• Title/Summary/Keyword: pulmonary edema

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PULMONARY EDEMA INDUCED BY BOSMINE SOAKED GAUZE IN ORTHOGNATHIC SURGER (악교정 수술시 보스민 거즈에 의해 나타난 폐부종 증례보고)

  • Kim, Jae-Gyung;Kim, Yong-Kwan;You, Jun-Young;Joo, Jin-Churl;Lee, Chang-Sun
    • Maxillofacial Plastic and Reconstructive Surgery
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    • v.20 no.2
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    • pp.148-151
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    • 1998
  • There are many vessels in oral and maxillofacial region. Therefore blood oozing is a frequent findings in this area when operation takes place. Beacuse of this, most oral and maxillofacial surgeons as well as operators of this region usually use bosmine soaked gauze for oozing control during operation. Theoretically overdose of systemic epiniephrine(bosmine) may lead to pulmonary edema due to secondary systemic volume overload. For this reason, oral and maxillofacial surgeons should give more attention to the use of bosmine soaked gauze. In spite of this possibility, epinephrine induced pulmonary edema using bosmine soaked gauze has not been reported. We experienced one case of pulmonary edema which was induced by epinephrine overdose by bosmine soaked gauze that has not been diluted by mistake in preparing bosmine soaked gauze. The authuors report a case with review of literature.

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A 45-Year-Old Man With Recurrent Dyspnea and Hemoptysis during Exercise: Exercise-Induced Pulmonary Hemorrhage/Edema

  • Kim, Dae Sung;Lee, Minhyeok;Kwon, Oh Jung;Jeong, Inbeom;Son, Ji Woong;Na, Moon Jun;Kwon, Sun Jung
    • Tuberculosis and Respiratory Diseases
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    • v.78 no.4
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    • pp.375-379
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    • 2015
  • A 45-year-old man presented with dyspnea and hemoptysis during exercise. A chest computed tomography (CT) revealed multifocal diffuse patchy ground glass opacity and interlobular septal thickening in both the lungs. Permeability pulmonary edema or pulmonary hemorrhage was suspected. Serologic studies for autoimmune disorders and vasculitis were negative. There was no laboratory evidence of coagulopathy, other hematopoietic disease or infectious disease. Considering correlation with exercise, we diagnosed exercise-induced pulmonary hemorrhage (EIPH) or exercise-induced pulmonary edema (EIPE). The patient was managed with antifibrinolytics, antibiotics, and antitussive agent. After a week, follow-up chest CT revealed completely resolved pulmonary hemorrhage. About 2 months after the first event, he visited again with dyspnea and hemoptysis during running. In the present study, we report a case of recurrent pulmonary hemorrhage after exercise.

Effects of Pyengpaetang Extracts on the Acute Pulmonary Edema induced by Oleic acid in dogs (평폐탕(平肺湯)이 Oleic acid로 유발(誘發)된 가견(家犬)의 급성폐수종(急性肺水腫)에 미치는 영향(影響))

  • Chung, Jae-Woo;Han, Sang-Whan;Choe, Sun-Ho
    • The Journal of Internal Korean Medicine
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    • v.11 no.2
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    • pp.1-15
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    • 1990
  • Certain oriental medication have been shown to be effective in decreasing pulmonary vascular resistance and increasing cardiac output in primary pulmonary artery pressure secondary to pulmonary edema. So oleic acid was administered in 14 dogs in order to induce acute lung injury. And we studied the hemodynamics and blood gas changes of Pyengpaetang(50mg, 100mg) with continuous postive pressure was ventilation in pulmonary edema. The pulmonary edema group, arterial oxygenation was improved after 5 and $10cmH_2O$ PEEP(positive end expiratory pressure), but cardiovascular system was depressed. Blood pressure and cardiac output were decreased, and CVP, MP AP, PCWP were increased. In Pyengpaetang(50mg) group, mean aortic pressure was decreased and PCWP(pulmonary capillary wedge pressure) was decreased remarkably, while there was a significant increase in cardiac output. And there was improvement in $PaO_2$ and $PaCO_2$ without hemodynamic changes after applying 5cm $H_2O$ PEEP, but arterial blood gases$(PaO_2,\;PaCO_2)$ were improved, while cardiovascular effects were depressed after cm $H_2O$ PEEP. In Pyengpaetang(100mg) treated group, there was no significant hemodynamic change. But mean pulmonary arterial pressure was significantly increased, and cardiac output was decreased significantly after applying the more degree of PEEP. And blood gases were not changed significantly after applying the more degree of PEEP. The above results suggest that the effects of Pyengpaetang(50mg) group is superior to those of Pyengpaetang(100mg) group on the effects of hemodynamics and gas exchanges in acute lung injury in dogs. So we can conclude that lower degree PEEP 5cm $H_2O$ is more beneficial in Pyengpaetang(50mg) treated group.

