• 대한수학회지
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• 제42권6호
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• pp.1169-1186
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• 2005

We study the properties of the transversal Killing and twistor spinors for a Riemannian foliation with a transverse spin structure. And we investigate the relations between them. As an application, we give a new lower bound for the eigenvalues of the basic Dirac operator by using the transversal twistor operator.

• 대한수학회보
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• 제57권5호
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• pp.1299-1306
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• 2020

An almost contact metric manifold is called a quasi contact metric manifold if the corresponding almost Hermitian cone is a quasi Kähler manifold, which was introduced by Y. Tashiro [9] as a contact O*-manifold. In this paper, we show that a quasi contact metric manifold with Killing characteristic vector field is a K-contact manifold. This provides an extension of the definition of K-contact manifold.

• 대한수학회보
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• 제49권6호
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• pp.1163-1178
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• 2012

In this paper, we study the geometry of Einstein half lightlike submanifolds M of a semi-Riemannian space form $\bar{M}(c)$ subject to the conditions: (a) M is screen conformal, and (b) the coscreen distribution of M is a conformal Killing one. The main result is a classification theorem for screen conformal Einstein half lightlike submanifolds of a Lorentzian space form with a conformal Killing coscreen distribution.

• 대한수학회지
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• 제46권6호
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• pp.1193-1206
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• 2009

C. Guenther studied the stability of DeTurck flows by using maximal regularity theory and center manifolds, but these arguments can not solve the stability of Ricci flows because the Ricci flow equation is not strictly parabolic. Recognizing this deficiency, the present paper considers and obtains the stability of Ricci flows, and of quasi-Ricci flows in view of some Killing conditions.

• 호남수학학술지
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• 제41권2호
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• pp.329-342
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• 2019

In this paper, we give a geometric approach to Killing magnetic flux surfaces in Euclidean 3-space and solve the differential equations which expressed the mentioned surfaces. Furthermore we give some examples and draw their pictures by using the programme Mathematica.

• 한국어병학회지
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• 제28권3호
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• pp.171-174
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• 2015

Previously, we had reported that some Miamiensis avidus, a major pathogen of scuticociliatosis in cultured olive flounder, strongly agglutinated by flounder immune sera could escape from the agglutinated mass within a few hours. In the present study, we observed that M. avidus not only escaped from the agglutinated mass but also conducted division(s) before shedding its old covering. Furthermore, ciliates that survived the antibody-dependent complement killing (ADCK) assay were not killed even when re-exposed to a freshly prepared ADCK assay. This result suggests that the liberated ciliates from the ADCK assay might change not only their i-antigen types but also the epitopes of major surface antigens, which debilitate antibody-mediated complement killing ability.

• 한국어병학회지
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• 제15권2호
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• pp.77-82
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• 2002

Effects of stress-induced suppression of humoral immunity on scuticociliate killing activity of olive flounder plasma were investigated. Changes in glucose level. alternative complement activity and Iysozyme activity of plasma by handling stress were analysed in relation to in vitri parasiticidal activity of plasma. The plasma glucose level was about two times higher in fish after a handling stress than in control fish. Plasma lysozyme activity and natural haemolytic activity were decreased in suessed fish.The scuticociliate killing activity of plasma was significantly lower in stressed fish than in non-stressed control fish. The present results indicated that stress-induced immunodepression could be a cause of scuticociliatosis occurrence in olive flounder.

• 대한수학회지
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• 제59권2호
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• pp.255-278
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• 2022

In this paper, first we introduce a new notion of generalized Killing structure Jacobi operator for a real hypersurface M in complex hyperbolic two-plane Grassmannians SU_2,m/S (U₂·U_m). Next we prove that there does not exist a Hopf real hypersurface in complex hyperbolic two-plane Grassmannians SU_2,m/S (U₂·U_m) with generalized Killing structure Jacobi operator.

