• Title/Summary/Keyword: 호남

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A Mid-Late Maturing, Multi-Disease Resistant and Good-Quality Rice Variety "Hwangkeumnuri" (벼 중만생 고품질 복합내병성 품종 "황금누리")

  • Kim, Ki Young;Shin, Mun Sik;Ko, Jae Kwon;Kim, Bo Kyeong;Ha, Ki Yong;Ko, Jong Cheol;Baek, Man Kee;Nam, Jeong Kwon;Kim, Young Doo;Choung, Jin Il;Noh, Gang Il;Kim, Woo Jae;Park, Hyun Su;Kang, Hyun Jung;Kim, Chung Kon
    • Korean Journal of Breeding Science
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    • v.40 no.2
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    • pp.188-191
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    • 2008
  • 'Hwangkeumnuri' is a japonica rice variety developed and registered by the rice breeding team of Honam Agricultural Research Institute, NICS, RDA in 2006. This variety was derived from a cross between 'Milyang 165' (Junambyeo) with good quality and high yield and HR14732-B-67-2-3 with multi-disease resistance. This variety has about 125 days growth duration from transplanting to harvesting in west-southern coast, Honam and Youngnam plain of Korea. It is about 76 cm in culm length and tolerance to lodging. In reaction to biotic and abiotic stresses, it shows resistance to blast, bacterial blight pathogen from $K_1$ to $K_3$ and stripe virus, but susceptible to other major diseases and insect pests. The milled rice of 'Hwangkeumnuri' exhibits translucent, relatively clear non-glutinous endosperm and midium short grain. It has similar amylose content of 18.9% and lower protein content of 6.2%, and good palatability of cooked rice compared with 'Nampyeongbyeo'. The milled rice yield performance of this variety is about 5.74 MT/ha in local adaptability test for three years. 'Hwangkeumnuri' would be adaptable to west-southern coast, Honam and Yeongnam plain of Korea.

The Possibility of Geographical Range Change of 'Ho-Nam' as a Place Name (지명 '호남(湖南)'의 형성과 지리적 범위 변화 가능성)

  • Cho, Sung-Wook
    • Journal of the Korean association of regional geographers
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    • v.14 no.3
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    • pp.199-211
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    • 2008
  • This study looks into existing discussion about the entry time and the geographical criteria point of 'Ho-Nam' as a place name. And discusses the reason which the geographical criteria point is not clear with emphasis on the possibility of the geographical range change of place name. The result is as follows. Firstly, the regional criteria of 'Ho-Nam' is Kum-River if we consider direction and the role as a regional barrier among the existing offered criteria. Secondary, the criteria of Ho-Nam and Ho-Seo might be different each other. A place name of Ho-Nam had appeared before Chosun period as a geographical concept and changed to the cultural concept during Chosun period. And Ho-Seo was established in Chosun period. But 'Ho-Nam=Jeon-la Province' and 'Ho-Seo=Chung-cheong Province' is established during Chosun period as a cultural concept. Thirdly, the geographical range of Ho-Nam of Go-Ryeo period and Chosun period were different each other. Fourthly, the criteria of Ho-Nam may be the Han-River if we consider the entry time and geographical range change.

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Cordycepin Inhibits LPS-induced Cell Migration and Invasion in Human Colorectal Carcinoma HCT116 cells through Down-regulation of Prostaglandin E2-EP4 Receptor (LPS 유도된 HCT116 인간 대장암세포에서 cordycepin의 prostaglandin E2-EP4 receptor 감소 조절을 통한 세포의 이동과 전이 억제 효과)

  • Jung Eun Kim;Bo-Ram Kim;Su Hui Seong;Jin-Ho Kim;Ha-Nul Lee;Chan Seo;Ji Min Jung;Su A Im;Kyung-Min Choi;Jin-Woo Jeong
    • Proceedings of the Plant Resources Society of Korea Conference
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    • 2023.04a
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    • pp.50-50
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    • 2023
  • Prostaglandin E2(PGE2), a major product of cyclooxygenase-2 (COX-2), plays an important role in the carcinogenesis of many solid tumors, including colorectal cancer. Because PGE2 functions by signaling through PGE2 receptors (Eps), which regulate tumor cell growth, invasion, and migration, there has been a growing amount of interest in the therapeutic potential of targeting Eps. In the present study, we investigated the role of EP4 on the effectiveness of cordycepin in inhibititing the migration and invasion of HCT116 human colorectal carcinoma cells. Our data indicate that cordycepin suppressed lipopolysaccharide (LPS)-enhanced cell migration and invasion through the inactivation of matrix metalloproteinases (MMP)-9 as well as the down-regulation of COX-2 expression and PGE2 production. These events were shown to be associated with the inactivation of EP4 and activation of AMP-activated protein kinase (AMPK). Moreover, the AMPK inhibitor, compound C, as well as AMPK knockdown via siRNA, attenuated the cordycepin-induced inhibition of EP4 expression. Cordycepin treatment also reduced the activation of CREB. These findings indicate that cordycepin suppresses the migration and invasion of HCT116 cells. Through modulating EP4 expression and the AMPK-CREB signaling pathway. Therefore, cordycepin has the potential to serve as a potent anti-cancer agent in therapeutic strategies against colorectal cancer metastasis.

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