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Bronchial Hyperresponsiveness in Chronic Renal Failure Undergoing Hemodialysis (만성 신부전 환자에서 혈액투석 전후의 기관지 과민반응검사)

  • Hwang, Young-Sil;Shim, Dae-Suk
    • Tuberculosis and Respiratory Diseases
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    • v.42 no.4
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    • pp.548-554
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    • 1995
  • Background: Cardiogenic pulmonary edema increases nonspecific airway responsiveness in humans and animals. Increased extravascular lung water from overt pulmonary edema to subclinical interstitial edema is a common finding in patients with chronic renal failure. Several studies carried out to assess pulmonary function disturbances in this condition have documented a reduction in forced expiratory volume that usually reverses after hemodialysis, suggesting airway edema as the underlying mechanism. This interstitial edema may also lead to nonspecific bronchial hyperresponsiveness. We hypothesized that patients with chronic renal failure may present nonspecific bronchial hyperresponsiveness due to subclinical interstitial pulmonary edema. Methods: We studied 18 chronic renal failure undergoing regular hemodialysis 3 times a week(New York Heart Association Class II) without concomittent disease. These patients were checked pulmonary function test and metacholine provocation test before hemodialysis and same procedure was repeated if responsive, after hemodialysis. Results: 1) 12 out of 18 patients before hemodialysis were reactive in metacholine provocation test(66.7%) before hemodialysis. This airway hyperresponsiveness were decreased after hemodialysis. 2) Pulmonary function was improved after hemodialysis and change in $FEV_1$ was correlated with change in weight(r=-0.62, p<0.01). 3) There was a close correlation between log $PD_{20}$ and $FEF_{25}$, which is one of the variables of the peripheral airways(r=0.58, p<0.05). Conclusion: We speculated interstitial pulmonary edema may play a significant role in bronchial hyperresponsiveness and lung function impaired in patients with chronic renal failure.

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ACUTE PULMONARY EDEMA CAUSED BY IMPAIRED SWITCHING FROM NASAL TO ORAL BREATHING DURING THE CALDWELL-LUC OPERATION RESULTING FROM ANESTHESIA: A CASE REPORT (전신마취 후 칼드웰럭씨 수술을 통한 extubation 시행시 발생한 급성 폐부종: 증례보고)

  • Oh, Min-Seok;Kim, Su-Gwan
    • Journal of the Korean Association of Oral and Maxillofacial Surgeons
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    • v.32 no.2
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    • pp.157-160
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    • 2006
  • Nasotracheal intubation is performed routinely in maxillofacial surgery to optimize visualization of the surgical field. The CaldwellLuc operation is an approach to the maxillary sinus through the labiogingival sulcus and canine fossa. The operation is used to treat chronic maxillary sinusitis, and involves curettage of the mucosa of the maxillary sinus and the creation of an inferior meatal antrostomy. After the operation, a nasal Foley catheter is inserted into the inferior nasal meatus for the discharge of blood and tissue fluid. Then, the nostril is packed with vaseline gauze. Before the patients awaken, they experience impaired switching from nasal to oral breathing. Pulmonary edema can result from excessive negative intrathoracic pressure caused by acute airway obstruction in patients breathing spontaneously. During anesthesia and sedation, airway obstruction can occur at the levels of the pharynx and larynx. Even in patients who are awake, alteration in the ability to change the breathing route from nasal to oral may affect breathing in the presence of an airway obstruction, causing this catastrophic event. We experienced a case in which acute pulmonary edema resulted from acute airway obstruction triggered by the patient's inability to switch the breathing route from nasal to oral during emergence from anesthesia.

A Case of Non-cardiogenic Pulmonary Edema caused by Nitrogen Dioxide Poisoning after Cutting Copper Pipe with an Oxyethylene Torch (산소 에틸렌 토치로 동파이프 절단작업 후 발생한 이산화질소 중독에 의한 비심인성 폐부종 1례)

  • JeGal, Yang-Jin;Ahn, Jong-Joon;Seo, Kwang-Won;Cha, Hee-Jeong;Kwon, Woon-Jung;Kim, Yang-Ho
    • Journal of The Korean Society of Clinical Toxicology
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    • v.4 no.2
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    • pp.175-179
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    • 2006
  • Welders are exposed to a number of hazards including metal fumes, toxic gases, electricity, heat, noise, and radiation such as ultraviolet and infrared light. We encountered a patient who developed non-cardiogenic pulmonary edema within a day after cutting copper pipe with an oxyethylene torch. The patient was a 26-year-old welder. He complained of dyspnea, generalized myalgia, and febrile sensation the following morning. The patient's chest X-ray and chest CT scan showed extensively distributed and ill-defined centrilobular nodules. Both his symptoms and chest X-ray abnormalities improved spontaneously. We attributed the patient's symptoms to non-cardiogenic pulmonary edema due to nitrogen dioxide, reasoning that: 1) the pipe consisted only of copper, according to material safety data sheet (MSDS); 2) a previous report in the literature demonstrated increased nitrogen dioxide levels under similar conditions; 3) the patient's clinical course and radiologic findings were very reminiscent of non-cardiogenic pulmonary edema following accidental exposure to nitrogen dioxide.

