• Journal of the Korean Society of Industry Convergence
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• v.17 no.4
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• pp.234-244
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• 2014

Recently it has been focused that the automobile engine has developed in a strong upward tendency for the use of the high viscosity and poorer quality fuels in achieving the high performance, fuel economy, and emission reduction. Therefore it is not easy to solve the problems between low specific fuel consumption and exhaust emission control at motor cars. In this study, it is designed and used the engine test bed which is installed with turbocharger and intercooler. In addition to equipped using CRDI by controlling injection timing with mapping modulator, it has been tested and analyzed the engine performance, combustion characteristics, and exhaust emission as operating parameters, and they were engine speeds(rpm), injection timing(bTDC), and engine load(%). From the result of an experimental analysis, peak cylinder pressure and the rate of pressure rise were increased, and the location of it was closer toward top dead center according to the increasing of engine speed and load, and with advancing injection timing. The combustion characteristics are effected by fuel injection timing due to be enhanced the mass burned fraction. Using the engine dynamometer for analyzing the engine performance, the engine torque and power have been enhanced according to advancing the fuel injection timing. In analyzing of exhaust emission, there has been a trade-off between PM and NOx with increasing of engine speed and load, and with advanced injection timing. The experimental data are shown that the formation of NOx has increased and PM, vice versa.

• Journal of Advanced Marine Engineering and Technology
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• v.28 no.6
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• pp.922-930
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• 2004

An experimental investigation was performed to study on the generation of air bubble and air core with swirling flow in a horizontal cicular tube. To determine some characteristics of the flow, 2D PIV technique is employed for velocity measurement in water. The experimental rig is manufactured from an acryl tube. The test tube diameter of 80mm, and a length of 3000mm. The used algorithm is the gray leve cross-correlation method(Kimura et al. 1986). An Ar-ion laser is used and the light from the laser(500mW) passes through a probe to make two-dimensional light sheet. In order to make coded images of the tracer particles on one frame, an AOM(Acoustic-Optical Modulator) is used. The maximum axial velocities showed near the test tube wall at y/D =0.1 and y/D =0.9 along the test tube. The higher Reynolds number increase, the lower axial velocities are showed in the center of the test tube. The air bubbles are generated from Re =10,000 and developed into air core from the recirculating water pump rpm equal 30Hz. The pressure and temperature are measured across the test tube at X/D=3.33.

• Korean Journal of Veterinary Research
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• v.36 no.1
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• pp.65-74
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• 1996

Atrial Natriuretic Peptide(ANP) is a hormone with potent natriuretic, diuretic and relaxing properties of vascular smooth muscle. Specific chemical modulator responsible for the ANP secretion has not yet been found. Although atrial stretch of stretch-release is to be a major stimulus for the secretion of ANP, the precise mechano-molecular transduction mechanism responsible for its evoked secretion remains to be elucidated. It is interested to clarify the effect of superoxide anion in the stretch-induced ANP secretion. In order to investigate the effectg of $H_2O_2$ in the regulation of ANP secretion, a perfused model of left atrium of rats was used. The results obtained were as follows; 1. The ANP secretion and the extracellular fluid(ECF) translocation were accentuated by the effect of repetitive atrial distension-reduction volume at atrial pressure($4cmH_2O$). 2. The dilution curve showed to be in parallel between pure atriopeptin III (AP III) and perfusated buffer. 3. $H_2O_2(5{\times}10^{-4}M)$ accenturated a strectch-release induced increase of the ANP secretion. The amount of released ANP was significantly(p<0.01) increased. These results suggest that the superoxide anion may be involved in the regulatory mechanism of mechanically activated ANP release.

• Journal of the Korean Society of Industry Convergence
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• v.18 no.4
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• pp.207-215
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• 2015

Since the oil shock of 1970's there was a strong upward tendency for the use of the high viscosity and poorer quality fuels. Therefore the misfiring engine occurs due to the decrease of quantity injected for lean burn and emission control in CI diesel engine. In this study, it is designed and used the test bed which is installed with turbocharger and intercooler. In addition to equipped using CRDI by controlling injection timing with mapping modulator, it has been tested and analyzed the engine performance, combustion characteristics, and emission as operating parameters.

