1 |
Xu, W., Ngo, L., Perez, G., Dokmanovic, M. and Marks, P. A. 2006. Intrinsic apoptotic and thioredoxin pathways in human prostate cancer cell response to histone deacetylase inhibitor. Proc. Natl. Acad. Sci. USA. 103, 15540-15545.
DOI
|
2 |
Kannangai, R., Wang, J., Liu, Q. Z., Sahin, F. and Torbenson, M. 2005. Survivin overexpression in hepatocellular carcinoma is associated with p53 dysregulation. Int. J. Gastrointest. Cancer 35, 53-60.
DOI
|
3 |
Kim, D. H., Hwang, J. S., Lee, I. H., Nam, S. T., Hong, J., Zhang, P., Lu, L. F., Lee, J., Seok, H., Pothoulakis, C., Lamont, J. T. and Kim, H. 2016. The insect peptide CopA3 increases colonic epithelial cell proliferation and mucosal barrier function to prevent inflammatory responses in the gut. J. Biol. Chem. 291, 3209-3223.
DOI
|
4 |
Adwan, L. I., Basha, R., Abdelrahim, M., Subaiea, G. M. and Zawia, N. H. 2011. Tolfenamic acid interrupts the de novo synthesis of the beta-amyloid precursor protein and lowers amyloid beta via a transcriptional pathway. Curr. Alzheimer Res. 8, 385-392.
DOI
|
5 |
Altieri, D. C. 2010. Survivin and IAP proteins in cell-death mechanisms. Biochem. J. 430, 199-205.
DOI
|
6 |
Kim, Y. H., Hwang, J. S., Yoon, I. N., Lee, J. H., Lee, J., Park, K. C., Seok, H. and Kim, H. 2021. The insect peptide CopA3 blocks programmed cell death by directly binding caspases and inhibiting their proteolytic activation. Biochem. Biophys. Res. Commun. 547, 82-88.
DOI
|
7 |
Chen, Y., Wang, X., Li, W., Zhang, H., Zhao, C., Li, Y., Wang, Z. and Chen, C. 2011. Sp1 upregulates survivin expression in adenocarcinoma of lung cell line A549. Anat. Rec. 294, 774-780.
DOI
|
8 |
Eslin, D., Sankpal, U. T., Lee, C., Sutphin, R. M., Maliakal, P., Currier, E., Sholler, G., Khan, M. and Basha, R. 2013. Tolfenamic acid inhibits neuroblastoma cell proliferation and induces apoptosis: a novel therapeutic agent for neuroblastoma. Mol. Carcinog. 52, 377-386.
DOI
|
9 |
Kawakami, H., Tomita, M., Matsuda, T., Ohta, T., Tanaka, Y., Fujii, M., Hatano, M., Tokuhisa, T. and Mori, N. 2005. Transcriptional activation of survivin through the NF-kappaB pathway by human T-cell leukemia virus type I tax. Int. J. Cancer 115, 967-974.
DOI
|
10 |
Kim, H., Kokkotou, E., Na, X., Rhee, S. H., Moyer, M. P., Pothoulakis, C. and Lamont, J. T. 2005. Clostridium difficile toxin A-induced colonocyte apoptosis involves p53-dependent p21(WAF1/CIP1) induction via p38 mitogen-activated protein kinase. Gastroenterology 129, 1875-1888.
DOI
|
11 |
Lamers, F., van der Ploeg, I., Schild, L., Ebus, M. E., Koster, J., Hansen, B. R., Koch, T., Versteeg, R., Caron, H. N. and Molenaar, J. J. 2011. Knockdown of survivin (BIRC5) causes apoptosis in neuroblastoma via mitotic catastrophe. Endocr. Relat. Cancer 18, 657-668.
DOI
|
12 |
Nam, S. T., Kim, D. H., Lee, M. B., Nam, H. J., Kang, J. K., Park, M. J., Lee, I. H., Seok, H., Lee, D. G., Hwang, J. S. and Kim, H. 2013. Insect peptide CopA3-induced protein degradation of p27Kip1 stimulates proliferation and protects neuronal cells from apoptosis. Biochem. Biophys. Res. Commun. 437, 35-40.
