• Title/Summary/Keyword: Damage cause

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Dispute Mediation Cases and Suggestions for Calculating Compensation for Dust Damage (먼지 피해의 환경분쟁조정 사례 분석과 배상액 산정안 제언)

  • Jeong-Ho Park
    • Journal of Environmental Science International
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    • v.32 no.10
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    • pp.693-701
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    • 2023
  • In this study, we analyzed 82 dust damage dispute mediation cases over the past 5 years and evaluated cases where the probability of damage was verified through dust concentration measurement, modeling prediction, and chemical composition analysis. The cause of dust damage was a construction site, which accounted for most of the damage (97%), and was closely related to the distance from the construction site, total floor area of the construction site, and construction duration. Compensation was decided in only 33% of dust damage cases, and in only 6% (five cases) were damages determined using scientific techniques such as dust measurement, and forecasting. The main criteria for determining compensation were whether administrative measures were taken and evidence of damage in the form of videos and photos. In the future, measuring or model for the amount of dust damage is necessary to determine whether the limit has been exceeded and to revise the standard for calculating compensation through various lines of evidence of dust damage.

A Research of Definition and Treatment of Dizziness in the Books on Cold Damage (상한문헌에 나타난 현훈의 정의와 치료법에 대한 연구)

  • Kim, Sang-Un;Jung, Hyun-Jong
    • The Journal of the Society of Korean Medicine Diagnostics
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    • v.18 no.3
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    • pp.149-174
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    • 2014
  • Objectives to study definition and treatment of dizziness in the books of Cold damage which are classified as symptoms of all time. Methods 1. Quote provisions related to dizziness in "Sanghanlon(傷寒論)" 2. Among the books of Cold damage which are classified as symptoms, select 19 literatures on dizziness with table of contents and collect data and categorize in two perspectives on theories and disease pattern. 3. Compare and draw a chart all data collected in above methods. Results & Conclusions 1. In the books on Cold damage, dizziness is expressed in 2 ways such as head dizziness(頭眩) and fainting(鬱冒). fainting is much more several symptoms than head dizziness and it is the difference that it has mental confusion. 2. The cause of head dizziness after promoting sweating, vomiting and purgation is that source qi(元氣) of upper energizer(上焦) is deficiency, and cause of head dizziness before using method of treatment is wind(風), heat(熱), phlegm(痰), blood deficiency(血虛) and etc. 3. Main cause of fainting is that deficiency is getting severe so that cold invades, and symptoms are blood deficiency, after giving birth and excess pattern of fire and heat. 4. Remedies for head dizziness are using Yeonggyechulgam-tang(苓桂朮甘湯), jinmu-tang(眞武湯), sosiho-tang(小柴胡湯), sagunja-tang(四君子湯), samul-tang(四物湯) and etc. 5. Insamsambaek-tang (人蔘三白湯) and Sayeok-tang(四逆湯) are used for deficiency pattern of fainting, and Dojeok-san(導赤散), Daeseunggi-tang(大承氣湯), and Hwangryeonhaedok-tang(黃連解毒湯) are used for excess pattern(實證).

Damage rate assessment of cantilever RC walls with backfill soil using coupled Lagrangian-Eulerian simulation

  • Javad Tahamtan;Majid Gholhaki;Iman Najjarbashi;Abdullah Hossaini;Hamid Pirmoghan
    • Geomechanics and Engineering
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    • v.36 no.3
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    • pp.231-245
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    • 2024
  • In recent decades, the protection and vulnerability of civil structures under explosion loads became a critical issue in terms of security, which may cause loss of lives and structural damage. Concrete retaining walls also restrict soils and slopes from displacements; meanwhile, intensive temporary loading may cause massive damage. In the current study, the modified Johnson-Holmquist (also known as J-H2) material model is implemented for concrete materials to model damages into the ABAQUS through user-subroutines to predict the blasting-induced concrete damages and volume strains. For this purpose, a 3D finite-element model of the concrete retaining wall was conducted in coupled Eulerian-Lagrangian simulation. Subsequently, a blast load equal to 500 kg of TNT was considered in three different positions due to UFC 3-340-02. Influences of the critical parameters in smooth blastings, such as distance from a free face, position, and effective blasting time, on concrete damage rate and destroy patterns, are explored. According to the simulation results, the concrete penetration pattern at the same distance is significantly influenced by the density of the progress environment. The result reveals that the progress of waves and the intensity of damages in free-air blasting is entirely different from those that progress in a dense surrounding atmosphere such as soil. Half-damaged elements in air blasts are more than those of embedded explosions, but dense environments such as soil impose much more pressure in a limited zone and cause more destruction in retaining walls.

