• Title/Summary/Keyword: decoy trap

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Attack Surface Expansion through Decoy Trap for Protected Servers in Moving Target Defense

  • Park, Tae-Keun;Park, Kyung-Min;Moon, Dae-Sung
    • Journal of the Korea Society of Computer and Information
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    • v.24 no.10
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    • pp.25-32
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    • 2019
  • In this paper, we propose a method to apply the attack surface expansion through decoy traps to a protected server network. The network consists of a large number of decoys and protected servers. In the network, each protected server dynamically mutates its IP address and port numbers based on Hidden Tunnel Networking that is a network-based moving target defense scheme. The moving target defense is a new approach to cyber security and continuously changes system's attack surface to prevent attacks. And, the attack surface expansion is an approach that uses decoys and decoy groups to protect attacks. The proposed method modifies the NAT table of the protected server with a custom chain and a RETURN target in order to make attackers waste all their time and effort in the decoy traps. We theoretically analyze the attacker success rate for the protected server network before and after applying the proposed method. The proposed method is expected to significantly reduce the probability that a protected server will be identified and compromised by attackers.

Human Periodontal Ligament Fibroblasts Support the Osteoclastogenesis of RAW264.7 Cells (치주인대섬유아세포가 파골세포분화에 미치는 영향)

  • Lee, Ho;Jeon, Yong-Seon;Choi, Seoung-Hwan;Kim, Hyung-Seop;Oh, Kwi-Ok
    • Journal of Periodontal and Implant Science
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    • v.32 no.4
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    • pp.733-744
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    • 2002
  • The fibroblasts are the principal cells in the periodontal ligament of peridontium. As the periodontal ligament fibroblasts (PDLF) show similar phenotype with osteoblasts, the PDLF are thought to play an important role in alveolar bone remodeling. Cell-to-cell contacted signaling is crucial for osteoclast formation. Recently it has been reported that PDLJ enhance the bone resorbing activity of osteoclasts differentiated from hematopoietic preosteoclasts. The aims of this study were to $clarify\;^{1)}$ the mechanism of PDLF-induced osteoclastogenesis $and\;^{2)}$ whether we can use preosteoclast cell line instead of primary hematopoietic preosteoclast cells for studying the mechanism of PDLF-induced osteoclastogenesis. Osteoclastic differentiation of mouse macrophage cell line RAW264.7 was compared with that of mouse bone marrow-derived M-CSF dependent cell (MDBM), a well-known hematopoietic preosteoclast model, by examining, 1) osteoclast-specific gene expression such as calcitonin receptor, M-CSF receptor (c-fms), cathepsin K, receptoractivator nuclear factor kappa B (RANK) ,2) generation of TRAP(+) multinucleated cells (MNCs), and 3) generation of resorption pit on the $OAAS^{TM}$ plate. RAW264.7 cultured in the medium containing of soluble osteoclast differentiation Factor (sODF) showed similar phenotype with MDBM-derived osteoclasts, those are mRNA expression pattern of osteoclast-specific genes, TRAP(+) MNCs generation, and bone resorbing abivity. Formation of resorption pits by osteoclastic MNCs differentiated from sODF-treated RAW264.7, was completely blocked by the addition of osteoprotegerin (OPG), a soluble decoy receptor for ODF, to the sODF-containing culture me야um. The effects of PDLF on differentiation of RAW264.7 into the TRAP(+) multinucleated osteoclast-like cells were examined using coculture system. PDLF were fxed with paraformaldehyde, followed by coculture with RAW264.7, which induced formation of TRAP(+) MNCs in the absence of additional treatment of sODF. When compared with untreated and fixed PDLF (fPDLF), IL-1 ${\beta}$-treated, or lipopolysaccha-ride-treated and then fixed PDLF showed two-folld increase in the supporting activity of osteoclastogenesis from RAW264.7 coculture system. There were no TRAP(+) MNCs formation in coculture system of RAW264.7 with PDLF of no fixation. These findigs suggested that we can replace the primary hematopoietic preosteoclasts for RAW264. 7 cell line for studying the mechanism of PDLF-induced osteoclastogenesis, and we hypothesize that PDLF control osteoclastogenesis through ODF expression which might be enhanced by inflammatory signals.

Investigation of natural habitats for Korean Musk deer (Moschus Moschiferus) (멸종 위기종 한국사향노루의 서식지 조사)

  • Kim, Jong-Taek;Kim, Geon-Joong;Kim, Hyeon-Cheol
    • Korean Journal of Veterinary Service
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    • v.30 no.3
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    • pp.459-466
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    • 2007
  • The Korean Musk deer (Moschus Moschiferus) is endangered due to heavy hunting and Massive destructions of natural habitat. They are included in CITES appendix II. In addition they are designated as a precious natural product in 1968 and listed as endangered species by the Government and Cultural Heritage Administration. At present, the number of musk deer in Korea is smaller than minimum viable population. Without increment of the population size, natural restoration is impossible. It is necessary to develop the artificial growth and re-enforcement methods in order to conservation Korean musk deer. Furthermore, It is necessary to ensure that we have an adequate individual group for artificial growth and re-enforcement. To ensure that we have an adequate individual group, it is necessary to know ecology of musk deer. This study is conducted to confirm and investigate of habitat of musk deer in order to capture individual musk deer. we investigated, confirmed habitats, found traces and captured using trap and decoy. Captured musk deer is male and 5.5 kg, this have canine tooth of 1.5cm, is estimated 15 months old. After capture, we measured each parts of body and tested to research of parasite infection. Strongyloides papillous was founded in the feces. It is essential to get healthy individual and establish of artificial growth technique.

