• Journal of Veterinary Clinics
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• v.30 no.2
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• pp.111-114
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• 2013

A 4-year-old spayed female Miniature Pinscher was presented with a 3.5-year history of elevated liver enzymes level. Continuous elevation of liver enzymes was found on repeated blood examinations and the dog was referred for further evaluation. The abdominal ultrasonography revealed that the gallbladder could not be visualized in any plane. The absence of gallbladder was suspected on ultrasonography. Computed tomography (CT) confirmed gallbladder agenesis (GBA). This is the first reported case of diagnosis of GBA in Miniature Pinscher by using CT.

• Journal of Veterinary Clinics
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• v.24 no.3
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• pp.410-413
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• 2007

A four-year old, female Miniature Pinscher was presented with intermittent coughing. Thoracic radiography and ultrasonography revealed the consolidation of left cranial lung lobe surrounded by pleural effusion and the extraction of left lung lobe was performed through thoracotomy. This case has the clinical significance because lung lobe torsion was diagnosed by non-cardiac ultrasonographic examination, therefore surgical correction was performed without delay and led good outcome. Moreover, the authors believe the present case was the first report on lung lobe torsion in Miniature Pinscher dog.

• Journal of Veterinary Clinics
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• v.23 no.3
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• pp.349-354
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• 2006

A 9-month-old, female Miniature Pinscher(MP) dog weighing 1.97kg was presented because of periodic syncopal episode for 5 months. This case was diagnosed as primary dilated cardiomyopathy based on respiratory distress history, weak femoral pulse, generalized cardiomegaly, pulmonary edema, marked dilation of left atrium(LA) and left ventricle(LV), decreased wall thickness of LV and interventricular septum(IVS), increased EPSS in echocardiography, and young age of onset in the absence of other cardiovascular disorders. The patient was stabilized by application of diuretics(Furosemide, 2 mg/kg, SC, q 1 hr) and venodilator(Nitroglycerine patch, 0.5 mg/kg, q 12 hrs). Clinical signs were improved with medical management of positive inotropic vasodilator(Pimobendan, 0.2 mg/kg, PO, q 12 hrs) and angiotensin-converting enzyme(ACE) inhibitor(benazepril, 0.5 mg/kg, PO, q 12 hrs), potassium gluconate gel(2 mEq/dog, PO, q 12 hrs) and, L-carnitine(50 mg/kg, PO, q 12 hrs). The dog still maintains stable clinical status 10 months after the first visit. We report the rare case of DCM in small breed dog, which corresponds to the diagnosis and treatment of typical DCM in large breed dog.

• Journal of Veterinary Clinics
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• v.31 no.3
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• pp.241-245
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• 2014

A miniature pinscher and two Maltese dogs were present with dyspnea, and radiography and computed tomography (CT) were performed. All dogs were diagnosed with giant bullous emphysema of the massive, hyperexpanded, and hypoattenuating bulla causing contralateral mediastinal shift. Giant bullous emphysema, the rarest form of bullous lung disease, is defined as a bulla that fills more than 30% of the hemithorax, and multi-detector CT scanning could provide the useful information to confirm a diagnosis of giant bullous emphysema in older dogs. The radiographic and computed tomographic features for giant bullous emphysema were described.

• Korean Journal of Veterinary Research
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• v.44 no.4
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• pp.621-624
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• 2004

A 13-year-old female Miniature Pincher was euthanized after suffering from respiratory insufficiency and seizure. At necropsy, firm tan masses approsimately 4 to 5 cm in diameter were noted at the right caudal love and left cranial lobe of the lung. On cross sections of the cerebellum and the verebrum, several compressive firm round masses, 0.5 to 1 cm in diameter, were noted. similar looking nodules were also present in the mediastinal lymph nodes. Histopathologically, the neoplastic masses consisted of cuboidal shape epithelial cells that form glandular structure. The neoplastic cells have hyperchromatic nuclei with prominent nucleoli and moderate amount of cytoplasm. The degree of mitosis was high (4-6/400X). Multiple areas of necrosis, hemorrhage, and tumor emboli were also noted. Metastasis to the mediastinal lymph nodes, pancreas, and brain was confirmed. Based on the gross and histopathologic examinations, a diagnosis of primary metastatic pulmonary adenocarcinoma was made. Clinicaly recognized neurological signs were therefore due to brain metastasis.