Effect of Chronic Ethanol Administration on Oxidative Stress and Cellular Defence System in Rat Myocardium (에탄올 장기 투여에 의한 쥐 심근조직의 산화적 스트레스와 생체내 항산화 효소활성의 변화)

  • 오세인
    • Journal of Nutrition and Health
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    • v.29 no.7
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    • pp.721-728
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    • 1996
  • The level of oxidative tissue damage caused by free radicals generated from ethanol oxidation was determined in the myocardium of chronic ethanol fed-rats and the protective action of various radical scavenging enzymes was monitored, also. Adult male Sprague-Dawley rats were given ethanol in an amount of 36% of total calories via Lieber-DeCarli liquid diet for 6 weeks. Control group was pair-fed with the diet containing isocaloric amount of dextrin-maltose instead of ethanol. Chronic ethanol administration resulted in the increased amount of myocardial thiobarbituric acid reactive substance(TBARS), th parameter of lipid peroxidation, under our experimental condition. Chronic ethanol ingestion did not cause any change in activities of either glutathione peroxidase or glutathione reductase and glucose-6-phosphate dehydrogenase were decreased after ethanol treatment. Therefore, chronic ethanol administration seemed to cause considerble changes in cellular defense function against oxidative tissue damage in rat myocardium through glutathione utilizing system and radical generation system. However the ultimate net result of chronic ethanol inestion on the myocardium of rat was the oxidative tissue damage revealed by increased TBARS content.

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Damage Cause Analysis for GCB of 154kv Power Plant (154kV 전력계통 가스차단기(GCB) 소손원인 조사분석)

  • Lee, Eun-Chun;Shin, Gang-Wook;Hong, Sung-Taek
    • Proceedings of the KIEE Conference
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    • 2000.07a
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    • pp.425-427
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    • 2000
  • The purpose of this study is to find the cause of the damage to the GCB(Gas Circuit Breaker), since the GCB of the 154kV power plant at the Hap-Cheon Dam has already been damaged twice. We researched the characteristics of this class of circuit breaker and the possible causes for this type of damage to the GCB using the FTA(Fault Tree Analysis) Method. We studied the optimal maintenance method of the GCB, the stability analysis and power serge protection of Hap-Cheon Dam, and the power serge and fault list of the power transmission line to help prevent a reoccurrence of the problem.

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A Case Study on the Assessment of Damaged Cause for the Damaged Reinforced Concrete Pier

  • Chai, Won-Kyu;Kim, Kwang-Il;Son, Young-Hyun
    • International Journal of Safety
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    • v.10 no.1
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    • pp.16-21
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    • 2011
  • In this thesis, appearance inspection, compressive strength of concrete test, arrangement of bar inspection, survey, and bearing stress analysis were performed on a damaged coping of reinforced concrete pier to investigate the damage cause. According to the performed a series of inspections, it was found that the coping of pier was damaged during PSC (Pre-stressed Concrete) beam construction. In this thesis, the repair method for damaged pier was studied. The repair procedure used in this thesis was follows : chipping for damaged part, clean by high-pressure, installation of wire mesh, coating of surface hardening, construction of section restoration material, copula grinding, and prevent coating for far-infrared radiation.

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A new damage index for detecting sudden change of structural stiffness

  • Chen, B.;Xu, Y.L.
    • Structural Engineering and Mechanics
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    • v.26 no.3
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    • pp.315-341
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    • 2007
  • A sudden change of stiffness in a structure, associated with the events such as weld fracture and brace breakage, will cause a discontinuity in acceleration response time histories recorded in the vicinity of damage location at damage time instant. A new damage index is proposed and implemented in this paper to detect the damage time instant, location, and severity of a structure due to a sudden change of structural stiffness. The proposed damage index is suitable for online structural health monitoring applications. It can also be used in conjunction with the empirical mode decomposition (EMD) for damage detection without using the intermittency check. Numerical simulation using a five-story shear building under different types of excitation is executed to assess the effectiveness and reliability of the proposed damage index and damage detection approach for the building at different damage levels. The sensitivity of the damage index to the intensity and frequency range of measurement noise is also examined. The results from this study demonstrate that the damage index and damage detection approach proposed can accurately identify the damage time instant and location in the building due to a sudden loss of stiffness if measurement noise is below a certain level. The relation between the damage severity and the proposed damage index is linear. The wavelet-transform (WT) and the EMD with intermittency check are also applied to the same building for the comparison of detection efficiency between the proposed approach, the WT and the EMD.

