• Journal of electromagnetic engineering and science
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• v.6 no.4
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• pp.203-208
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• 2006

Channel estimation and data detection for OFDM systems over time- and frequency-selective channels are investigated. Relying on the complex exponential basis expansion channel model, a pilot-embedded channel estimation scheme with low computational complexity and spectral efficiency is proposed. A periodic pilot sequence is superimposed at a low power on information bearing sequence at the transmitter before modulation and transmission. The channel state information(CSI) can be estimated using the first-order statistics of the received data. In order to enhance the performance of channel estimation, we recover the transmitted data which can be exploited to estimate CSI iteratively. Simulation results show that the proposed method is suitable for doubly-selective channel estimation for the OFDM systems and the performance of the proposed method can be better than that of the Wiener filter method under some conditions. Through simulations, we also analyze the factors which can affect the system performances.

• Journal of information and communication convergence engineering
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• v.3 no.4
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• pp.172-175
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• 2005

In this paper, a new method to get the optimum channel capacity of a Multiple-Input Multiple­Output (MIMO) system is presented. The proposed method exploits the diagonal structure of channel matrix to maximize the channel capacity. The diagonal format of the channel matrix is formed by multiplying the transmitted signal with the pre-compensated channel PCC) matrix. Numerical simulations show that the proposed method exploiting the diagonal structure of channel matrix could significantly increase the system capacity compared with the system without applying the diagonal structure of channel matrix.

• The Korean Journal of Physiology and Pharmacology
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• v.7 no.6
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• pp.333-339
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• 2003

It has been demonstrated that an unidentified cytosolic factor(s) reduces $K_{ACh}$ channel function. Therefore, this study attempted to elucidate the cytosolic factor. Fresh cytosol isolated from normal heart (FC) depressed the $K_{ACh}$ channel activity, but cytosol isolated from the ischemic hearts (IC) did not modulate the channel function. Electrophorectic analysis revealed that a protein of ${\sim}80 kDa was markedly reduced or even lost in IC. By using peptide sequencing analysis and Western blot, this 80 kDa protein was identified as transferrin (receptor-mediated $Fe^{3+}$ transporter, 76 kDa). Direct application of transferrin (100 nM) to the cytoplasmic side of inside-out patches decreased the open probability ($P_o$, 12.7${\pm}6.4%, n=4) without change in mean open time (${\tau}_o$, $98.5{\pm}1.3$%, n=4). However, the equimolar apotransferrin, which is free of $Fe^{3+}$, had no effect on the channel activity (N*$P_o$, $129.1{\pm}13.5$%, n=3). Directly applied $Fe^{3+}$ (100 nM) showed results similar to those of transferrin (N*$P_o$: $21.1{\pm}3.9$%, n=5). However $Fe^{2+}$ failed to reduce the channel function (N*$P_o$, $106.3{\pm}26.8$%, n=5). Interestingly, trivalent cation La3+ inhibited N*$P_o$ of the channel ($6.1{\pm}3.0$%, n=3). Taken together, these results suggest that $Fe^{3+}$ bound to transferrin can modulate the $K_{ACh}$ channel function by its electrical property as a polyvalent cation.

• Journal of the Institute of Convergence Signal Processing
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• v.3 no.4
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• pp.67-73
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• 2002

There have been increased concerns on mobile stratospheric communication system(SCS) for the purpose of advanced service of personal and high speed communication systems. In fact, this SCS is considered and studied for IMT-2000 service by ITU. Although, it is important to make accurate channel model for prediction of the SCS performance, there is no measured channel data in this system. Thus, in this paper, we estimate the performance of SCS bye use of channel model provided by Corazza(2) and modified by You(3). And also, the effects of channel codings on system performance are analyzed by deriving bit error performance based on realistic Rician log-normal fading channel models. The performance results are divided into three kinds of areas with three kinds of elevation angles 20$^\cire$, 45$^\cire$, and 80$^\cire$. And also the effects of forward error correction channel codings on system performance with Hamming(7,4), HCH( IS,7) and convolutional code of constraint length 3 and code rate R=1/2.

