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A Case of Pheochromocytoma Accompanied with Alveolar Hemorrhage and Cardiogenic Pulmonary Edema  

Jeong, Jong Pil (Department of Internal Medicine, Gwangju Christian Hospital)
Ban, Hee Jung (Department of Pulmonary and Critical Care Medicine, Chonnam National University Medical School)
Kim, Soo Ock (Department of Internal Medicine, Miraero21 Hospital)
Son, Jun Gwang (Department of Internal Medicine, Seonam University College of Medicine)
Ju, Jin Yung (Department of Pulmonary and Critical Care Medicine, Chonnam National University Medical School)
Kwon, Yong Soo (Department of Pulmonary and Critical Care Medicine, Chonnam National University Medical School)
Oh, In Jae (Department of Pulmonary and Critical Care Medicine, Chonnam National University Medical School)
Kim, Kyu Sik (Department of Pulmonary and Critical Care Medicine, Chonnam National University Medical School)
Kim, Yu Il (Department of Pulmonary and Critical Care Medicine, Chonnam National University Medical School)
Lim, Sung Chul (Department of Pulmonary and Critical Care Medicine, Chonnam National University Medical School)
Kim, Young Chul (Department of Pulmonary and Critical Care Medicine, Chonnam National University Medical School)
Publication Information
Tuberculosis and Respiratory Diseases / v.64, no.3, 2008 , pp. 219-223 More about this Journal
Abstract
Pheochromocytoma is derived from the chromaffin tissue. The typical finding of pheochromocytoma is paroxysmal hypertension accompanied with various signs and symptoms that are due to the excess of catecholamines or other bioactive substances. Yet the diagnosis is sometimes difficult to make because its clinical presentation is quite variable. Especially, hemoptysis is a very rare symptom, so the diagnosis is often missed or delayed. Without making the correct diagnosis and then subsequently administering treatment, the condition may be fatal. We herein report on a 68 year-old woman who was admitted because of abdominal pain and hemoptysis. The initial radiologic findings suggested pulmonary edema with alveolar hemorrhage. The urine catecholamine levels were elevated and she developed catecholamine-induced cardiomyopathy. We performed bronchial arterial embolization and we administered alpha blocker medication for controlling the hemoptysis and hypertension. After the temporary symptomatic improvement, her clinical course was aggravated by pneumonia and pulmonary edema. In spite of performing definitive surgery for pheochromocytoma, she died of postoperative hemodynamic instability.
Keywords
Pheochromocytoma; Alveolar hemorrhage; Cardiogenic pulmonary edema;
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