• Tribology and Lubricants
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• v.30 no.3
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• pp.183-188
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• 2014

In this paper, we present our analysis of tribological failure examples for an anti-lock brake system(ABS) in a car. The study range of this paper is to improve the quality of ABS system by analyzing with sensor, computer, actuator and oil lines. In the first example, the brake leak from hydraulic supply line in a caliper on the rear left side of the ABS hydraulic modulator. This produces the sponge phenomenon, where the car does not brake even when the driver operates the brake pedal. The hydraulic unit operating ABS is actuator that play role regulating drive condition according with the oil pressure supplied with wheel of a car. In the second example, the service man does not completely tighten the fixed bolt after repairing the car. This causes the ABS warning lamp to light up as the ABS wheel speed sensor cannot detect whether the ABS has been activated. In the third example, the ABS electronic control unit is separated from the soldered part of the inner circuit board. Consequently, the ABS fails in control because the ABS motor pump receives no-signal for the hydraulic unit. The wheel speed sensor has to large durability because of giving signal of acting condition to computer by detected the acceleration and deceleration of wheel of a car. In the fourth example, the ABS warning lamp lights up of when cracks propagate in the circuit board soldering part. The circuit of this computer is very important part for input and output the operating signal of system. Such failures can aggravate the durability of the ABS. Thus, the ABS needs to be optimized to eliminate malfunction phenomenon.

• The Korean Journal of Physiology and Pharmacology
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• v.2 no.5
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• pp.573-580
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• 1998

The study aims to identify the mechanism (s) underlying the altered vasodilatory responses of the pial artery of spontaneously hypertensive rats (SHR) under a hypothesis that calcitonin gene-related peptide (CGRP) exerts a modulator role in the autoregulation of cerebral blood flow (CBF). The animals were divided into four groups: 1) Sprague-Dawley rats (SDR), 2) Wistar rats (WR), 3) SHR with high blood pressure $(BP{\ge}150\;mmHg),$ and 4) SHR with normotensive BP $({\le}150\;mmHg).$ The lower limit of CBF autoregulation in SHR shifted to a higher BP $(82.8{\pm}9.3\'mmHg,\;P<0.05)$ than that in SDR $(58.9{\pm}5.7\;mmHg)$. In SHR, whether the BP levels were high or normotensive, the vasodilator responses to a stepwise hypotension were significantly attenuated unlike with SDR and WR. When artificial cerebrospinal fluid (CSF) containing capsaicin $(3{\times}10^{-7}\;M)$ was suffused over the cortical surface, a transient increase in pial arterial diameter was observed in the SHR with high or normotensive BP. In contrast, SDR and WR showed a large increase in diameter, and the increase was sustained for over 10 minutes. In line with these results, the basal releases of CGRP-like immunoreactivity (CGRP-LI) in the isolated pial arteries from SHR with high and normotensive BP were $12.5{\pm}1.4\;and\;9.8{\pm}2.8\;fmole/mm^2/60\;min\;(P<0.05)$, while those from SDR and WR were $25.5{\pm}3.1\;and\;24.6{\pm}3.1\;fmole/mm^2/60\;min,$ respectively. The isolated basilar arteries showed similar results to those of the pial arteries in SHR. Thus, it is summarized that, in the SHR, the reduced autoregulatory vasodilator responses to stepwise hypotension and capsaicin may be, in part, ascribed to the decreased release of CGRP from the perivascular sensory nerve fibers of the pial arteries, and that altered vasomotor activity in SHR may not be related with the hypertensive tone.

• Journal of the Korean Society of Industry Convergence
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• v.20 no.2
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• pp.105-113
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• 2017

