• 방송공학회논문지
• /
• 제11권3호
• /
• pp.302-310
• /
• 2006

국내 위성 디지털 멀티미디어 방송 (DMB : Digital Multimedia Broadcasting)의 전송 표준방식은 DS-CDM-QPSK 방식으로 위성중계기와 지상중계기 (Gap-filler)를 통하여 다채널 고품질의 멀티미디어 방송 서비스를 제공할 수 있다. 그러나 다중 경로 페이딩 채널 환경에서는 방송 채널을 식별하는 Walsh 부호간의 직교성이 상실되고 다중 채널 간섭 (MCI : Multi-channel Interference)이 증가하여 수신 성능이 저하된다. 본 논문에서는 위성 DMB의 수신 성능을 향상시키기 위해 탭 선택 알고리즘을 사용한 칩 등화 수신기를 제안하고 기존의 Rake 수신기와 수신 성능을 비교 분석한다.

• The Korean Journal of Physiology and Pharmacology
• /
• 제20권1호
• /
• pp.25-33
• /
• 2016

Ion channels in carcinoma and their roles in cell proliferation are drawing attention. Intracellular $Ca^{2+}$ ($[Ca^{2+}]_i$)-dependent signaling affects the fate of cancer cells. Here we investigate the role of $Ca^{2+}$-activated $K^+$ channel (SK4) in head and neck squamous cell carcinoma cells (HNSCCs) of dif-ferent cell lines; SNU-1076, OSC-19 and HN5. Treatment with $1{\mu}M$ ionomycin induced cell death in all the three cell lines. Whole-cell patch clamp study suggested common expressions of $Ca^{2+}$-activated $Cl^-$ channels (Ano-1) and $Ca^{2+}$-activated nonselective cation channels (CAN). 1-EBIO, an activator of SK4, induced outward $K^+$ current (ISK4) in SNU-1076 and OSC-19. In HN5, ISK4 was not observed or negligible. The 1-EBIO-induced current was abolished by TRAM-34, a selective SK4 blocker. Interestingly, the ionomycin-induced cell death was effectively prevented by 1-EBIO in SNU-1076 and OSC-19, and the rescue effect was annihilated by combined TRAM-34. Con-sistent with the lower level of ISK4, the rescue by 1-EBIO was least effective in HN5. The results newly demonstrate the role of SK4 in the fate of HNSCCs under the $Ca^{2+}$ overloaded condition. Pharmacological modulation of SK4 might provide an intriguing novel tool for the anti-cancer strategy in HNSCC.

• The Korean Journal of Physiology and Pharmacology
• /
• 제10권2호
• /
• pp.95-99
• /
• 2006

Employing electrophysiological and cell proliferation assay techniques, we studied the effects of $Ca^{2+}$ -activated $K^+$ channel modulators on the proliferation of human dermal fibroblasts, which is important in wound healing. Macroscopic voltage-dependent outward $K^+$ currents were found at about -40 mV stepped from a holding potential of -70 mV. The amplitude of $K^+$ current was increased by NS1619, a specific large-conductance $Ca^{2+}$-activated $K^+$ (BK) channel activator, but decreased by iberiotoxin (IBTX), a specific BK channel inhibitor. To investigate the presence of an intermediate-conductance $Ca^{2+}$-activated $K^+$ (IK) channels, we pretreated the fibroblasts with low dose of TEA to block BK currents, and added 1-EBIO (an IK activator). 1-EBIO recovered the currents inhibited by TEA. When various $Ca^{2+}$-activated $K^+$ channel modulators were added into culture media for 1∼3 days, NS1619 or 1-EBIO inhibited the cell proliferation. On the other hand, IBTX, clotrimazole or apamin, a small conductance $Ca^{2+}$-activated $K^+$ channel (SK) inhibitor, increased it. These results suggest that BK, IK, and SK channels might be involved in the proliferation of human dermal fibroblasts, which is inversely related to the channel activation.

• 대한전자공학회:학술대회논문집
• /
• 대한전자공학회 2007년도 하계종합학술대회 논문집
• /
• pp.343-344
• /
• 2007

In this paper, we study the signed-DLMS adaptive algorithm of wireless repeater for solving shadow region due to propagation between base station and mobile station. The the signed-DLMS algorithm reduces interference signals from multipath and solves the oscillation problem of repeater by estimation and cancellation. To efficiently reject interference signal, the signed-DLMS adaptive algorithm is applied. The computational complexities of the signed-DLMS are reduced verse standard LMS algorithm. Wireless ICS repeater based on signed-DLMS reduces the cost and is able to increase channel capacities.

