• The Journal of the Institute of Internet, Broadcasting and Communication
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• v.13 no.2
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• pp.137-142
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• 2013

Direction of arrival is estimating for desire signal direction among received signal on antenna in spatial. In this paper, we were an estimation a receiving signal direction of arrival using multi beam forming in radar. We proposed, by signal direction of arrival estimation method, an algorithm which combine spatial correlation matrix weight value and beam steering algorithm in this paper. Through simulation, we were analysis a performance to compare general algorithm and proposal algorithm. In direction of arrival estimation, proposed algorithm is effectivity to decrease processing time because it is not doing an eigen decomposition. We showed that proposal algorithm improve more target estimation than general algorithm.

• Journal of the Korea Society of Computer and Information
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• v.13 no.4
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• pp.39-44
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• 2008

Wireless channel exists interference by multipath a component. Adaptation array antenna that remove this interference a component forms null point about interference signal and maximizes gains about target signal. If target signal and correlative coherent interference signal are received, there is problem that is removed from arrangement output to target signal. And, adaptation array antenna is shortcoming that is sensitive in directivity error. Therefore, in this paper, introduce each existing algorithm to solve directivity error about coherent interference, and proposed beam forming technique that minimize degree of freedom loss and damage because analyzes the problem and reduces coherent interference and directivity error.

• BMB Reports
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• v.38 no.3
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• pp.334-342
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• 2005

Recently, the existence of T cells with dual T cell receptor (TCR) in the immune system is generally accepted, while it has been controversial whether signals through one TCR would affect the functions of the other. In this study T cells expressing two different TCR were obtained from cross-hybrids of LCMV and AND TCR transgenic mice specific for the gp33 and peptide fragment of PCC (fPCC), respectively. Peptide stimulation demonstrated that the dual TCR T cells functioned independently in an antigen-specific manner. To examine whether the tolerance targeted for the one TCR affects the responsiveness of the other, the cross-hybrids were treated with gp33. Although T cells from F1 mice were rendered anergenic to gp33, no functional changes to fPCC were observed in terms of cellular proliferation and IL-2 secretion, suggesting that the dual TCR T cells remained reactive to fPCC. We therefore propose that signaling through the TCR is receptor-specific and 'negative dominance' of one TCR by tolerance induction is not applicable in this dual TCR system.

• The Journal of Korea Institute of Information, Electronics, and Communication Technology
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• v.9 no.3
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• pp.291-297
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• 2016

In this paper, we are comparative analysis of exit algorithm and proposal algorithm for desired target direction of arrival estimation in correlation signal system. Proposed algorithm in this paper is to decrease target direction of arrival an estimation error probability using bayesian, maximum posterior, and MUSIC algorithm in order to decrease direction of arrival error probability as optimize and use linear constrained minimum variance to update weight value. Through simulation, we were comparative analysis proposed algorithm and exit MUSIC algorithm. In case SNR is 10dB and antenna element arrays are 9 and 12, We show the superior performance of the proposed method relative to the class method to decrease of signal estimation error probability about 11% and 13%, respectively.

• Molecules and Cells
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• v.44 no.11
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• pp.795-804
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• 2021

Memory T (T_M) cells play an important role in the long-term defense against pathogen reinvasion. However, it is still unclear how these cells receive the crucial signals necessary for their longevity and homeostatic turnover. To understand how T_M cells receive these signals, we infected mice with lymphocytic choriomeningitis virus (LCMV) and examined the expression sites of neural cadherin (N-cadherin) by immunofluorescence microscopy. We found that N-cadherin was expressed in the surroundings of the white pulps of the spleen and medulla of lymph nodes (LNs). Moreover, T_M cells expressing high levels of killer cell lectin-like receptor G1 (KLRG1), a ligand of N-cadherin, were co-localized with N-cadherin⁺ cells in the spleen but not in LNs. We then blocked N-cadherin in vivo to investigate whether it regulates the formation or function of T_M cells. The numbers of CD127^hiCD62L^hi T_M cells in the spleen of memory P14 chimeric mice declined when N-cadherin was blocked during the contraction phase, without functional impairment of these cells. In addition, when CD127^loKLRG1^hi T_M cells were adoptively transferred into anti-N-cadherin-treated mice compared with control mice, the number of these cells was reduced in the bone marrow and LNs, without functional loss. Taken together, our results suggest that N-cadherin participates in the development of CD127^hiCD62L^hi T_M cells and homing of CD127^loKLRG1^hi T_M cells to lymphoid organs.