Recently the direct injection diesel engine is the most efficient one available for road vehicles, so this fundamental advantage suggests the compression injection diesel engine are a wise choice for future development efforts. The compression ignition diesel engine, with its bigger compression ratios if compared to the SI engine, offers a higher thermodynamic efficiency, also additionally the diesel engine with its less pumping losses due to the throttled intake charge as in a SI engine has higher fuel economy. But the largest obstacle to the success of this engine is meeting emission standards for Nitric oxides and particulate matter while maintain fuel consumption advantage over currently available engines. Thus its use should be largely promoted, however, diesel engine emits more Nitric oxides and particulate matter than other competing one. There has been a trade-off between PM and NOx, so efforts to reduce NOx have increased PM and vice versa, but trap change this situation and better possibility emerge for treating NOx emission with engine related means, such as injection timing, equivalence ratio, charge composition, and engine speed. The common rail direct injection system is able to adjust the fuel injection timing in a compression ignition engine, so this electronically controlled injection system can reduce the formation of NOx gas without increase in soot. In this study it is designed and used the engine test bed which is installed with turbocharge and intercooler. In addition to equipped using CRDI by controlling injection timing with mapping modulator, it has been tested and analyzed the engine performance, combustion characteristics, and exhaust emission as operating parameters.

• Journal of Chest Surgery
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• v.39 no.12 s.269
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• pp.879-890
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• 2006

Background: The dysfunction of multiple organs is found to be caused by reactive oxygen species as a major modulator of microvascular injury after hemorrhagic shock. Hemorrhagic shock, one of many causes inducing acute lung injury, is associated with increase in alveolocapillary permeability and characterized by edema, neutrophil infiltration, and hemorrhage in the interstitial and alveolar space. Aggressive and rapid fluid resuscitation potentially might increased the risk of pulmonary dysfunction by the interstitial edema. Therefore, in order to improve the pulmonary dysfunction induced by hemorrhagic shock, the present study was attempted to investigate how to reduce the inflammatory responses and edema in lung. Material and Method: Male Sprague-Dawley rats, weight 300 to 350 gm were anesthetized with ketamine(7 mg/kg) intramuscular Hemorrhagic Shock(HS) was induced by withdrawal of 3 mL/100 g over 10 min. through right jugular vein. Mean arterial pressure was then maintained at $35{\sim}40$ mmHg by further blood withdrawal. At 60 min. after HS, the shed blood and Ringer's solution or 5% albumin was infused to restore mean carotid arterial pressure over 80 mmHg. Rats were divided into three groups according to rectal temperature level($37^{\circ}C$[normothermia] vs $33^{\circ}C$[mild hypothermia]) and resuscitation fluid(lactate Ringer's solution vs 5% albumin solution). Group I consisted of rats with the normothermia and lactate Ringer's solution infusion. Group II consisted of rats with the systemic hypothermia and lactate Ringer's solution infusion. Group III consisted of rats with the systemic hypothermia and 5% albumin solution infusion. Hemodynamic parameters(heart rate, mean carotid arterial pressure), metabolism, and pulmonary tissue damage were observed for 4 hours. Result: In all experimental groups including 6 rats in group I, totally 26 rats were alive in 3rd stage. However, bleeding volume of group I in first stage was $3.2{\pm}0.5$ mL/100 g less than those of group II($3.9{\pm}0.8$ mL/100 g) and group III($4.1{\pm}0.7$ mL/100 g). Fluid volume infused in 2nd stage was $28.6{\pm}6.0$ mL(group I), $20.6{\pm}4.0$ mL(group II) and $14.7{\pm}2.7$ mL(group III), retrospectively in which there was statistically a significance between all groups(p<0.05). Plasma potassium level was markedly elevated in comparison with other groups(II and III), whereas glucose level was obviously reduced in 2nd stage of group I. Level of interleukine-8 in group I was obviously higher than that of group II or III(p<0.05). They were $1.834{\pm}437$ pg/mL(group I), $1,006{\pm}532$ pg/mL(group II), and $764{\pm}302$ pg/mL(group III), retrospectively. In histologic score, the score of group III($1.6{\pm}0.6$) was significantly lower than that of group I($2.8{\pm}1.2$)(p<0.05). Conclusion: In pressure-controlled hemorrhagic shock model, it is suggested that hypothermia might inhibit the direct damage of ischemic tissue through reduction of basic metabolic rate in shock state compared to normothermia. It seems that hypothermia should be benefit to recovery pulmonary function by reducing replaced fluid volume, inhibiting anti-inflammatory agent(IL-8) and leukocyte infiltration in state of ischemia-reperfusion injury. However, if is considered that other changes in pulmonary damage and inflammatory responses might induce by not only kinds of fluid solutions but also hypothermia, and that the detailed evaluation should be study.