• Journal of Embryo Transfer
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• v.19 no.2
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• pp.101-112
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• 2004

Chloride($Cl^-$) channels play critical roles in cell homeostasis and its specific functions such as volume regulation, differentiation, secretion, and membrane stabilization. The presence of these channels have been reported in all kinds of cells and even in frog oocytes. These essential role of $Cl^-$­ channels in cell homeostasis possibly play any role in egg homeostasis and in the early stage of development, however, there has been no report about the presence of $Cl^-$­ channel in the mammalian oocyte. This study was performed to elucidate the presence of $Cl^-$­ channels in hamster eggs. When allowing only $Cl^-$­ to pass through the channel of the egg membrane by using impermeant cation such as N-methyl-D-glucamine(NMDG), single channel currents were recorded. These channel currents showed typical long-lasted openings interrupted by rapid flickering. Mean open $time({\tau}o)$ was 43${\pm}$10.14 ms(n=9, at 50 mV). The open probability(Po) was decrease with depolarization. The current-voltage relation showed outward rectification. Outward slop conductance(32${\pm}$5.4 pS, n=22) was steeper than the inward slop conductance(10${\pm}$1.3 pS). Under the condition of symmetrical 140 mM NaCl, single channel currents were reversed at 0 mV(n=4). This reversal potential(Erev) was shifted from 0 mV at 140 mM concentration of internal NaCl(140 mM [Na+]i) to ­9.8${\pm}$0.5 mV(n=4) at 70 mM [Na+]i and 11.5${\pm}$1.9 mV at 280 mM [Na+]i(n=4) respectively, strongly suggesting that these are single $Cl^-$­ channel currents. To examine further whether this channel has pharmacological property of the $Cl^-$­ channel, specific Cl­ channel blockers, IAA-94(Indanyloxyacetic acid-94) and DIDS(4, 4'-diisothiocyan ostillben- 2-2'disulfonic acid) were applied. IAA-94 inhibited the channel current in a dose-dependent manner and revealed a rapid and flickering block. From these electrophysiological and pharmacological resluts, we found the novel $Cl^-$­ channel present in the hamster oocyte membrane. The first identification of $Cl^-$­ channel in the hamster oocyte may give a clue for the further study on the function of $Cl^-$­ channel in the fertilization and cell differentiation.

• Journal of Physiology & Pathology in Korean Medicine
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• v.24 no.4
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• pp.592-597
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• 2010

Cinnamomi Ramulus is one of the medicinal plants that have been used to improve various diseases caused by insufficient blood circulation. This study was performed for the investigation of vasodilation efficacy ethanol extract of Cinnamomi Ramulus (CR). CR exhibited vascular relaxation against phenylephrine (PE, $10^{-6}M$)-, KCl- and NaF-induced contraction in rat thoracic aorta. In addition, its relaxation was endothelium-independent. Treatment of potassium channel blockers such as gilbenclamide (Gli, $10^{-5}M$), tetraethylammonium (TEA, 1 mM) and 4-aminopyridine (4-AP, 0.2 mM) did not effect on the relaxation of CR. The relaxant effects were also not inhibited by pre-treatment of rat aorta with L-NAME ($10^{-4}M$), methylene blue ($10^{-5}M$), indomethacin ($10^{-5}M$), and atropine ($10^{-6}M$). However, nifedipine ($10^{-5}M$), L-type $Ca^{2+}$ channel blocker, in part attenuated the relaxation of CR ($0.2\;mg/m{\ell}$), but SK&F96365 ($3{\times}10^{-5}M$), receptor activated $Ca^{2+}$ channel blocker and 2-APB ($10^{-4}M$), store operated $Ca^{2+}$ channel blocker did not affact dilation of CR. These findings suggest that the endothelium-independent relaxation effect of CR is partly related with inhibition of $Ca^{2+}$ influx via voltage dependent $Ca^{2+}$ channel.

