1 |
Janaki Ramaiah, M. and Vaishnave, S. 2018. BMI1 and PTEN are key determinants of breast cancer therapy: A plausible therapeutic target in breast cancer. Gene 678, 302-311.
DOI
|
2 |
Katzung, B. G. 2018. Basic & Clinical Pharmacology, pp. 409, 14th ed., McGraw-Hill: 1325 Avenue of the Americas New York, NY, USA.
|
3 |
Lee, K. E., Cho, K. O., Choi, Y. S. and Kim, S. Y. 2016. The neuroprotective mechanism of ampicillin in a mouse model of transient forebrain ischemia. Kor. J. Physiol. Pharmacol. 20, 185-192.
DOI
|
4 |
Leung, C., Lingbeek, M., Shakhova, O., Liu, J., Tanger, E., Saremaslani, P., Van Lohuizen, M. and Marino, S. 2004. Bmi1 is essential for cerebellar development and is overexpressed in human medulloblastomas. Nature 428, 337-341.
DOI
|
5 |
Li, A., Choi, Y. S., Dziema, H., Cao, R., Cho, H. Y., Jung, Y. J. and Obretan, K. 2010. Proteomic profiling of the epileptic dentate gyrus. Brain Pathol. 20, 1077-1089.
DOI
|
6 |
Li, J., Vangundy, Z. and Poi, M. 2020. PTC209, a specific inhibitor of BMI1, promotes cell cycle arrest and apoptosis in cervical cancer cell lines. Anticancer Res. 40, 133-141.
DOI
|
7 |
Liu. J., Cao, L., Chen, J., Song, S., Lee, I. H., Quijano, C., Liu, H., Keyvanfar, K., Chen, H., Cao, L. Y., Ahn, B. H., Kumar, N. G., Rovira, I. I., Xu, X. L., Van Lohuizen, M., Motoyama, N., Deng, C. X. and Finkel, T. 2009. Bmi1 regulates mitochondrial function and the DNA damage response pathway. Nature 459, 387-392.
DOI
|
8 |
Masukawa, L. M., Uruno, K., Sperling, M., O'Connor, M. J. and Burdette, L. J. 1992. The functional relationship between antidromically evoked field responses of the dentate gyrus and mossy fiber reorganization in temporal lobe epileptic patients. Brain Res. 579, 119-127.
DOI
|
9 |
Park, I. K., Morrison, S. J. and Clarke, M. F. 2004. Bmi1, stem cells, and senescence regulation. J. Clin. Invest. 113, 175-179.
DOI
|
10 |
Pathan, S. A., Jain, G. K., Akhter, S., Vohora, D., Ahmad, F. J. and Khar, R. K. 2010. Insights into the novel three 'D's of epilepsy treatment: drugs, delivery systems and devices. Drug Discov. Today 15, 717-732.
DOI
|
11 |
Polli, R. S., Malheirosm, J. M., Dos Santos, R., Hamani, C., Longo, B. M., Tannús, A., Mello, L. E. and Covolan, L. 2014. Changes in hippocampal volume are correlated with cell loss but not with seizure frequency in two chronic models of temporal lobe epilepsy. Front Neurol. 5, 111.
|
12 |
Racine, R. J. 1972. Modification of seizure activity by electrical stimulation. II. motor seizure. Electroencephalogr. Clin. Neurophysiol. 32, 281-294.
DOI
|
13 |
Represa, A., Jorquera, I., Le Gal La Salle, G. and Ben-Ari, Y. 1993. Epilepsy induced collateral sprouting of hippocampal mossy fibers: does it induce the development of ectopic synapses with granule cell dendrites? Hippocampus 3, 257-268.
|
14 |
Shibley, H. and Smith, B. N. 2002. Pilocarpine-induced status epilepticus results in mossy fiber sprouting and spontaneous seizures in C57BL/6 and CD-1 mice. Epilepsy Res. 49, 109-120.
DOI
|
15 |
Reynolds, J. P., Miller-Delaney, S. F., Jimenez-Mateos, E. M., Sano, T., McKiernan, R. C., Simon, R. P. and Henshall, D. C. 2015. Transcriptional response of polycomb group genes to status epilepticus in mice is modified by prior exposure to epileptic preconditioning. Front Neurol. 6, 46.
DOI
|
16 |
Schmeiser, B., Zentner, J., Prinz, M., Brandt, A. and Freiman, T. M. 2017. Extent of mossy fiber sprouting in patients with mesiotemporal lobe epilepsy correlates with neuronal cell loss and granule cell dispersion. Epilepsy Res. 129, 51-58.
DOI
|
17 |
Schmidt, D. 2009. Drug treatment of epilepsy: options and limitations. Epilepsy Behav. 15, 56-65.
DOI
|
18 |
Valk-Lingbeek, M. E., Bruggeman, S. W. and van Lohuizen,M. 2004. Stem cells and cancer; the polycomb connection.Cell 118, 409-418.
DOI
|
19 |
Stapels, M., Piper, C., Yang, T., Li, M., Stowell, C., Xiong, Z. G., Saugstad, J., Simon, R. P., Geromanos, S., Langridge, J., Lan, J. Q. and Zhou, A. 2010. Polycomb group proteins as epigenetic mediators of neuroprotection in ischemictolerance. Sci. Signal. 3, 10.
|
20 |
Trahair, T. N., Liu, T., Wainwright, B. J., Ding, H. F. andMarshall, G. M. 2013. Direct effects of Bmi1 on p53 proteinstability inactivates oncoprotein stress responses in embryonalcancer precursor cells at tumor initiation. Oncogene 32,3616-3626.
