• The Journal of Internal Korean Medicine
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• v.13 no.2
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• pp.27-34
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• 1992

This study has been carried out to investigate the cause, symptom and treatment of Gi-Chun (氣喘) by referring to 32 literatures. The results were obtained as follows; 1. The factor causing Gi-Chun (氣喘) is almost damage of feeling (七情損傷). 2. The symptom of Gi-Chun (氣喘) is as follows. agony, breathing urgent, cold hand and feet, body tired, low appetite 3. The treatment of Gi-Chun (氣喘) is as follows. Sunpaeganggi(宣肺降氣), Jihaepyungchun(止咳平喘), gudam(去痰) 4. The drugs(herb) of Gi-Chun (氣喘) is as follows. Samatang(四磨湯), Sachiltang(四七湯), Sojaganggitang(蘇子降氣湯), Gilgyungjigaktang(桔梗枳殼湯), Gamisachiltang(加味四七湯), Gamisoyosan(加味逍遙散).

• Annals of Clinical Neurophysiology
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• v.8 no.1
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• pp.74-77
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• 2006

Repetitive nerve stimulation is a simple and widely used technique to demonstrate neuromuscular transmission defect. A significant decremental response for repetitive hypoglossal nerve stimulation was obtained from the surface recordings in the tongue of a patient with dysarthria and dysphagia. Repetitive hypoglossal nerve stimulation test may be useful in diagnosis of myasthenia gravis with bulbar symptoms only. We utilized repetitive hypoglossal nerve stimulation with tongue recordings and diagnosed a case of myasthenia gravis.

• Communications of the Korean Mathematical Society
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• v.7 no.1
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• pp.1-14
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• 1992

• KCID journal
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• v.10 no.2
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• pp.88-100
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• 2003

• Proceedings of the Korean Society of Agricultural Engineers Conference
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• 2004.11a
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• pp.14-14
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• 2004

• Proceedings of the Korean Society of Agricultural Engineers Conference
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• 2004.11a
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• pp.4-4
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• 2004

• Proceedings of the KIEE Conference
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• 1987.07a
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• pp.293-296
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• 1987

• 대한미세수술학회:학술대회논문집
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• 1992.11a
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• pp.66-66
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• 1992

• Journal of Oriental Physiology
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• v.14 no.2 s.20
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• pp.179-187
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• 1999

To investigate how Jagamchotang provent cellular injury by a certain starting point on reperfusion injury after ischemia in myocardial cell, conducted MTT assay, LM stydy and measured LDH secretion, heart rate and nitric oxide(NO), and got the following results. 1. Jagamchotang did not injure cells even in $20{\mu}g/ml$. 2. Jaganchotang repressed the toxicity of mitochondria and cell membrane in reperfusing after ischemia and repressed the contraction of promontory of myocardial cell and reduction of the number of cells. Also maintained regular heart rate and reduced the number of heart rate. 3. Synthesis of NO by Jagamchotang in ischemia increased 1.9 times than a control. 4. When reperfusing with sodium nitropruside (SNO), NO donor in ischemia repressed the toxicity of mitochondria as the case of reperfusing with Jagamchotang in ischemia. Therefore, putting these findings together, it. can be said the effect of Jagamchotang in ischemia will be closely related with generation of NO.

• Proceedings of the Korean Society for Noise and Vibration Engineering Conference
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• 2009.10a
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• pp.661-662
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• 2009