• 한국수산과학회지
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• 제31권6호
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• pp.852-858
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• 1998

1996년 7월 마산만의 해수를 0.8 $\mu$m filter에 여과한 여과액과 f/2배지에서 배양한 P. micans와의 혼합배양액에서 P. micans를 사멸시키는 해양세균을 분리하였다. 분리균의 형태학적 및 생화학적 검사와 균체지방산을 분석하여 동정한 결과 Pseudomonas 속으로 동정되어 Pseudomonas sp. LG-2로 명명하였으며, 그 살조특성을 조사한 결과는 아래와 같다. Pseudomonas sp. LG-2의 개체수와 배양여과액의 농도가 높을수록 P. micans의 사멸효과가 높게 나타났다. $1.3\times10^5,\;1.3\times10^6$ cells/ml의 농도로 접종한 시험관의 P. micans는 급격히 감소하여 배양 7일 후에는 $10^2$ cells/ml 이하로 사멸되었다. 또한, 배양석과액의 최종농도가 $30\%$일 경우에는 급격히 감소하여 배양 3일 후 거의 사멸하였다. Pseudomonas sp. LG-2의 성장시기에 따른 배양여과액의 사멸효과는 잠복기의 경우 P. micans의 성장에 큰 영향을 미치지 않았으나, 대수증식기 중기의 배양여과액은 접종 5일 후 P. micans의 개체수를 1/2로 감소시켰으며, 정상기의 배양여과액은 접종 후 급격히 감소시켜 배양 3일 후 대부분 사멸시켰다. Alexandrium tamarense, Prorocentrum micans, Scrippsiella trochoidea, Gymnodinium sanguineum, Cochlodinium polykrikoides의 5종의 적조원인 편모조류에 대한 Pseudomonas sp. LG-2의 살조특이성은 P. micans와 S. trochoidea에 살조효과를 나타내었다.

• Animal cells and systems
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• 제8권2호
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• pp.125-133
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• 2004

We have extended our previous work that cross-linking CD4 molecules using specific MAb induced antigen nonspecific, MHC unrestricted killing of virally infected target cells by CD$4^+$We have extended our previous work that cross-linking CD$4^+$ molecules using specific MAb induced antigen nonspecific, MHC unrestricted killing of virally infected target cells by CD$4^+$ T cells. The killing activity of antibody activated CD$4^+$T cells was completely blocked by herbimycin A, a protein tyrosine kinase (PTK) inhibitor, but not by bisindolylamaleimide, a protein kinase C (PKC) inhibitor. Herbimycin A treated human or bovine peripheral blood CD$4^+$T cells lacked PTK activity and failed to kill virally infected target cells even after cross-linking of CD4 molecules. The CD$4^+$cross-linking failed to induce effector cell proliferation or the transcription of TNF${\beta}$ Upregulation of TNF${\beta}$ was induced by incubating the antibody activated effector cells with BHV-1 infected D17 target cells for 10 h. Anti-TNF${\beta}$ antibody partially abolished (13-44%) the direct effector cell-mediated antiviral cytotoxicity. However, this antibody neutralized 70 to 100% of antiviral activity of effector and target cell culture supernatants against BHV-1 infected D17 cells. The inhibition level of the antiviral activity by the antibody was dependent on the effector and target cell ratio. These results support the hypothesis that increased p$56^ICK enzyme activity in effector cells transduces a signal critical for effector cell recognition of viral glycoproteins expressed on the target cells. Following target cell recognition, lytic cytokines known to participate in target cell killing were produced. A better understanding of the killing activity displayed by CD$4^+$T lymphocytes following surface receptor cross-linking will provide insight into the mechanisms of cytotoxic activity directed toward virally-infected cells.T cells. The killing activity of antibody activated CD$4^+$T cells was completely blocked by herbimycin A, a protein tyrosine kinase (PTK) inhibitor, but not by bisindolylamaleimide, a protein kinase C (PKC) inhibitor. Herbimycin A treated human or bovine peripheral blood CD4T cells lacked PTK activity and failed to kill virally infected target cells even after cross-linking of CD4molecules. The CD4 cross-linking failed to induce effector cell proliferation or the transcription of TNF$\beta$. Upregulation of TNF$\beta$ was induced by incubating the antibody activated effector cells with BHV-1 infected D17 target cells for 10 h. Anti-TNF$\beta$ antibody partially abolished (13-44%) the direct effector cell-mediated antiviral cytotoxicity. However, this antibody neutralized 70 to 100% of antiviral activity of effector and target cell culture supernatants against BHV-1 infected D17 cells. The inhibition level of the antiviral activity by the antibody was dependent on the effector and target cell ratio. These results support the hypothesis that increased $56^ICK enzyme activity in effector cells transduces a signal critical for effector cell recognition of viral glycoproteins expressed on the target cells. Following target cell recognition, lytic cytokines known to participate in target cell killing were produced. A better understanding of the killing activity displayed by CD$4^+$T lymphocytes following surface receptor cross-linking will provide insight into the mechanisms of cytotoxic activity directed toward virally-infected cells.