Green Tea (-)EGCG Induces the Apoptotic Death of Lung Cancer Cells via Activation of c-Jun N-terminal Kinase 1 and Activating Protein-1 |
박지선
(전북대학교 식품영양학과)
신미경 (원광대학교 식품영양학과) 손희숙 (전북대학교 식품영양학과) 박래길 (원광대학교 의과대학 미생물학교실) 김명선 (원광대학교 의과대학 미생물학교실) 정원훈 (원광보건대학) |
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Identification of an oncoprotein-and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain
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DOI ScienceOn |
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Effect of (-)-epigallocatechin gallate on leukemic blast cells from patients with acute myeloblastic leukemia
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Cell cycle dysregulation by green tea polyphenol epigallocatechin-3-gallate
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DOI ScienceOn |
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Induction of programmed cell death(apoptosis) in human leukemia cells by catechins compounds
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Cancer-preventive effects of drinking tea among a japanese population
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DOI ScienceOn |
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Polyphenols as cancer chemopreventive agents
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The stress-activated protein kinase subfamily of c-Jun kinases
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DOI ScienceOn |
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Angiogenesis inhibited by drinking tea
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DOI ScienceOn |
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Role of the stress-activated/c-Jun NH2-terminal protein kinase pathway in the cellular response to adriamycin and other chemotherapeutic drugs
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DOI ScienceOn |
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Hypotensive effect of tea catechins on blood pressure of rats
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Why drinking green tea could prevent cancer
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DOI ScienceOn |
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Apoptosis in the immune system
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DOI ScienceOn |
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NF-kapa B-mediated chemoresistance in breast cancer cells
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DOI ScienceOn |
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Opiates promote Tcell apoptosis through JNK and caspase pathway
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DOI ScienceOn |
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Effect of green tea catechins on ploasma cholesterol level in cholesterol-fed rats
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DOI |
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Apoptosis signal by death receptors
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DOI ScienceOn |
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EGCG, a major component of green tea, inhibits tumour growth by inhibiting VEGF induction in human colon carcinoma cells
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Dopamine induces apoptosis through an oxidation-involved SAPK/JNK activation pathways
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DOI ScienceOn |
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Post-initiation inhibitory effects of green tea catechins on 7, 12-dimethylbenz[a]anthracene-induced mammary gland carcinogenesis in female sprague-Dawley rats
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DOI ScienceOn |
21 |
A novel SAPK/JNK kinase, MKK7,stimulated by TNF-α and cellular stresses
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DOI |
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Cancer inhibition by green tea
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DOI ScienceOn |
23 |
Protective effects of morphine in peroxynitrite-induced apoptosis of primary rat neonatol astrocytes:potential involvement of G protein and phosphatidylinositol 3-ki-nase(PI 3-kinase)
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DOI ScienceOn |
24 |
Programmed cell death:apoptosis and oncogenesis
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DOI ScienceOn |
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Apoptosis:the physiologic pathway of cell death
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26 |
Fas-independent apoptosis induced by UVC in p53-mutated human epithelial tumor A431 cells through activation of caspase-8 and JNK/SAPK
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DOI ScienceOn |
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Programmed cell death and the control of cell survival
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DOI |
28 |
Platelet aggregation inhibitory activity of tea extracts
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DOI |
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Reduction of blood glucose levels by tea catechin
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DOI |
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Green tea constituent epigallocatechin-3-gallate and induction of apoptosis and cell cycle arrest in human carcinoma cells
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31 |
Activation of caspase cascades in Korean mistletoe(Viscum album var. coloratum) lectin-Ⅱ-induced apoptosis of human myeloeukemic U 937 cells
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DOI ScienceOn |
32 |
Antibacterial activity of tea polyphenols against phytopathogenic bacteria
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DOI |
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Size control:the regulation of cell numbers in animal development
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DOI ScienceOn |
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Epigallocathechin-3 gallate selectively inhibits the PDGF-BB-induced intracellular signaling transduction pathway in vascular smooth muscle cells and inhibits transformation of sistransfected NIH 3T3 fibroblasts and human glioblastoma cells(A172)
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DOI |
35 |
Induction of apoptosis in prostate cancer cell lines by the green tea component, (-)-epigallocatechin-3-gallate
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DOI ScienceOn |
36 |
Bax-induced apoptosis as a nover gene therapy approach for carcinoma of the cervix
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DOI ScienceOn |
37 |
Induction of apoptosis in human stomach cancer cells by green tea catechins
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38 |
Signaling transduction by the c-Jun N-terminal kinase
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Requirement for ceramide-initiated SAPK/JNK signalling in stress-induced apoptosis
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DOI ScienceOn |
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Chemotherapy-induced apoptisis
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DOI |
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Apoptosis in the pathogenesis and treatment of disease
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MAPKs:new JNK expands the group
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43 |
Caenorhabditis elegans EGL-1 disrupts the interaction of CED-9 with CED-4 and promotes CED-3 activation
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DOI ScienceOn |
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Preferential expansion and survival of B lymphocytes based on VH framework 1 and framework 3 expression:"positive" selection in apendix of normal and VH-mutant rabbits
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DOI ScienceOn |