1 |
Rastaldo R, Pagliaro P, Cappello S, et al. Nitric oxide and cardiac function. Life Sci 2007;81:779-93.
DOI
ScienceOn
|
2 |
Boo YC. Shear stress stimulates phosphorylation of protein kinase A substrate proteins including endothelial nitric oxide synthase in endothelial cells. Exp Mol Med 2006;38:453.
DOI
|
3 |
Corson MA, James NL, Latta SE, Nerem RM, Berk BC, Harrison DG. Phosphorylation of endothelial nitric oxide synthase in response to fluid shear stress. Circ Res 1996;79:984-91.
DOI
ScienceOn
|
4 |
Shesely EG, Maeda N, Kim HS, et al. Elevated blood pressures in mice lacking endothelial nitric oxide synthase. Proc Natl Acad Sci U S A 1996;93:13176-81.
DOI
|
5 |
Palmer RM, Rees DD, Ashton DS, Moncada S. L-arginine is the physiological precursor for the formation of nitric oxide in endothelium-dependent relaxation. Biochem Biophys Res Commun 1988;153:1251-6.
DOI
ScienceOn
|
6 |
Fozard JR, Part ML. Haemodynamic responses to NG-monomethyl-Larginine in spontaneously hypertensive and normotensive Wistar-Kyoto rats. Br J Pharmacol 1991;102:823-6.
DOI
|
7 |
Hsieh NK, Chang HR, Hu CT, Chen HI. Effects of nitric oxide donor and nitric oxide synthase inhibitor on the resistance, exchange and capacitance functions of the canine intestinal vasculature. Vascul Pharmacol 2008;48:122-8.
DOI
|
8 |
Fitch RM, Vergona R, Sullivan ME, Wang YX. Nitric oxide synthase inhibition increases aortic stiffness measured by pulse wave velocity in rats. Cardiovasc Res 2001;51:351-8.
DOI
ScienceOn
|
9 |
Sakai H, Hara H, Tsai AG, Tsuchida E, Intaglietta M. Constriction of resistance arteries determines l-NAME-induced hypertension in a conscious hamster model. Microvasc Res 2000;60:21-7.
DOI
|
10 |
de Wit C, Schafer C, von Bismarck P, Bolz SS, Pohl U. Elevation of plasma viscosity induces sustained NO-mediated dilation in the hamster cremaster microcirculation in vivo. Pflugers Arch 1997;434:354-61.
DOI
|
11 |
Tsai AG, Acero C, Nance PR, et al. Elevated plasma viscosity in extreme hemodilution increases perivascular nitric oxide concentration and microvascular perfusion. Am J Physiol Heart Circ Physiol 2005;288:H1730-9.
DOI
|
12 |
Cabrales P, Intaglietta M, Tsai AG. Increase plasma viscosity sustains microcirculation after resuscitation from hemorrhagic shock and continuous bleeding. Shock 2005;23:549-55.
|
13 |
Chatpun S, Cabrales P. Cardiac mechanoenergetic cost of elevated plasma viscosity after moderate hemodilution. Biorheology 2010;47:225-37.
|
14 |
Cabrales P, Tsai AG. Plasma viscosity regulates systemic and microvascular perfusion during acute extreme anemic conditions. Am J Physiol Heart Circ Physiol 2006;291:H2445-52.
DOI
|
15 |
Cabrales P, Tsai AG, Intaglietta M. Increased plasma viscosity prolongs microhemodynamic conditions during small volume resuscitation from hemorrhagic shock. Resuscitation 2008;77:379-86.
DOI
|
16 |
Pacher P, Nagayama T, Mukhopadhyay P, Batkai S, Kass DA. Measurement of cardiac function using pressure-volume conductance catheter technique in mice and rats. Nat Protoc 2008;3:1422-34.
DOI
ScienceOn
|
17 |
Chatpun S, Cabrales P. Exogenous intravascular nitric oxide enhances ventricular function after hemodilution with plasma expander. Life Sci 2012;90:39-46.
DOI
|
18 |
Chatpun S, Cabrales P. Effects of plasma viscosity modulation on cardiac function during moderate hemodilution. Asian J Transfus Sci 2010;4:102-8.
DOI
|
19 |
Amezcua JL, Palmer RM, de Souza BM, Moncada S. Nitric oxide synthesized from L-arginine regulates vascular tone in the coronary circulation of the rabbit. Br J Pharmacol 1989;97:1119-24.
DOI
ScienceOn
|
20 |
Pabla R, Curtis MJ. Effects of NO modulation on cardiac arrhythmias in the rat isolated heart. Circ Res 1995;77:984-92.
DOI
|
21 |
Rees DD, Palmer RM, Schulz R, Hodson HF, Moncada S. Characterization of three inhibitors of endothelial nitric oxide synthase in vitro and in vivo. Br J Pharmacol 1990;101:746-52.
DOI
ScienceOn
|
22 |
Pfeiffer S, Leopold E, Schmidt K, Brunner F, Mayer B. Inhibition of nitric oxide synthesis by NG-nitro-L-arginine methyl ester (L-NAME): requirement for bioactivation to the free acid, NG-nitro-L-arginine. Br J Pharmacol 1996;118:1433-40.
DOI
|
23 |
Crystal GJ, Zhou X, Halim AA, Alam S, El-Orbany M, Salem MR. Nitric oxide does not modulate whole body oxygen consumption in anesthetized dogs. J Appl Physiol (1985) 1999;86:1944-9.
DOI
|
24 |
Ward JE, Angus JA. Acute and chronic inhibition of nitric oxide synthase in conscious rabbits: role of nitric oxide in the control of vascular tone. J Cardiovasc Pharmacol 1993;21:804-14.
DOI
|
25 |
Bryan S, Alexander-Lindo R, McGrowder D. The effect of nitric oxide inhibitors and s-nitrosothiols on hemodynamic parameters in an animal model. Open Access Animal Physiology 2011;3:1-8.
|
26 |
Biwer LA, Broderick TL, Xu H, Carroll C, Hale TM. Protection against LNAME-induced reduction in cardiac output persists even after cessation of angiotensin-converting enzyme inhibitor treatment. Acta Physiol (Oxf) 2013;207:156-65.
DOI
|
27 |
Herring N, Paterson DJ. Endothelial nitric oxide synthase and heart rate. Circulation 2002;106:e5; author reply e5.
DOI
|
28 |
Widdop RE, Gardiner SM, Kemp PA, Bennett T. The influence of atropine and atenolol on the cardiac haemodynamic effects of NG-nitro-L-arginine methyl ester in conscious, Long Evans rats. Br J Pharmacol 1992;105:653-6.
DOI
|
29 |
Chen Y, Traverse JH, Du R, Hou M, Bache RJ. Nitric oxide modulates myocardial oxygen consumption in the failing heart. Circulation 2002;106:273-9.
DOI
|
30 |
Crystal GJ, Zhou X. Nitric oxide does not modulate the increases in blood flow, O2 consumption, or contractility during CaCl2 administration in canine hearts. Cardiovasc Res 1999;42:232-9.
DOI
|