• 제목/요약/키워드: toxicosis

검색결과 39건 처리시간 0.02초

하태독법(下胎毒法)에 대한 문헌적(文獻的) 고찰(考察) (A study on fetal toxicosis removal therapy)

  • 장규태;김장현;강미선
    • 대한한방소아과학회지
    • /
    • 제17권1호
    • /
    • pp.29-51
    • /
    • 2003
  • This study was progressed for oriental medical care of neonates by fetal toxicosis removal therapy, and the results as follows: 1. The purpose of fetal toxicosis removal therapy : It removes dirty blood in neonatal mouth and prevents diseases. 2. Kinds of fetal toxicosis removal therapy : Radix Glycyrrhizae(甘草), Rhizoma Coptidis(黃連), Cinnabaris-Mel(朱蜜), Calculus Bovis(牛黃), Sojae Semen Praepartum, Juice of Semen Allii Tuberosi(非汁) and mercury(汞粉). 3. A period of fetal toxicosis removal therapy : At birth, 3rd day or so, one week or so, after umbilical omission. 4. Methods of fetal toxicosis removal therapy: Purgative therapy(下法) and Emetic therapy(吐法).

  • PDF

갑상선기능항진증(T3 중독증) 치험 1례에 대한 보고 (A clinical case report of Hyperthyroidism(T3 toxicosis) patient)

  • 이주호;최정식;조충식;김철중
    • 혜화의학회지
    • /
    • 제16권2호
    • /
    • pp.225-228
    • /
    • 2007
  • Objective : This study was designed to evaluate oriental medicine therapy on a hyperthyroidism(T3 toxicosis) patient Methods : The clinical data was analyzed on a patient with hyperthiroidism(T3 toxicosis). This patient was treated with oriental medicine therapy only. After treatment, we analyzed a paient clinical symptom and thyroid function(T3, free T4, TSH) Results : After treatment, improvement was seen in clinical symptom, thyroid function test. conclusion : The study suggests that oriental medicine therapy is effective on the treatment of hyperthyroidism(T3 toxicosis).

  • PDF

부자(附子)오두(烏頭) 중독증(中毒症)에 관한 소고(小考) (A Study on Bu一ja(<附子>, Oh-du<烏頭>) Toxicosis)

  • 나창수;윤여충
    • 대한한의학회지
    • /
    • 제16권1호통권29호
    • /
    • pp.451-467
    • /
    • 1995
  • In order to study on Bu-ja(Oh-du) toxicosis, the outhor investigated many documents. The results obtained were as follows: 1. Toxicosis of Bu-ja(Oh-du) arised mainly from carelessness in use. 2. In the symptoms of Bu-ja(Oh-du), Heart System(心系) symptom often came out. 3. The general and local paralysis, falling of blood pressure in Heart System(心系) symptom : vertigo, cramp in Liver System(肝系) symptom : nausea, vomiting in Spleen System (脾系) symptom : difficulty of breathing in Lung System(肺系) symptom : wasting disease in Kidney System (腎系) symptom frequently originated from toxicosis of Bu-ja(Oh-du). 4. In the detoification method, there are atropin injection, carrying out a gastric lavage, and supplying a liquid medicine from the western medicine : taking ginger-licorice root decoction, taking mung beans thick decoction and getting vomited with soaked cinnamon from the oriental medicine. And also keeping warm and oxygen inhalation were used to counteract poison. 5. A pregnant woman, heat of excess type(實熱), yang sydrome(陽證 )must not be taken Bu-ja(Oh-du).

  • PDF

실업계 고교생의 컴퓨터 중독과 일탈행동 실태 연구 (A Study of Computer Toxicosis and Deviant Behavior in Vocational High School Students)

  • 최낙관;정종인
    • 컴퓨터교육학회논문지
    • /
    • 제7권3호
    • /
    • pp.79-89
    • /
    • 2004
  • 정보통신기술의 발달과 정보화로 인한 역기능이 새로운 사회적인 문제로 등장하고 있는데 그 중의 하나가 청소년들의 컴퓨터 중독이다. 이러한 중독증을 측정하기 위해 Kimberly S. Young이 개발한 검사지를 많이 사용하는 바, 본 연구에서는 그와 병행하여 한국정보문화진흥원이 개발한 한국형 인터넷중독검사(K-척도)를 사용하고 그 결과를 기준으로 연구하였다. K-척도의 진단기준에 따라서 분류한 결과 일반사용자 534명(64.8%), 잠재적위험 사용자 229명(27.8%), 고위험 사용자 61명(7.4%)으로 나타났다. 본 연구는 이들 고위험 사용자들의 컴퓨터 중독의 충분조건과 필요조건(표출행동)에 대하여 연구했고 이들을 예방하기위한 지도 방향을 탐색해 본다.

