symbols is needed. Various interpolation techniques such as linear interpolation, low-pass filtering interpolation, cubic interpolation and DFT interpolation are employed to estimate the non-pilot sub-carriers. In this paper, we investigate the conventional DFT-based channel estimation for noise reduction and channel response interpolation. The conventional method has performance degradation by distortion called "edge effect" or "border effect". In order to mitigate the distortion, we propose an improved DFT-based channel estimation with channel response mirroring. This technique can efficiently mitigate the distortion caused by the DFT of channel response discontinuity. Simulation results show that the proposed method has better performance than the conventional DFT-based channel estimation in terms of MSE.

• Journal of the Institute of Electronics Engineers of Korea SD
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• v.39 no.10
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• pp.1-8
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• 2002

C- V method is a means to determine the effective channel length for miniaturized MOSFET's. This method achieves L$_{eff}$ by extracting a unique channel length independent extrinsic overlap length($\Delta$L) at a critical gate bias point. In this paper, we conducted an experiment on two different C-V methods. L$_{eff}$ extracted from experiment is compared with L$_{eff}$ simulated from a two-dimensional (2-D) device simulator, and the accuracy of C-V method for L$_{eff}$ extraction is analyzed.

• Journal of Korea Multimedia Society
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• v.14 no.8
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• pp.965-980
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• 2011

In this paper, the use of conditional Fuzzy C-Means (CFCM) aimed at estimation of desired states of an unknown digital communication channel is investigated for blind channel equalization. In the proposed CFCM, a collection of clustered centers is treated as a set of pre-defined desired channel states, and used to extract channel output states. By considering the combinations of the extracted channel output states, all possible sets of desired channel states are constructed. The set of desired states characterized by the maximal value of the Bayesian fitness function is subsequently selected for the next fuzzy clustering epoch. This modification of CFCM makes it possible to search for the optimal desired channel states of an unknown channel. Finally, given the desired channel states, the Bayesian equalizer is implemented to reconstruct transmitted symbols. In a series of simulations, binary signals are generated at random with Gaussian noise, and both linear and nonlinear channels are evaluated. The experimental studies demonstrate that the performance (being expressed in terms of accuracy and speed) of the proposed CFCM is superior to the performance of the existing method exploiting the "conventional" Fuzzy C-Means (FCM).

• The Korean Journal of Physiology and Pharmacology
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• v.4 no.6
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• pp.445-453
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• 2000

The present investigation tested the hypothesis that the activation of protein kinase G (PKG) leads to a phosphorylation of $Ca^{2+}-activated$ potassium channel $(K_{Ca}\;channel)$ and is involved in the activation of $K_{Ca}$ channel activity in cerebral arterial smooth muscle cells of the rabbit. Single-channel currents were recorded in cell-attached and inside-out patch configurations of patch-clamp techniques. Both molsidomine derivative 3-morpholinosydnonimine-N-ethylcarbamide $(SIN-1,\;50\;{\mu}M)$ and 8-(4-Chlorophenylthio)-guanosine-3',5'-cyclic monophosphate $(8-pCPT-cGMP,\;100\;{\mu}M),$ a membrane-permeable analogue of cGMP, increased the $K_{Ca}$ channel activity in the cell-attached patch configuration, and the effect was removed upon washout of the drugs. In inside-out patches, single-channel current amplitude was not changed by SIN-1 and 8-pCPT-cGMP. Application of ATP $(100\;{\mu}M),$ cGMP $(100\;{\mu}M),$ ATP+cGMP $(100\;{\mu}M\;each),$ PKG $(5\;U/{\mu}l),$ ATP $(100\;{\mu}M)+PKG\;(5\;U/{\mu}l),$ or cGMP $(100\;{\mu}M)+PKG\;(5\;U/{\mu}l)$ did not increase the channel activity. ATP $(100\;{\mu}M)+cGMP\;(100\;{\mu}M)+PKG\;(5\;U/{\mu}l)$ added directly to the intracellular phase of inside-out patches increased the channel activity with no changes in the conductance. The heat-inactivated PKG had no effect on the channel activity, and the effect of PKG was inhibited by 8-(4-Chlorophenylthio)-guanosine-3',5'-cyclic monophosphate, Rp-isomer $(Rp-pCPT-cGMP,\;100\;{\mu}M),$ a potent inhibitor of PKG or protein phosphatase 2A (PP2A, 1 U/ml). In the presence of okadaic acid (OA, 5 nM), PP2A had no effect on the channel activity. The $K_{Ca}$ channel activity spontaneously decayed to the control level upon washout of ATP, cGMP and PKG, and this was prevented by OA (5 nM) in the medium. These results suggest that the PKG-mediated phosphorylations of $K_{Ca}$ channels, or some associated proteins in the membrane patch increase the activity of the $K_{Ca}$ channel, and the activation may be associated with the vasodilating action.

• Journal of the Institute of Electronics Engineers of Korea SD
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• v.47 no.10
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• pp.1-6
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• 2010

In this paper, RF Capacitance-Voltage(C-V) curve of short-channel MOSFET has been extracted from the room temperature to $225^{\circ}C$ using a RF method based on measured S-parameter data, and its high-temperature dependent characteristics are empirically modeled. It is observed that the voltage shift according to the variation of temperature in the weak inversion region of RF C-V curves is lower than the threshold voltage shift, but it is confirmed that this phenomenon is unexplainable with a long-channel theoretical C-V equation. The new empirical equation is developed for high-temperature dependent modeling of short-channel MOSFET C-V curves. The accuracy of this equation is demonstrated by observing good agreements between the modeled and measured C-V data in the wide range of temperature. It is also confirmed that the channel capacitance decreases with increasing temperature at high gate voltage.