Recognition of DNA Damage in Mammals

  • Lee, Suk-Hee
    • BMB Reports
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    • v.34 no.6
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    • pp.489-495
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    • 2001
  • DNA damage by UV and environmental agents are the major cause of genomic instability that needs to be repaired, otherwise it give rise to cancer. Accordingly, mammalian cells operate several DNA repair pathways that are not only responsible for identifying various types of DNA damage but also involved in removing DNA damage. In mammals, nucleotide excision repair (NER) machinery is responsible for most, if not all, of the bulky adducts caused by UV and chemical agents. Although most of the proteins involved in NER pathway have been identified, only recently have we begun to gain some insight into the mechanism by which proteins recognize damaged DNA. Binding of Xeroderma pigmentosum group C protein (XPC)-hHR23B complex to damaged DNA is the initial damage recognition step in NER, which leads to the recruitment of XPA and RPA to form a damage recognition complex. Formation of damage recognition complex not only stabilizes low affinity binding of XPA to the damaged DNA, but also induces structural distortion, both of which are likely necessary for the recruitment of TFIIH and two structure-specific endonucleases for dual incision.

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Gamma-Irradiation and Doxorubicin Treatment of Normal Human Cells Cause Cell Cycle Arrest Via Different Pathways

  • Lee, Seong Min;Youn, BuHyun;Kim, Cha Soon;Kim, Chong Soon;Kang, ChulHee;Kim, Joon
    • Molecules and Cells
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    • v.20 no.3
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    • pp.331-338
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    • 2005
  • Ionizing radiation and doxorubicin both produce oxidative damage and double-strand breaks in DNA. Double-strand breaks and oxidative damage are highly toxic and cause cell cycle arrest, provoking DNA repair and apoptosis in cancer cell lines. To investigate the response of normal human cells to agents causing oxidative damage, we monitored alterations in gene expression in F65 normal human fibroblasts. Treatment with ${\gamma}$-irradiation and doxorubicin altered the expression of 23 and 68 known genes, respectively, with no genes in common. Both agents altered the expression of genes involved in cell cycle arrest, and arrested the treated cells in $G_2M$ phase 12 h after treatment. 24 h after ${\gamma}$-irradiation, the percentage of $G_1$ cells increased, whereas after doxorubicin treatment the percentage of $G_2M$ cells remained constant for 24 h. Our results suggest that F65 cells respond differently to ${\gamma}$-irradiation- and doxorubicin-induced DNA damage, probably using entirely different biochemical pathways.

Clinical and Pathological Aspects of Filarial Lymphedema and Its Management

  • Shenoy, R.K.
    • Parasites, Hosts and Diseases
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    • v.46 no.3
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    • pp.119-125
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    • 2008
  • Lymphatic filariasis, transmitted by mosquitoes is the commonest cause of lymphedema in endemic countries. Among 120 million infected people in 83 countries, up to 16 million have lymphedema. Microfilariae ingested by mosquitoes grow into infective larvae. These larvae entering humans after infected mosquito bites grow in the lymphatics to adult worms that cause damage to lymphatics resulting in dilatation of lymph vessels. This earliest pathology is demonstrated in adults as well as in children, by ultrasonography, lymphoscintigraphy and histopathology studies. Once established, this damage was thought to be irreversible. This lymphatic damage predisposes to bacterial infection that causes recurrent acute attacks of dermato-lymphangio-adenitis in the affected limbs. Bacteria, mainly streptococci gain entry into the lymphatics through 'entry lesions' in skin, like interdigital fungal infections, injuries, eczema or similar causes that disrupt integrity of skin. Attacks of dermato-lymphangio-adenitis aggravates lymphatic damage causing lymphedema, which gets worse with repeated acute attacks. Elephantiasis is a late manifestation of lymphatic filariasis, which apart from limbs may involve genitalia or breasts. Lymphedema management includes use of antifilarial drugs in early stages, treatment and prevention of acute attacks through 'limb-hygiene', antibiotics and antifungals where indicated, and physical measures to reduce the swelling. In selected cases surgery is helpful.