• Proceedings of the ESK Conference
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• 1997.10a
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• pp.179-185
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• 1997

작업장 뿐만 아니라 일상생활에서도 결코 무시될 수 없는 것이 레져활동으로 인한 소음이다. 최근 급격히 늘어가는 개인 휴대용 카세트(Personal Cassette Players: PCP)사용은 심각한 소음성난청 발생이 우려된다. 본 연구는 PCP사용에 관련된 여러 인자들이 청력손실과 어떻게 얼마나 관련이 있는가를 파악 하고 PCP를 일정시간 사용했을 때 오는 청력 손실을 정량적으로 파악, 분석하는데 근본 목적을 둔다. 정상 청력을 가진 피실험자 18명의 대학생들을 대상으로 정상 청취조건 그리고 배경소음(지하철)에 노출 중과 후의 사용환경과 소음원으로 Hard Music, Soft Music 및 Language/News를 고려하여 평균소음도와 최고 소음도를 측정하였다. 또한 현실적 상황을 고려한 2시간 정도의 PCP의 청취 전과 후의 청력변화를 측정하였다. 통계 분석 결과, 사용환경과 소음원에 대해 휴대용 카세트 청취 소음수준이 유의한 차이가 있었으며, 특히 Hard Music 청취시에는 95dB, 그리고 지하철 소음하에서는 92dB의 높은 (위험한) 청취수 준을 보였다. 최고소음수준의 경우 NIOSH기준을 초과하는 117dB 정도의 매우 위험한 수준까지 나타났다. 또한 2 시간 정도의 PCP소음에 노출 전 후의 청력손실이 주파수 대역에 걸쳐 유의하게 나타났으며, 특히, 가장 민감한 주파수 대역인 4000Hz에서 최고 7dB 정도의 청력 손실이 발생하였다. 본 연구 결과에 따라, PCP사용에 대한 청력보호대책안을 제시하고자 한다.

• Proceedings of the KOR-BRONCHOESO Conference
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• 1976.06a
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• pp.85.1-85
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• 1976

Audiological evaluation was performed for 184 female weavers (351 ears), who were 18~25 years old and have been working under exposure of 103~105 ㏈a of machinary noise. With the duration of employment, they were divided into five groups; under 1 year, 1~2 years, 2~3 years, 3~4 years and over 4 years. The following results were obtained: 1. The incidence ;of noise induced hearing loss was 39.7% (36.2% of ears) and rapidly increased during first 1~2 years. 2. The average maximum hearing loss on the pure tone audiograms appeared at 4,000Hz with .about 40 ㏈ of hearing level. The average hearing level at high frequencies(3,000Hz~8,000Hz) showed below 25㏈ of hearing level in all groups and had the tendency to increase slightly with the duration of working, but the most of hearing losses developed during first one year except at 8,000Hz. 3. The average hearing level at speech frequencies was within normal range but the cases of hearing level over 25 ㏈ at 2,000Hz was 27.1%. 4. The cases with maximum hearing loss at 4,000Hz were 72.7%, but the cases of maximum hearing loss at the other frequencies (3,000Hz, 6,000Hz .and 8,000Hz) were also observed and they increased slightly in numbers with the duration of working. The noise induced hearing loss under the exposure of 103~105㏈a for 5 years, seemed to develope during first 1~2 years and there after slightly .increased hearing loss was showed but no remarkable changes were observed.

• KOREAN ASSOCIATION OF OCCUPATIONAL HEALTH NURSES
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• v.13 no.3 s.48
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• pp.37-41
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• 2006

• KOREAN ASSOCIATION OF OCCUPATIONAL HEALTH NURSES
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• v.13 no.3 s.48
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• pp.10-15
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• 2006

• 월간산업보건
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• s.274
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• pp.11-21
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• 2011

• The Science & Technology
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• s.543
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• pp.51-53
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• 2014

• Proceedings of the Korean Institute of Industrial Safety Conference
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• 2003.05a
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• pp.271-282
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• 2003

산업현장에서 기계류 사용의 증가에 따라 소음 및 진동에 의한 근로자들의 소음성 난청 등의 직업병이 증가하는 실정이다. 특히 산업현장 등에서 널리 사용되는 공기압축기는 고압ㆍ고속의 공기 유동으로부터 발생하는 소음과 피스톤, 스크류 등의 진동 및 마찰에 의해 큰 소음이 발생하는 대표적 산업기계로 인식되고 있다. 이러한 고소음 기계류에 의한 소음원의 차폐 또는 저감을 위한 공학적 대응능력을 향상시켜 소음을 저감하고 국제적 기계류의 안전규제에 대하여 능동적인 대처능력을 확보하기 위함이 본 연구의 목적이다.

• 방재와보험
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• s.109
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• pp.70-71
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• 2005

현대인에게 숙면은 다음 날 일상생활의 활력을 주는 필수요소이다. 코골이는 숙면을 방해해 일상생활의 능률이 떨어뜨릴 뿐 아니라 같이 잠자리에 드는 가족도 덩달아 숙면을 취할 수 없게 만들고 심지어는 코고는 소리에 의해 소음성 난청이 유발되기도 한다.

• Journal of Life Science
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• v.14 no.6 s.67
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• pp.913-919
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• 2004

Mitochondrial DNA mutations have been reported in recent years in association with sensorineural hering loss. The purpose of this study is to identify the association between the noise-induced sensorineural hearing loss and the A to G mutation at nucleotide 3243, 1555, 7445 of mitochondrial DNA. Study subjects were established by history and chart review, and audiological and clinical data were obtained. Blood was sampled from 214 normal controls, 102 noise-induced hearing loss, and 28 sensorineural hearing loss. The DNA of these individuals were extracted, and mitochondrial DNA fragments were analyzed by polymerase chain reaction. Subsequently, the coding sequence of mitochondrial DNA 3243, 1555, 7445 were sequenced, and compared to the normal sequence, and all sequence variations were analyzed by restriction enzymes. Mitochondrial DNA mutations $(3243A{\rightarrow}G,\;1555A{\rightarrow}4G,\;7445A{\rightarrow}G)$ were not detected by polymerase chain reactions in any patients with noise-induced hearing loss, sensorineural hearing loss, and normal controls. The DNA sequencing of PCR products did not revealed an A to G substitution at nucleotide 3243, 1555, 7445 of mitochondrial DNA. The noise-induced sensorineural hearing loss was not associated with mitochondrial DNA mutation $(3243A{\rightarrow}G,\;1555A{\rightarrow}4G,\;7445A{\rightarrow}G)$.

• Transactions of the Korean Society for Noise and Vibration Engineering
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• v.25 no.1
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• pp.5-12
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• 2015

According to the analysis of special medical examination and work environment monitoring data, the rate of C1 and D1 on noise hazard exceeded 90 % among those of total hazardous factors. The rate of company exceeding noise exposure limit was also more than 90 %. The analysis result shows that main ages diagnosed with C1 and D1 was age of 50s. The majority scale company having workers diagnosed with C1 and D1 was the companies employing 5~49 workers. Types of industries which have a large number of companies exceeding noise exposure limit were automobile and trailer manufacturing, metal processing industry and primary metal manufacturing. A large number of work processes exceeding noise exposure limit were forming and processing work, cutting and bending work and grinding. To reduce the number of company exceeding noise exposure limit, the reduction counterplan should be focused on the type of industry and the work process which exceeded noise exposure limit frequently. However, the reduction counterplan is preemptively necessary to the type of industry and the work process which exceeded noise exposure limit consecutively if the purpose of reduction counterplan is not to merely reduce the number of company exceeding noise exposure limit but to abate workers' suffering from noise.