• 한국음향학회지
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• 제37권6호
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• pp.451-456
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• 2018

최근 장거리 수중음향통신 연구가 활발하게 진행되고 있다. 장거리 수중음향통신 연구를 위한 해상실험은 해양환경의 영향을 크게 받고 높은 비용과 많은 시간이 소요된다. 따라서 장거리 환경에서 수중음향통신 시스템의 성능을 예측하고 검증하기 위한 시뮬레이터에 대한 연구가 필요하다. 본 논문에서는 장거리 수중음향통신 시뮬레이터의 개발을 목적으로 중장거리 다중송신환경에서 수중음향통신 시뮬레이터로서 VirTEX(Virtual Time series EXperiment)의 성능을 분석하고, 해상 실험 결과와 비교 검증하였다. 송신 신호는 탐침을 위한 Chirp신호와 16QAM 방식으로 변조된 통신 신호를 사용하였다. VirTEX를 통해 수신 신호를 모의하고 다중송신환경에 의해 발생하는 채널 간 간섭을 적응 수동형 시역전을 이용하여 보상하여 통신 성능을 분석하였다. 또한 2005년 7월 이탈리아의 Elba island 북쪽 해역에서 수행한 FAF05(Focused Acoustic Field 2005) 실험 데이터와 비교하여 이를 검증하였다.

• JSTS:Journal of Semiconductor Technology and Science
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• 제16권4호
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• pp.470-480
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• 2016

The reversal of a silicon chip to find out its security structure is common and possible at the present time. Thanks to reversing, it is possible to use a probing attack to obtain useful information such as personal information or a cryptographic key. For this reason, security-related blocks such as DES (Data Encryption Standard), AES (Advanced Encryption Standard), and RSA (Rivest Shamir Adleman) engines should be located in the lower layer of the chip to guard against a probing attack; in this regard, the addition of a silicon-surface-protection layer onto the chip surface is a crucial protective measure. But, for manufacturers, the implementation of an additional silicon layer is burdensome, because the addition of just one layer to a chip significantly increases the overall production cost; furthermore, the chip size is increased due to the bulk of the secure logic part and routing area of the silicon protection layer. To resolve this issue, this paper proposes a practical silicon-surface-protection method using a metal layer that increases the security level of the chip while minimizing its size and cost. The proposed method uses a shift register for the alternation and variation of the metal-layer data, and the inter-connection area is removed to minimize the size and cost of the chip in a more extensive manner than related methods.

• Journal of Ginseng Research
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• 제48권4호
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• pp.384-394
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• 2024

Background: Herpes simplex virus type 1 (HSV-1), known to latently infect the host's trigeminal ganglion, can lead to severe herpes encephalitis or asymptomatic infection, potentially contributing to neurodegenerative diseases like Alzheimer's. The virus generates reactive oxygen species (ROS) that significantly impact viral replication and induce chronic inflammation through NF-κB activation. Nuclear factor E2-related factor 2 (Nrf2), an oxidative stress regulator, can prevent and treat HSV-1 infection by activating the passive defense response in the early stages of infection. Methods and results: Our study investigated the antiviral effects of ginsenoside Rg5, an Nrf2 activator, on HSV-1 replication and several host cell signaling pathways. We found that HSV-1 infection inhibited Nrf2 activity in host cells, induced ROS/NF-κB signaling, and triggered inflammatory cytokines. However, treatment with ginsenoside Rg5 inhibited ROS/NF-κB signaling and reduced inflammatory cytokines through NRF2 induction. Interestingly, the Nrf2 inhibitor ML385 suppressed the expression of NAD(P)H quinone oxidoreductase 1(NQO1) and enhanced the expression of KEAP1 in HSV-1 infected cells. This led to the reversal of VP16 expression inhibition, a protein factor associated with HSV-1 infection, thereby promoting HSV-1 replication. Conclusion: These findings suggest for the first time that ginsenoside Rg5 may serve as an antiviral against HSV-1 infection and could be a novel therapeutic agent for HSV-1-induced neuroinflammation.