• 한국방송∙미디어공학회:학술대회논문집
• /
• 한국방송공학회 2014년도 추계학술대회
• /
• pp.56-58
• /
• 2014

본 논문에서는 압축 도메인에서 고속으로 움직임을 검출하고 해당 구간을 저장 하는 알고리즘을 제안한다. 제안하는 알고리즘은 H.264/AVC 기반의 압축 비트스트림에서 움직임 벡터와 참조프레임을 이용하여 움직임이 있는 프레임을 검출하고 움직임 유무에 따라 GOP 단위로 저장하는 과정을 수행한다. 압축도메인에서 움직임 검출과 구간 저장을 수행함으로써 복잡도를 낮추고 비디오 저장을 위한 공간을 절약해 실시간 다채널 영상 처리에 최적화 된 성능을 제공한다. 제안하는 움직임 검출 및 저장 시스템은 single thread 환경에서 실시간으로 평균 2957 프레임을 처리 가능하며, Multi thread의 경우 30 fps 영상 98개 채널을 실시간으로 처리 가능하다.

• 대한전자공학회:학술대회논문집
• /
• 대한전자공학회 2008년도 하계종합학술대회
• /
• pp.77-78
• /
• 2008

With advent of 3G mobile communication services, video telephony becomes one of the major services. However, due to a narrow channel bandwidth, the current video telephony services have not yet reached a satisfied level. In this paper, we propose an ROI (Region-Of-Interest) based improvement of visual quality for video telephony services with the H.264|MPEG-4 Part 10 (AVC: Advanced Video Coding) codec. To this end, we propose a face detection and tracking method to define ROI for the AVC codec based video telephony. Experiment results show that our proposed ROI based method allowed for improved visual quality in both objective and subjective perspectives.

• 한국생물물리학회:학술대회논문집
• /
• 한국생물물리학회 2003년도 정기총회 및 학술발표회
• /
• pp.41-41
• /
• 2003

Small conductance calcium-activated potassium channels (or SKCa channels) are potassium selective, voltage-independent, and activated by intracellular calcium concentration. These channels play important roles in excitable cells such as neuron in the central nervous system (Vergara et al., 1998). The activity of SKCa channels underlies the slow afterhyperpolarization that inhibits neuronal cell firing (Hille, 1991; Vergara et al.,1998). Until now, N-terminal region of rSK2 isn't characterized. To study the role of N-terminus, we constructed the N-terminal deletion mutant and characterized by electrophysiological means. Interestingly, N-terminal deletion mutant be trafficked to membrane couldn't evoke any ionic currents. Thus, N-terminal region has a role in functional rSK2 channel formation. To elucidate the function of N-terminal region, (His)6-conjugated protein was purified and filtrated by affinity column chromatography. Surprisingly, N-terminal region was shown in tetramer size that was supported by cross-linking result. Thus, we predicted that N-terminal region might be involved in the tetramerization of rSK2.

• 경영정보학연구
• /
• 제25권3호
• /
• pp.121-138
• /
• 2023

모바일 기술의 진보는 미디어 산업에 혁신적인 변화를 가져왔다. 모바일 기기가 등장함에 따라 미디어 소비에서의 시공간적 제약이 사라졌다. 본 연구는 한국의 온디맨드 서비스 제공업체의 미디어 소비 데이터셋을 사용하여 모바일 채널의 도입이 개인의 영상 시청 행태에 미치는 영향을 분석하였다. 분석 결과 모바일 채널 채택이 TV 기반의 총 영상 시청 시간과 시청 콘텐츠 수를 유의하게 증가시킨다는 사실을 발견하였다. 본 연구 결과는 모바일 채널이 기존 TV 채널에 보완적인 역할을 한다는 것을 시사한다. 본 연구는 신흥 Over-the-Top(OTT) 시장에서 소비자 사용에 관한 이론 및 실무적 통찰력을 제공한다.

• 대한전자공학회:학술대회논문집
• /
• 대한전자공학회 2000년도 하계종합학술대회 논문집(1)
• /
• pp.181-184
• /
• 2000

In this paper, performance of an optical CDMA system with PCCC and SCCC is simulated and compared. It is assumed that optical channel is an intensity modulated (IM) channel and direct-detection scheme is employed to detect the received optical signal. The modulation scheme used is pulse-position-modulation (PPM). The performance is evaluated in terms of bit error probability. From the simulation results, it is shown that turbo coding offers considerable coding gain with reasonable encoding/decoding complexity.

• BMB Reports
• /
• 제44권10호
• /
• pp.635-646
• /
• 2011

Due to its high external and low internal concentration the $Ca^{2+}$ ion is used ubiquitously as an intracellular signaling molecule, and a great many $Ca^{2+}$-sensing proteins have evolved to receive and propagate $Ca^{2+}$ signals. Among them are ion channel proteins, whose $Ca^{2+}$ sensitivity allows internal $Ca^{2+}$ to influence the electrical activity of cell membranes and to feedback-inhibit further $Ca^{2+}$ entry into the cytoplasm. In this review I will describe what is understood about the $Ca^{2+}$ sensing mechanisms of the three best studied classes of $Ca^{2+}$-sensitive ion channels: Large-conductance $Ca^{2+}$-activated $K^+$ channels, small-conductance $Ca^{2+}$-activated $K^+$ channels, and voltage-gated $Ca^{2+}$ channels. Great strides in mechanistic understanding have be made for each of these channel types in just the past few years.