• 전기의세계
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• v.23 no.2
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• pp.55-61
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• 1974

Nature of magnetic creep phenomena in low coercive force films(Ni 80%-Fe 20%) in form of narrow channels imbedded in high coercive force films is studied in this work. Aluminium is evaporated on the hot glass substrate and eched free in the shape of narrow channels by photoetoetching method. then, Permalloy(Ni 80%, Fe 20%) is deposited on these Aluminium substrate under the uniform field of 30(Oe) to introduce anisotropy. Permalloy film on Al has a high coercive force and one on the substrate devoid of Al has how coercive force. Magnetic revers domain which is introduced at the end of channel grows under the a.c field in hard axis direction, in spite of very weak d.c field in easy axis direction. This creeping is investigated as a function of external fields and channel widths. Permalloy film thickness is 500.angs.-900.angs. and channel widths are 40, 51, 65, 81, 115.mu. respectively. Creeping increases as external field increases while it decreases with channel width decrease. Creep velocity in channels depends on the a.c field along hard axis, d.c field along easy axis and channel widths and its range is 1-10cm/sec in this experiment. From study of dependence of creep velocity on channel width, it can be concluded that creep velocity is expressed in form of v=v$_{0}$ exp .alpha.(H-H$_{0}$) where .alpha. is a function of a.c field along hard axis and H is driving d.c field along easy axis, H$_{0}$ is not a coercive force of film as usuall expected but the d.c threshold field along easy axis which is a function of channel width. This characteristic is also confirmed by the study of dependence of creep velocity upon easy axis field strength. Value of .alpha. obtained is 1.3-2.3cm/sec We depending upon film charactor, hard axis field strength and frequency.uency.

• YAKHAK HOEJI
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• v.45 no.3
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• pp.282-286
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• 2001

To investigate the different mechanism between ATP and compound 48/80 (C$_{48}$80/)-induced histamine release, we observed effects of calcium antagonists in histamine release of rat peritoneal mast cells. Verapamil and diltiazem (voltage-dependent calcium channel blocker) and TMB-8 (a blocker of intracellular calcium release) significantly inhibited ATP-induced histamine release, but did not inhibit $C_{48}$80/-induced histamine release. Econazole (a blocker of receptor-operated calcium channel) dose-dependently inhibited both ATP and $C_{48}$80/-induced histamine release, but inhibitory effect of econazole in ATP-induced histamine release was more potent than that in $C_{48}$80/-induced histamine. EGTA dose-dependently inhibited ATP and $C_{48}$80/-induced histamine release, but $C_{48}$80/-induced histamine release was slightly inhibited by high concentrations (>2 mM) of EGTA. These results suggest that ATP-induced histamine release is related to broth intracellular calcium release and extracellular calcium influx via voltage-dependent calcium channel and receptor-operated calcium channel. $C_{48}$80/-induced histamine release is related to extracellular calcium influx, especially by receptor-operated calcium channel rather than voltage-dependent calcium channel.

• Biomolecules & Therapeutics
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• v.14 no.2
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• pp.119-124
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• 2006

This study was performed to investigate the influence of the spinal adenosine $A_1$ receptors on the central regulation of blood pressure (BP) and heart rate (HR), and to define whether its mechanism is mediated by cyclic AMP (cAMP), cyclic GMP (cGMP) or potassium channel. Intrathecal (i.t.) administration of drugs at the thoracic level were performed in anesthetized, artificially ventilated male Sprague-Dawley rats. I.t. injection of adenosine $A_1$ receptor agonist, $N^6$-cyclohexyladenosine (CHA; 1, 5 and 10 nmol) produced dose dependent decrease of BP and HR and it was attenuated by pretreatment of 50 nmol of 8-cyclopentyl-1,3-dimethylxanthine, a specific adenosine $A_1$ receptor antagonist. Pretreatment with a cAMP analogue, 8-bromo-cAMP, also attenuated the depressor and bradycardiac effects of CHA (10 nmol), but not with cGMP analogue, 8-bromo-cGMP. Pretreatment with a ATP-sensitive potassium channel blocker, glipizide (20 nmol) also attenuated the depressor and bradycardiac effects of CHA (10 nmol). These results suggest that adenosine $A_1$ receptor in the spinal cord plays an inhibitory role in the central cardiovascular regulation and that this depressor and bradycardiac actions are mediated by cAMP and potassium channel.

• The Journal of Korean Institute of Communications and Information Sciences
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• v.33 no.2A
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• pp.174-182
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• 2008

For the UHF EPC C1 Gen2 RFID system, to overcome the performance degradation in wireless channel environments, we proposed a modified tag structure which supports channel coding and its applied methodology. The reader consistently monitors the wireless channel status and sets to use channel coding functions for tags and reader only if wireless channel is corrupted. Compared with previous tag complexity, only negligible gates are needed to implement channel coding. By simulation we showed an obvious performance improvement of the identification reliability.

• The Journal of the Korea institute of electronic communication sciences
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• v.14 no.5
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• pp.837-844
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• 2019

In this paper, we propose the AMBM(: Adaptive Multi-channel Backoff Machisum) -Mac protocol to provide high throughput for non-safety applications in VANET(: Vehicular Ad Hoc Networks) environment. The proposed protocol guarantees the quality of service of non-safety packets by dynamically adjusting CW(: Channel Window) of WSA(: WAVE Service Advertisement) to maximize throughput between non-safety packets of different priority. It also shows that allocating a large amount of time for channel coordination and time slot reservation for SC and dynamically adjusting CW and CCI as nodes increase to reduces transmission delay than IEEE 1609.9, C-MAC(: Coordinated multi-channel MAC, and Q-VCI(: QoS Variable CCH Interval) protocols.