• Journal of the Korea Institute of Information and Communication Engineering
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• v.14 no.4
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• pp.1005-1014
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• 2010

When reader collect tags, we found that they tend to get together to specific interface in Multi-Interface Multi-Channel 2.4GHz Active RFID system. To solve this problem, we designed the LP-Combind and AP-Balanced protocol for load distribution between interfaces, then verified its superiority of the performance through the simulation. There are three problems to implement designed protocols in hardware of firmware-level. first, tag selects randomly the channel of reader and reader need the method which can change the channel of tags. second, reader has the synchronization problem between reader and tag. third, reader has problem that MCU of reader have to operate simultaneously dual interface. To slove this problems, we designed the message and implemented method for tag channel change and the protocol in order to adjust synchronization between reader and tag, Therefore, we compared and analyzed the performance of protocols by experiment. If LP windows size is same, the performance of LP-Combined protocol and AP-Balanced protocol which lower collision probability by its load distribution is more outstanding than single interface protocol performance.

• Journal of the Korean Institute of Telematics and Electronics A
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• v.32A no.8
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• pp.133-139
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• 1995

In this paper, the frequency separation locking and interval stabilization of 4-channel DFB-LDs have been demonstrated using a fiber Fabry-Perot filter with an free spectral range of 100GHz. Frequency fluctuation and locking range of each channel were appeared to be within 15MHz and over 12GHz, respectively. Back-reflection curve from the fiber Fabry-Perot filter was used for the extraction of an error signal in order to increase the number of accomodable channels and extinction ratio.

• The Journal of Korean Institute of Communications and Information Sciences
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• v.21 no.4
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• pp.1008-1018
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• 1996

For assigning channel in a cellular sireless network, we purpose satisfying constraints with a related probability between cells and channels on the channel interference matrix formed by m terminals and n channels. And we purpose to get efficient channel assigning to limited channels in a wireless cell using parallelism of neural networks. In this paper, we solve the problem according to the number of requeirements when channel change with 11-533 procession elements. We demonstrate efficiency of proposed algorithm through same simulations in a specific time period.

• The Korean Journal of Physiology and Pharmacology
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• v.4 no.6
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• pp.445-453
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• 2000

The present investigation tested the hypothesis that the activation of protein kinase G (PKG) leads to a phosphorylation of $Ca^{2+}-activated$ potassium channel $(K_{Ca}\;channel)$ and is involved in the activation of $K_{Ca}$ channel activity in cerebral arterial smooth muscle cells of the rabbit. Single-channel currents were recorded in cell-attached and inside-out patch configurations of patch-clamp techniques. Both molsidomine derivative 3-morpholinosydnonimine-N-ethylcarbamide $(SIN-1,\;50\;{\mu}M)$ and 8-(4-Chlorophenylthio)-guanosine-3',5'-cyclic monophosphate $(8-pCPT-cGMP,\;100\;{\mu}M),$ a membrane-permeable analogue of cGMP, increased the $K_{Ca}$ channel activity in the cell-attached patch configuration, and the effect was removed upon washout of the drugs. In inside-out patches, single-channel current amplitude was not changed by SIN-1 and 8-pCPT-cGMP. Application of ATP $(100\;{\mu}M),$ cGMP $(100\;{\mu}M),$ ATP+cGMP $(100\;{\mu}M\;each),$ PKG $(5\;U/{\mu}l),$ ATP $(100\;{\mu}M)+PKG\;(5\;U/{\mu}l),$ or cGMP $(100\;{\mu}M)+PKG\;(5\;U/{\mu}l)$ did not increase the channel activity. ATP $(100\;{\mu}M)+cGMP\;(100\;{\mu}M)+PKG\;(5\;U/{\mu}l)$ added directly to the intracellular phase of inside-out patches increased the channel activity with no changes in the conductance. The heat-inactivated PKG had no effect on the channel activity, and the effect of PKG was inhibited by 8-(4-Chlorophenylthio)-guanosine-3',5'-cyclic monophosphate, Rp-isomer $(Rp-pCPT-cGMP,\;100\;{\mu}M),$ a potent inhibitor of PKG or protein phosphatase 2A (PP2A, 1 U/ml). In the presence of okadaic acid (OA, 5 nM), PP2A had no effect on the channel activity. The $K_{Ca}$ channel activity spontaneously decayed to the control level upon washout of ATP, cGMP and PKG, and this was prevented by OA (5 nM) in the medium. These results suggest that the PKG-mediated phosphorylations of $K_{Ca}$ channels, or some associated proteins in the membrane patch increase the activity of the $K_{Ca}$ channel, and the activation may be associated with the vasodilating action.