• International Journal of Internet, Broadcasting and Communication
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• v.14 no.4
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• pp.163-172
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• 2022

The purpose of this study is to investigate the changes in the number of repetitions and the repetition rate according to the exercise mode when performing 65%1RM resistance exercise at the 1-minute rest interval and the 3-minute rest interval. Sixteen healthy male subjects were treated with Bench press and Biceps curl of 65%1RM intensity at 1 and 3 minute rest intervals. The number of repetitions for each set of 1 minute rest interval showed a significant decrease from 1set to 5set in bench press. biceps curl showed a significant decrease from 1set to 4set. The repetition rate according to the exercise mode with a 1-minute rest interval showed a significant difference from 2sets to 4sets. In the repetition rate for each set, bench press showed a significant decrease from 1set to 5set. biceps curl showed a significant decrease from set 1 to set 4. The number of repetitions according to the exercise mode with a 3-minute rest interval showed a significant difference from 2sets to 5sets. In the number of repetitions for each set, bench press showed a significant decrease from 1set to 5set. biceps curl showed a significant decrease from 1set to 4set. The repetition rate according to the exercise mode with a 3-minute rest interval showed a significant difference from 2sets to 5sets. In the repetition rate for each set, bench press showed a significant decrease from 1 set to 5 sets. biceps curl showed a significant decrease from 1set to 4set. In summary, the decrease in the number of repetitions according to the set progression in the resistance exercise of the endurance depends on the exercise mode, and the increase of the rest interval or the decrease of the weight-intensity should be considered when aiming for more exercise.

• Journal of the military operations research society of Korea
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• v.8 no.1
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• pp.53-70
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• 1982

This thesis develops a more realistic and applicable new set covering model that is adjusted and supplied by the existing set covering models, and induces an algorithm for solving the new set covering model, and applies the new model and the algorithm to an actual set covering problems. The new set covering model introduces a probabilistic covering aistance ($0{\eqslantless}p{\eqslantless}1$)or time($0{\eqslantless}p{\eqslantless}1$) instead of a deterministic covering distance(0 or 1) or time (0 or 1) of the existing set covering model. The existing set covering model has not considered the merit of the overcover of customers. But this new set covering model leads a concept of this overcover to a concept of the parallel system reliability. The algorithm has been programmed on the UNIVAC 9030 for solving large-scale covering problems. An application of the new set covering model is presented in order to determine the locations of the air surveillance radars as a set covering problem for a case-study.

• Korean Journal of Microbiology
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• v.53 no.4
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• pp.286-291
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• 2017

In eukaryotic cells, histone modification is an important mechanism to regulate the chromatin structure. The methylation of the fourth lysine on histone H3 (H3K4) by Set1 complex is one of the various well-known histone modifications. Set1 complex has seven subunits including Swd2, which is known to be important for H2B ubiquitination dependent on H3K4 methylation. Swd2 was reported to regulate Set1's methyltransferase activity by binding to near RNA recognition motif (RRM) domain of Set1 and to act as a component of CPF (Cleavage and Polyadenylation Factors) complex involved in RNA 3' end processing. According to the recent reports, two functions of Swd2 work independently of each other and the lethality of Swd2 knockout strain was known to be caused by its function as a component of CPF complex. In this study, we found that Swd2 could influence the Set1's stability as well as histone methyltransferase activity through the association with RRM domain of Set1. Also, we found that ${\Delta}swd2$ mutant bearing truncated-Set1, which cannot interact with Swd2, lost its lethality and grew normally. These results suggest that the dual functions of Swd2 in H3K4 methylation and RNA 3' end processing are not independent in Saccharomyces cerevisiae.

• Nuclear Engineering and Technology
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• v.25 no.3
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• pp.457-469
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• 1993

JEF-1-based 50-group cross section set for fast reactor calculations was generated using NJOY system. The set was then examined by analyzing measured integral quantities such as criticality and central reaction rate ratios for 27 fast critical assemblies. The calculated results using the new set were also compared with those of ENDF/B-IV or-V-based fast set. In general, the JEF-1-based set shows an improvement in predicting measured integral quantities in comparison with the previous set. With a few exceptions, JEF-1 results are comparable to those of ENDF/B-V.