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Acute Pulmonary Edema Caused by Inhalation of Nitrogen Dioxide (이산화질소(Nitrogen Dioxide ; $NO_2$) 흡입에 의한 폐부종 1예)

  • Doh, Sung-Kyoung;Jeong, Hong-Bae;Koh, Young-Min;Yoon, Yoon-Bo;Chung, Yeon-Tae
    • Tuberculosis and Respiratory Diseases
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    • v.44 no.6
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    • pp.1408-1413
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    • 1997
  • A 68 year-old male was admitted with complaint of dyspnea and nonproductive cough which developed 6 hours after accidental inhalation of nitrogen dioxide. On admission, acute pulmonary edema and severe hypoxemia were found. With oxygen and bronchodilator therapy, diffuse alveolar consolitation and his dyspnea were improved from the following day. He was discharged at 8th hospital day with prednisolone 30mg daily for prevention of bronchiolitis obliterans. During 6 weeks of follow up, there was no evidence of bronchiolitis obliterans.

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A Literature Review On Postpartum Deep Venous Thrombosis (산후(産後) 심부정맥혈전증(深部靜脈血栓症)에 관한 문헌적(文獻的) 고찰(考察))

  • Kim, Yeon-Tae
    • Journal of Korean Medical classics
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    • v.35 no.4
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    • pp.63-75
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    • 2022
  • Objectives : The purpose of this study is to examine the significance of Korean Medical literature on representative symptoms of postpartum deep venous thrombosis (DVT), such as lower extremity edema and pulmonary embolism. Methods : Pattern differentiation, cause, symptoms and treatment related to DVT and pulmonary embolism as written in Korean Medical texts were studied comparatively. Results : In Korean Medical understanding, DVT could be linked to lower extremity edema due to blood stagnation, while pulmonary embolism could be linked to coughing, asthma and nose bleed due to stagnated blood. Conclusions : To prevent relapse and manage its after-effects, Korean Medical treatment for DVT and pulmonary embolism is much in need.

A Case of Pheochromocytoma Accompanied with Alveolar Hemorrhage and Cardiogenic Pulmonary Edema (심인성 폐부종과 폐포성 출혈을 보인 갈색세포종 1예)

  • Jeong, Jong Pil;Ban, Hee Jung;Kim, Soo Ock;Son, Jun Gwang;Ju, Jin Yung;Kwon, Yong Soo;Oh, In Jae;Kim, Kyu Sik;Kim, Yu Il;Lim, Sung Chul;Kim, Young Chul
    • Tuberculosis and Respiratory Diseases
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    • v.64 no.3
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    • pp.219-223
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    • 2008
  • Pheochromocytoma is derived from the chromaffin tissue. The typical finding of pheochromocytoma is paroxysmal hypertension accompanied with various signs and symptoms that are due to the excess of catecholamines or other bioactive substances. Yet the diagnosis is sometimes difficult to make because its clinical presentation is quite variable. Especially, hemoptysis is a very rare symptom, so the diagnosis is often missed or delayed. Without making the correct diagnosis and then subsequently administering treatment, the condition may be fatal. We herein report on a 68 year-old woman who was admitted because of abdominal pain and hemoptysis. The initial radiologic findings suggested pulmonary edema with alveolar hemorrhage. The urine catecholamine levels were elevated and she developed catecholamine-induced cardiomyopathy. We performed bronchial arterial embolization and we administered alpha blocker medication for controlling the hemoptysis and hypertension. After the temporary symptomatic improvement, her clinical course was aggravated by pneumonia and pulmonary edema. In spite of performing definitive surgery for pheochromocytoma, she died of postoperative hemodynamic instability.

A Case of Pulmonary Edema by Idiopathic Rupture of Mitral Chordae Tendinae (승모판 건삭 파열로 인한 급성 폐부종 1예)

  • Park, Hye Kyeong;Kang, Yeun Jeong;Choi, Sang Bong;Park, I Nae;Jeung, Hoon;Hur, Jin Won;Lee, Hyun Kyung;Yum, Ho Kee;Lee, Hyuk Pyo;Jang, Ji Min;Kim, Joung Sook;Choi, Soo Jeon
    • Tuberculosis and Respiratory Diseases
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    • v.63 no.5
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    • pp.458-461
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    • 2007
  • The typical radiographic findings of pulmonary edema from the increased hydrostatic pressure shows centrally localized consolidation, which is known as a "butterfly or bat's wing" pattern. These terms describe the anatomic distribution of edema that uniformly involve the hilum and medulla of the lung but not the peripheral region of the lung parenchyma (cortex). We present a case of butterfly wing-like pulmonary edema on a chest radiograph by mitral regurgitation due to an idiopathic chordal rupture.