DOI
|
13 |
Liu, Y. B., Gao, X., Deeb, D., Brigolin, C., Zhang, Y., Shaw, J., Pindolia, K. and Gautam, S. C. 2014. Ubiquitin-proteasomal degradation of antiapoptotic survivin facilitates induction of apoptosis in prostate cancer cells by pristimerin. Int. J. Oncol. 45, 1735-1741.
DOI
|
14 |
Kim, S. J., Kim, I. W., Kwon, Y. N., Yun, E. Y. and Hwang, J. S. 2012. Synthetic Coprisin analog peptide, D-CopA3 has antimicrobial activity and pro-apoptotic effects in human leukemia cells. J. Microbiol. Biotechnol. 22, 264-269.
DOI
|
15 |
Li, D., Hu, C. and Li, H. 2018. Survivin as a novel target protein for reducing the proliferation of cancer cells. Biomed. Rep. 8, 399-406.
|
16 |
Na, X., Zhao, D., Koon, H. W., Kim, H., Husmark, J., Moyer, M. P., Pothoulakis, C. and LaMont, J. T. 2005. Clostridium difficile toxin B activates the EGF receptor and the ERK/MAP kinase pathway in human colonocytes. Gastroenterology 128, 1002-1011.
DOI
|
17 |
Nicolas, M., Noe, V. and Ciudad, C. J. 2003. Transcriptional regulation of the human Sp1 gene promoter by the specificity protein (Sp) family members nuclear factor Y (NF-Y) and E2F. Biochem. J. 371, 265-275.
DOI
|
18 |
Pothoulakis, C. and Lamont, J. T. 2001. Microbes and microbial toxins: paradigms for microbial-mucosal interactions II. The integrated response of the intestine to Clostridium difficile toxins. Am. J. Physiol. Gastrointest. Liver Physiol. 280, G178-183.
DOI
|
19 |
Park, S., Park, J. A., Jeon, J. H. and Lee, Y. 2019. Traditional and novel mechanisms of Heat Shock Protein 90 (HSP90) inhibition in cancer chemotherapy including HSP90 cleavage. Biomol. Ther. 27, 423-434.
DOI
|
20 |
Lu, L. F., Kim, D. H., Lee, I. H., Hong, J., Zhang, P., Yoon, I. N., Hwang, J. S. and Kim, H. 2017. Potassium acetate blocks clostridium difficile toxin A-induced microtubule disassembly by directly inhibiting histone deacetylase 6, thereby ameliorating inflammatory responses in the gut. J. Microbiol. Biotechnol. 26, 693-699.
DOI
|
21 |
Tomicic, M. T., Christmann, M. and Kaina, B. 2010. Topotecan triggers apoptosis in p53-deficient cells by forcing degradation of XIAP and survivin thereby activating caspase-3-mediated Bid cleavage. J. Pharmacol. Exp. Ther. 332, 316-325.
DOI
|
22 |
Papineni, S., Chintharlapalli, S., Abdelrahim, M., Lee, S. O., Burghardt, R., Abudayyeh, A., Baker, C., Herrera, L. and Safe, S. 2009. Tolfenamic acid inhibits esophageal cancer through repression of specificity proteins and c-Met. Carcinogenesis 30, 1193-1201.
DOI
|
23 |
Raj, D., Liu, T., Samadashwily, G., Li, F. and Grossman, D. 2008. Survivin repression by p53, Rb and E2F2 in normal human melanocytes. Carcinogenesis 29, 194-201.
DOI
|
24 |
Small, S., Keerthivasan, G., Huang, Z., Gurbuxani, S., Crispino, J. D. 2010. Overexpression of survivin initiates hematologic malignancies in vivo. Leukemia 24, 1920-1926.
DOI
|
25 |
Xu, R., Zhang, P., Huang, J., Ge, S., Lu, J. and Qian, G. 2007. Sp1 and Sp3 regulate basal transcription of the survivin gene. Biochem. Biophy. Res. Commun. 356, 286-292.
DOI
|