EDGE: An Enticing Deceptive-content GEnerator as Defensive Deception

  • Li, Huanruo;Guo, Yunfei;Huo, Shumin;Ding, Yuehang
    • KSII Transactions on Internet and Information Systems (TIIS)
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    • v.15 no.5
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    • pp.1891-1908
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    • 2021
  • Cyber deception defense mitigates Advanced Persistent Threats (APTs) with deploying deceptive entities, such as the Honeyfile. The Honeyfile distracts attackers from valuable digital documents and attracts unauthorized access by deliberately exposing fake content. The effectiveness of distraction and trap lies in the enticement of fake content. However, existing studies on the Honeyfile focus less on this perspective. In this work, we seek to improve the enticement of fake text content through enhancing its readability, indistinguishability, and believability. Hence, an enticing deceptive-content generator, EDGE, is presented. The EDGE is constructed with three steps: extracting key concepts with a semantics-aware K-means clustering algorithm, searching for candidate deceptive concepts within the Word2Vec model, and generating deceptive text content under the Integrated Readability Index (IR). Furthermore, the readability and believability performance analyses are undertaken. The experimental results show that EDGE generates indistinguishable deceptive text content without decreasing readability. In all, EDGE proves effective to generate enticing deceptive text content as deception defense against APTs.

Development of High-Speed Real-Time Signal Processing Unit for Small Millimeter-wave Tracking Radar (소형 밀리미터파 추적 레이다용 고속 실시간 신호처리기 개발)

  • Kim, Hong-Rak;Park, Seung-Wook;Woo, Seon-Keol;Kim, Youn-Jin
    • The Journal of the Institute of Internet, Broadcasting and Communication
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    • v.19 no.1
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    • pp.9-14
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    • 2019
  • A small millimeter-wave tracking radar is a pulse-based radar that searches, detects, and tracks a target in real time through a TWS (Track While Scan) method for a traps target on the sea with a large RCS running at low speed. It is necessary to develop a board equipped with a high-speed CPU to acquire and track target information through LPRF, DBS, and HRR signal processing techniques for a trap target operating various kinds of dexterous objects such as chaff and decoy, We designed a signal processor structure including DFT (Discrete Fourier Transform) module design that can perform real - time FFT operation using FPGA (Field Programmable Gate Array) and verified the signal processor implemented through performance test.

STUDY ON THE REGULATION OF OSTEOCLAST AND T CELL ACTIVATION VIA CELL MEMBRANE PROTEINS OF TNF FAMILY, CD137 LIGAND AND RANK LIGAND (TNF계 CD137L 및 RANKL의 파골세포와 T 세포에 대한 활성조절)

  • Hong, Sung-Joon;Park, Jae-Hong;Lee, Hyeon-Woo;Lee, Keung-Ho
    • Journal of the korean academy of Pediatric Dentistry
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    • v.35 no.4
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    • pp.597-606
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    • 2008
  • Resorption of alveolar bone in periodontitis is due to excessive differentiation and activation of osteoclasts. Bacterial antigens causing periodontitis activates CD4 T cells, which leads to expressing RANK ligand (RANKL) on CD4 T cells. RANKL binds RANK on preosteoclasts or osteoclasts, and enhances the differentiation preosteoclasts into osteoclasts and the activation of mature osteoclasts. CD137, one of TNF receptor (TNFR) family, expressed on activated T cells binds with CD137 ligand (CD137L) on antigen presenting cells. Cross-linking of CD137 by CD137L acts as T cell co-stimulatory signals and, therefore, enhances the activation of T cell. In this study, I elucidated the biological responses of CD137L on (pre)osteoclasts and RANKL on T cells in the context of in vivo interaction between T cells and osteoclasts. RAW264.7, murine monocytic cells, constitutively express CD137L. Ligation of CD137L with anti-CD137L mAb inhibited RANKL-induced osteoclast formation in a dosedependent manner. Bone marrow cells are expressed CD137L by the treatment with M-CSF. Cross-linking of CD137L abolished M-CSF/ RANKL-evoked the formation of multi-nucleated osteoclasts. Both mouse CD4 and CD8 T cells are expressed RANKL following their activation. Ligation of RANKL with OPG, the decoy receptor for RANKL, inhibited both CD4 and CD8 T cell proliferation. These effects were attributed to RANKL-induced apoptosis. These data indicate that CD137L and RANKL on osteoclasts and T cells, respectively provide them with inhibitory signal.

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