• Korean Journal of Environmental Agriculture
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• v.30 no.2
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• pp.189-195
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• 2011

BACKGROUND: Wastewaters were collected from 25 sites of two industrial areas of Mymensingh and Gazipur in Bangladesh to assess metallic toxicity of wastewater for irrigation usage. METHODS AND RESULTS: The analyzed wastewaters were slightly alkaline to alkaline in nature and were problematic for irrigation except 3 samples. As per TDS values, 9 samples were rated as fresh water and the rest 16 were classified as brackish water. EC and SAR reflected that all samples were medium salinity (C2), high salinity (C3), very high salinity (C4) and low alkalinity (S1) hazard classes expressed as C2S1, C3S1 and C4S1. Wastewaters of different industries were graded as excellent, good, permissible and doubtful for irrigation purpose as per SSP. According to hardness ($H_T$), wastewater were under moderately hard, hard and very hard classes. Cd, Cr and Cu ions were treated as toxicant for irrigating soils and crops. Zn was problematic for long-term irrigation. The concentrations of Pb, Fe and Na were far below the toxic levels. Synergistic relationships were observed between pH-EC, pH-TDS, EC-TDS, SAR-SSP and SSP-hardness. CONCLUSION(s): If wastewater is applied for irrigation due to the fresh water shortage, it can contaminate soil due to some toxic metal ions.

• Journal of Environmental Health Sciences
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• v.45 no.5
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• pp.520-528
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• 2019

Objectives: This study assessed the cytotoxicity of the metallic mordant cupric acetate (CA) and the protective effect of Albizzia julibrissin (AJ) leaf extract on CA-induced cytotoxicity in NIH3T3 fibroblasts. Methods: For this study, cell viability and antioxidative effects such as the inhibitory activity of lipid peroxidation (LP) and superoxide anion-radical (SAR) scavenging activity were assessed. Results: CA significantly decreased cell viability in a dose-dependent manner, and the $XTT_{50}$ value was measured as $55.0{\mu}M$ of CA. The cytotoxicity of CA was determined as highly toxic by Borenfreund and Puerner's toxic criteria. The catalase antioxidant significantly increased cell viability diminished by CA-induced cytotoxicity in these cultures. Regarding the protective effect of AJ leaf extract on CA-induced cytotoxicity, AJ leaf extract remarkably increased the SAR scavenging ability and the inhibitory ability of LP. From these findings, it is suggested that oxidative stress is involved in the cytotoxicity of CA, and AJ leaf extract effectively protected CA-induced cytotoxicity via antioxidative effects. Conclusions: Natural resources like AJ leaf extract may be a putative therapeutic agent for treatment or alleviation of the toxicity induced by CA metallic mordant.

• Korean Journal of Clinical Laboratory Science
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• v.51 no.2
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• pp.235-244
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• 2019

This study examined the neurotoxicity of aluminum sulfate (AS), an environmental pollutant, and the protective effect of Phrymaleptostachya var. asiatica HARA (PLVAH) extract on the neurotoxicity induced by AS in the cultured C6 glioma cells. For this study, the cell viability and antioxidative effects, such as electron donating (ED) activity, lipid peroxidation (LP) activity, and superoxide anion-radical (SAR) scavenging activity, were analyzed. AS decreased the cell viability significantly in a dose-dependent manner and the $XTT_{50}$ value was measured at $120.0{\mu}M$ of AS. The neurotoxicity of AS was determined to be mid-toxic by Borenfreund and Puerner's toxic criteria. In addition, the catalase (CAT), antioxidant enzyme remarkably increased the cell viability injured by AS-induced neurotoxicity in these cultures. Regarding the protective effect of the PLVAH extract on AS-induced neurotoxicity, PLVAH extract significantly increased the ED ability, and the inhibitory ability of the LP and SAR scavenging ability. These findings suggest that oxidative stress is involved in the cytotoxicity of AS, and the PLVAH extract effectively protected against AS-induced neurotoxicity by its antioxidative effects. Natural resources, such as the PLVAH extract may be a putative therapeutic agent for the treatment of the toxicity induced by heavy metallic compounds, such as AS correlated with the oxidative stress.