DOI
|
21 |
Wang, M. C., Li, C. L., Cui, J., Jiao, M., Wu, T., Jing, L.and Nan, K. J. 2015. BMI-1, a promising therapeutic targetfor human cancer. Oncol. Lett. 10, 583-588.
DOI
|
22 |
Cavarsan, C. F., Malheiros, J., Hamani, C., Najm, I. and Covolan, L. 2018. Is mossy fiber sprouting a potential therapeutic target for epilepsy? Front Neurol. 9, 1023.
DOI
|
23 |
Xu, X., Wang, Z., Liu, N., Zhang, P., Liu, H., Qi, J. andTu, Y. 2018. The mechanism of BMI1 in regulating cancerstemness maintenance, metastasis, chemo- and radiationresistance. Cancer Transl. Med. 4, 59-63.
DOI
|
24 |
Siddique, H. R. and Saleem, M. 2012. Role of BMI1, a stem cell factor, in cancer recurrence and chemoresistance: preclinical and clinical evidences. Stem Cells 30, 372-378.
DOI
|
25 |
Abdouh, M., Chatoo, W., El, H. J., David, J., Ferreira, J. and Bernier, G. 2012. Bmi1 is down-regulated in the aging brain and displays antioxidant and protective activities in neurons. PLoS One 7, e31870.
DOI
|
26 |
Barabino, A., Plamondon, V., Abdouh, M., Chatoo, W., Flamier, A., Hanna, R., Zhou, S., Motoyama, N., Hebert, M., Lavoie, J. and Bernier, G. 2016. Loss of Bmi1 causes anomalies in retinal development and degeneration of cone photoreceptors. Development 143, 1571-1584.
DOI
|
27 |
Calao, M., Sekyere, E. O., Cui, H. J., Cheung, B. B., Thomas, W. D., Keating, J., Chen, J. B., Raif, A., Jankowski, K., Davies, N. P., Bekkum, M. V., Chen, B., Tan, O., Ellis, T., Norris, M. D., Haber, M., Kim, E. S., Shohet, J. M., Trahair, T. N., Liu, T., Wainwright, B. J., Ding, H. F. and Marshall, G. M. 2013. Direct effects of Bmi1 on p53 protein stability inactivates oncoprotein stress responses in embryonal cancer precursor cells at tumor initiation. Oncogene 32, 3616-3626.
DOI
|
28 |
Chatoo, W., Abdouh, M., David, J., Champagne, M. P., Ferreira, J., Rodier, F. and Bernier, G. 2009. The polycomb group gene Bmi1 regulates antioxidant defenses in neurons by repressing p53 pro-oxidant activity. J. Neurosci. 29, 529-542.
DOI
|
29 |
Courel, M., Friesenhahn, L. and Lees, J. A. 2008. E2f6 and Bmi1 cooperate in axial skeletal development. Dev. Dyn. 237, 1232-1242.
DOI
|
30 |
Chen, Z., Brodie, M. J., Liew, D. and Kwan, P. 2018. Treatment outcomes in patients with newly diagnosed epilepsy treated with established and new antiepileptic drugs: a 30-year longitudinal cohort study. JAMA Neurol. 75, 279-286.
DOI
|
31 |
Davies, J. A. 1995. Mechanisms of action of antiepileptic drugs. Seizure 4, 267-271.
DOI
|
32 |
El Hajjar, J., Chatoo, W., Hanna, R., Nkanza, P., Tétreault, N., Tse, Y. C., Wong, T. P., Abdouh, M. and Bernier, G. 2019. Heterochromatic genome instability and neurodegeneration sharing similarities with Alzheimer's disease in old Bmi1+/- mice. Sci. Rep. 9, 594.
DOI
|
33 |
de Lanerolle, N. C., Kim, J. H., Williamson, A., Spencer, S. S., Zaveri, H. P., Eid, T. and Spencer, D. D. 2003. A retrospective analysis of hippocampal pathology in human temporal lobe epilepsy: evidence for distinctive patient subcategories. Epilepsia 44, 677-687.
DOI
|
34 |
Dey, A., Xiong, X., Crim, A., Dwivedi, S. K. D., Mustafi, S. B., Mukherjee, P., Cao, L., Sydorenko, N., Baiazitov, R., Moon, Y. C., Dumble, M., Davis, T. and Bhattacharya, R. 2018. Evaluating the mechanism and therapeutic potential of PTC-028, a novel inhibitor of BMI-1 function in ovarian cancer. Mol. Cancer Ther. 17, 39-49.
DOI
|
35 |
Ding, X., Lin, Q., Ensenat-Waser, R., Rose-John, S. and Zenke, M. 2012. Polycomb group protein Bmi1 promotes hematopoietic cell development from embryonic stem cells. Stem Cells Dev. 21, 121-132.
DOI
|
36 |
Heng, K., Haney, M. M. and Buckmaster, P. S. 2013. Highdose rapamycin blocks mossy fiber sprouting but not seizures in a mouse model of temporal lobe epilepsy. Epilepsia 54, 1535-1541.
DOI
|
37 |
Ginjala, V., Nacerddine, K., Kulkarni, A., Oza, J., Hill, S. J., Yao, M., Citterio, E., van Lohuizen, M. and Ganesan, S. 2011. Bmi1 is recruited to DNA breaks and contributes to DNA damage-induced H2A ubiquitination and repair. Mol. Cell Biol. 31, 1972-1982.
DOI
|
38 |
Gu, M., Shen, L., Bai, L., Gao, J., Marshall, C., Wu, T., Ding, J., Miao, D. and Xiao, M. 2014. Heterozygous knockout of the Bmi-1 gene causes an early onset of phenotypes associated with brain aging. Age (Dordr) 36, 129-139.
DOI
|