• Journal of the Korean Society of Propulsion Engineers
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• v.16 no.5
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• pp.67-73
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• 2012

Failure mechanisms for a solid propellant were discussed, and the stip-yield and the inherent flaw models were applied to them. Then, failure assessment diagram methods were investigated by means of modified two models. Fracture toughness tests using CCT(center-cracked tension) specimens made of a solid propellant were conducted to generate the failure assessment diagrams. The failure assessment diagram of the inherent flaw model was normalized in order to compare with that of the modified strip-yield model. From the comparison of two failure assessment diagrams, it was found that the failure assessment diagram of the modified inherent flaw model more conservatively assesses the failure than that of the modified strip-yield model.

• Proceedings of the Korean Society of Propulsion Engineers Conference
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• 2012.05a
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• pp.532-538
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• 2012

Failure mechanisms for a highly filled elastomer were discussed, and the stip-yield and the inherent flaw models were applied to them. Then, failure assessment diagram methods were investigated by means of modified two models. Fracture toughness tests using CCT(center-cracked tension) specimens made of a highly filled elastomer were conducted to generate the failure assessment diagrams. The failure assessment diagram of the inherent flaw model was normalized in order to compare with that of the modified strip-yield model. From the comparison of two failure assessment diagrams, it was found that the failure assessment diagram of the modified inherent flaw model more conservatively assesses the failure than that of the modified strip-yield model.

• Journal of KIISE
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• v.42 no.11
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• pp.1349-1360
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• 2015

In order to improve software quality, it is necessary to efficiently and effectively remove software defects in source codes. In the development field, defects are removed according to removal ratio or severity of defects. There are several studies on the removal of defects based on software quality attributes, and several other studies have been done to improve the software quality using classification of the severity of defects, when working on projects. These studies have thus far been insufficient in terms of identifying if there exists relationships between defects or whether any type of defect is more important than others. Therefore, in this study, we collected various types of software defects, standards organization, companies, and researchers. We modeled the defects types using an ANP model, and developed the weighted severities of the defects types, with respect to the general application software, using the ANP model. When general application software is developed, we will be able to use the weight for each severity of defect type, and we expect to be able to remove defects efficiently and effectively.

• Journal of Korea Game Society
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• v.2 no.1
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• pp.55-61
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• 2002

유 무선환경에서 침입 패턴이 다양화되고, 모바일의 편리성이 강조되면서 네트워크 대역폭이 다양한 전송 기반을 요구하고 있다. 그리고 무선기반의 자료가 급증하고 있어, 무선환경에서의 침입탐지시스템 성능에 문제가 될 수 있다. 그러므로 게이트웨이를 근간으로 한 무선전송 기반을 보호하고, 컴퓨터 운영체제 상에 내재된 보안상의 결함을 보호하기 위하여 기존의 운영체제 내에 보안 기능을 추가한 운영체제이며, 커널의 핵심 부분을 인지하여 무선기반의 시스템 사용자에 대한 식별 및 인증, 강제적 접근 통제, 임의적 접근 통제, 해킹 대응 등의 보안 기능 요소들을 갖추게 하여 보안성에 강한 시스템 유지를 요구한다. 그러므로 감시대상의 정보를 미리 알고, WAP환경하에서 감시대상에 유효한 침입패턴만을 검사하도록 침입패턴 데이터베이스를 분리하는 모델을 제시하여, 이러한 문제점에 대한 해결책을 제시하고자 하였다. 따라서 기존 침입탐지시스템의 패턴 데이터베이스를 분석하였고, 이를 적절히 분리하여 이를 다시 운영체제에 반영하는 기법이다. 그리고 이를 제시한 모델을 검증하고자 실제 구현과 실험을 통해 이를 검증하였다.

• Journal of Life Science
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• v.26 no.3
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• pp.376-386
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• 2016

Apoptosis induction has been proposed as an efficient mechanism by which malignant tumor cells can be removed following chemotherapy. The intrinsic mitochondria-dependent apoptotic pathway is frequently implicated in chemotherapy-induced tumor cell apoptosis. Since DNA-damaging agent (DDA)-induced apoptosis is mainly regulated by the tumor suppressor protein p53, and since more than half of clinical cancers possess inactive p53 mutants, microtubule-damaging agents (MDAs), of which apoptotic effect is mainly exerted via p53-independent routes, can be promising choice for cancer chemotherapy. Recently, we found that the apoptotic signaling pathway induced by MDAs (nocodazole, 17α-estradiol, or 2-methoxyestradiol) commonly proceeded through mitotic spindle defect-mediated prometaphase arrest, prolonged Cdk1 activation, and subsequent phosphorylation of Bcl-2, Mcl-1, and Bim in human acute leukemia Jurkat T cells. These microtubule damage-mediated alterations could render the cellular context susceptible to the onset of mitochondria-dependent apoptosis by triggering Bak activation, Δψm loss, and resultant caspase cascade activation. In contrast, when the MDA-induced Bak activation was inhibited by overexpression of anti-apoptotic Bcl-2 family proteins (Bcl-2 or Bcl-xL), the cells in prometaphase arrest failed to induce apoptosis, and instead underwent mitotic slippage and endoreduplication cycle, leading to formation of populations with 8N and 16N DNA content. These data indicate that cellular apoptogenic mechanism is critical for preventing polyploid formation following MDA treatment. Since the formation of polyploid cells, which are genetically unstable, may cause acquisition of therapy resistance and disease relapse, there is a growing interest in developing new combination chemotherapies to prevent polyploidization in tumors after MDA treatment.