• Journal of Life Science
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• v.27 no.10
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• pp.1104-1110
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• 2017

SET1A is a histone H3K4 methyltransferase that catalyzes di- and trimethylation of histone H3 at lysine 4 (H3K4). Mono-, di-, and trimethylations on H3K4 (H3K4me1, H3K4me2, and H3K4me3, respectively) are generally correlated with gene activation. Although H3K4 methylation is associated with the stimulation of adipogenesis of 3T3-L1 preadipocytes, it remains unknown whether SET1A plays a role in the regulation of adipogenesis of 3T3-L1 preadipocytes. Here, we investigated whether SET1A regulates 3T3-L1 preadipocytes' adipogenesis and characterized the mechanism involved in this regulation. SET1A expression increased during 3T3-L1 preadipocytes' adipogenesis. Consistent with the increased SET1A expression, the global H3K4me3 level had also increased on day 2 after the induction of adipogenesis in 3T3-L1 adipocytes. SET1A knockdown using siRNA in 3T3-L1 preadipocytes inhibited 3T3-L1 preadipocytes' adipogenesis, as assessed by Oil Red O staining and the expression of adipogenic genes, indicating that SET1A stimulates the adipogenesis of 3T3-L1 preadipocytes. SET1A knockdown inhibited the cell proliferation of 3T3-L1 cells during mitotic clonal expansion (MCE) via down-regulation of the cell cycle gene cyclin E1, as well as the DNA synthesis gene, dihydrofolate reductase. Furthermore, SET1A knockdown repressed peroxisome proliferator-activated receptor gamma ($PPAR{\gamma}$) expression during the late stage of adipogenesis. These results indicate that SET1A stimulates MCE and $PPAR{\gamma}$ expression, which leads to the promotion of 3T3-L1 preadipocytes' adipogenesis.

• Bulletin of the Korean Mathematical Society
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• v.53 no.4
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• pp.959-970
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• 2016

In this paper, we generalize the stability for an n-dimensional cubic functional equation in Banach space to set-valued dynamics. Let $n{\geq}2$ be an integer. We define the n-dimensional cubic set-valued functional equation given by $$f(2{{\sum}_{i=1}^{n-1}}x_i+x_n){\oplus}f(2{{\sum}_{i=1}^{n-1}}x_i-x_n){\oplus}4{{\sum}_{i=1}^{n-1}}f(x_i)\\=16f({{\sum}_{i=1}^{n-1}}x_i){\oplus}2{{\sum}_{i=1}^{n-1}}(f(x_i+x_n){\oplus}f(x_i-x_n)).$$ We first prove that the solution of the n-dimensional cubic set-valued functional equation is actually the cubic set-valued mapping in [6]. We prove the Hyers-Ulam stability for the set-valued functional equation.

• Honam Mathematical Journal
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• v.27 no.3
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• pp.405-419
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• 2005

We present some properties characterizing the Mandelbrot set of quadratic rational maps. Any quadratic rational map is conjugate to either $z^2+c$ or ${\lambda}(z+1/z)+b$. For ${\mid}{\lambda}{\mid}=1$, we find the figure of the Mandelbrot set $M_{\lambda}$, the set of parameters b for which the Julia set of ${\lambda}(z+1/z)+b$ is connected. It is seen to be the whole complex plane if ${\lambda}{\neq}1$, but it is intricate fractal if ${\lambda}=1$. This supplements the work already investigated for the case ${\mid}{\lambda}{\mid}>1$.

• Journal of the Chungcheong Mathematical Society
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• v.9 no.1
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• pp.107-113
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• 1996

For continuous maps f of the circle to itself, we show that (1) the set of ${\omega}$-limit points is contained in the set of nonwandering points of $f^n$ for all $n{\geq}1$. (2) if the set of turning points of f is finite, then the set of accumulation points of non wandering set is contained in the set of non wandering points of $f^n$ for all $n{\geq}1$.

• Journal of the Chungcheong Mathematical Society
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• v.28 no.1
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• pp.29-38
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• 2015

We compute the Hausdorff and packing dimensions of the cylindrical lower or upper local dimension set for a self-similar measure having different minimum and maximum of the local dimension on a self-similar set satisfying the open set condition.

• The Pure and Applied Mathematics
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• v.5 no.1
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• pp.13-16
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• 1998

For continuous maps f of the circle to itself, we show that (1) every $\omega$-limit point is recurrent (or almost periodic) if and only if every $\omega$-limit set is minimal, (2) every $\omega$-limit set is almost periodic, then every $\omega$-limit set contains only one minimal set.