  • PDF

국내에서 발생한 면양의 만성 동중독증 예의 관찰 (Chronic Ovine Copper Toxicosis in Korea)

  • 김대용;권오경;서일복
    • 한국임상수의학회지
    • /
    • 제15권2호
    • /
    • pp.455-459
    • /
    • 1998
  • Ten sheeps from one farm had sudden onset of anorexia, jaundice and hemoglobinuria and died within 3 days after showing clinical signs during 3 months period. Postmortom examination was performed on one case and copper concentrations in the livers kidney and serum of the necmpsied minim were analysed. Grossly, the conjunctive, subcutaneous tissue and abdominal fat were generally icteric. The liver was enlarged with yellowish orange in color. The kidney was enlarged with dark red in color and the urinary bladder was filled with dark red urine. Histopathologically, centrilobular hepatocellular necrosis, neutrophilic infiltrations bile stasis and aggregation of fine granules-laden macrophages in the portal area were noted in the liver. Most of the Bowman's spaces and renal tubules were filled with homogenous eosinophilic fluid. Extramedullary hematopoiesis was noted in the submandibular lymph node. Copper concentrations in serum, liver and kidney of the necropsied animal were 25.0, 2732.8 and 471.3 ppm respectively. Based on the gross and histopathological findings and the high copper concentrations in the organs, this case was diagnosed as chronic copper toxicosis. Possible etiology on this outbreak is also discussed.

  • PDF

진돗개에서 ivermectin의 독성 I. 임상증상과 혈액학적 변화 관찰 (Toxicity of ivermectin in Jindo-dogs 1. Clinical and hematological observation)

  • 이채용;오석일;이정길
    • 대한수의학회지
    • /
    • 제37권4호
    • /
    • pp.855-862
    • /
    • 1997
  • Ivermectin is a synthetic derivative of the naturally occurring avermectin $B_{1a}$ (22, 23-dihydroavermectin $B_{1a}$) and $B_{1b}$ (22, 23-dihydroavermectin $B_{1b}$), It is widely used as antiparasitic and pesticidal agents because of its remarkably potent and broad spectrum of antiparasitic activity. Although the drug has shown excellent anthelmintic effects, development of toxicosis in some animals such as the Collie species of dog is well documented. However, no studies have been reported on the toxic effects of the drug in Korean native animals such as the Jindo dog. The toxic effect of ivermectin was evaluated in 25 Jindo dogs divided into five groups which were orally administered with ivermectin at dosage levels of $200{\mu}g/kg$, $300{\mu}g/kg$, $600{\mu}g/kg$ and $2,500{\mu}g/kg$ of body weight, respectively. Toxic signs were not observed in the groups receiving $200{\mu}g/kg$ and $300{\mu}g/kg$ B.W. ivermectin. One dog developed mild clinical signs of toxicosis in the group receiving $600{\mu}g/kg$ dosage of ivermectin. In the group with $2,500{\mu}g/kg$ dosage, all dogs developed mild (salivation, drooling, vomiting, mydriasis, and/or confusion) and/or moderate (ataxia and tremors) clinical signs of toxicosis. Hematologic changes were not observed in the groups receiving $200{\mu}g/kg$, $300{\mu}g/kg$ and $600{\mu}g/kg$ B.W. ivermectin. In the groups receiving $2,500{\mu}g/kg$ B.W., total erythrocyte counts, total and differential leukocyte counts and hemoglobin levels were not affected by drug. Aspartate aminotransferase levels were increased after administration of ivermectin, while serum cholesterol and blood glucose levels were decreased.

  • PDF

Thoroughbred 경주마에서 amitraz 중독증 치료 1례 (A case of treatment on amitraz toxicosis in a Thoroughbred racehorse)

  • 양재혁;송희은;이경갑;지영흔;우호춘;임윤규
    • 대한수의학회지
    • /
    • 제50권3호
    • /
    • pp.253-257
    • /
    • 2010
  • A 3-year-old female Thoroughbred racehorse was presented following the accidental oral and skin administration of amitraz. This case report describes the clinical signs and the treatment of this horse. Clinical signs of amitraz toxicosis are associated with the stimulation of alpha2-adrenergic receptors. Amitraz is seldom fatal because the effects can be reversed by alpha2-adrenergic antagonists. The horse displayed typical clinical signs of colic, including pawing, small hard drops, tranquillisation, depression, ataxia, muscular incoordination and impaction colic lasting up to 7 days. The syndrome was accompanied by mild dehydration. The horse survived after persistent symptomatic treatment, including the giving of intravenous fluids, antibiotics, multiple doses of mineral oil per os, nonsteroidal anti-inflammatory drugs and dexamethasone intramuscularly and intravenously.

개의 ethylene glycol 중독 (Ethylene glycol toxicosis in a dog)

  • 진영화;우계형;김재훈;황의경;서일복
    • 한국수의병리학회지
    • /
    • 제2권2호
    • /
    • pp.147-151
    • /
    • 1998
  • A five-month old female Great Dane with a history of depression and convulsion was submitted to the Pathology and Diagnosis Reference Division of National Veterinary Research and Quarantine service. The dog had ingested concentrated antifreeze and died 12 days after the sudden onset of clinical signs. Main gross lesion was limited to swollen kidneys with diffusely fine granularity on the cut surface. Principle microscopic lesions were focused on the kidneys including dilated Bowman's space, thickened glomerular basement membrane, many light yellowish crystals, and proteinaceous casts in some tubules. Some neutrophils and lymphocytes were focally infiltrated in interstitium of the cortex and medulla. Crystals were angular or quite irregular in shape and arranged in sheave or rosette form. Under polarized light, the crystals were birefringent, which is typical of calcium oxalate crystals. Crystals were more concentrated in the cortex than in the medulla. A few crystals were also observed in the small blood vessels of the brain. Based upon the history of ingestion of antifreeze and the identification of calcium oxalate crystals in the tissue sections of the kidney and brain, this case was diagnosed as ethylene glycol toxicosis in a dog.

  • PDF