1 |
Chalkiadaki, A, Igarashi, M, Nasamu, A. S, Knezevic, J. and Guarente, L. 2014. Muscle-specific SIRT1 gain-of-function increases slow-twitch fibers and ameliorates pathophysiology in a mouse model of Duchenne muscular dystrophy. PLoS Genet. 10, e1004490.
DOI
|
2 |
Goldman, S. J., Taylor, R., Zhang, Y. and Jin, S. 2010. Autophagy and the degradation of mitochondria. Mitochondrion 10, 309-315.
DOI
|
3 |
Gomes, L. C., Di Benedetto, G. and Scorrano, L. 2011. During autophagy mitochondrial elongate, are spared from degradation and sustain cell viability. Nat. Cell Biol. 13, 589-598.
DOI
|
4 |
Hanna, R. A., Quinsay, M. N., Orogo, A. M., Giang, K. and Rikka, S., et al. 2012. Microtubule-associated protein 1 light chain 3 (LC3) interacts with Bnip3 protein to selectively remove endoplasmic reticulum and mitochondria via autophagy. J. Biol. Chem. 287, 19094-19104.
DOI
|
5 |
Irrcher, I., Liubicic, V. and Hood, D. A. 2009. Interactions between ROS and AMP kinase activity in the regulation of PGC-1alpha transcription in skeletal muscle cells. Am. J. Physiol. Cell Physiol. 296, C116-123.
DOI
|
6 |
Twig, G., Elorza, A., Molina, A. J., Mohamed, H. and Wikstrom, J. D., et al. 2008. Fission and selective fusion govern mitochondrial segregation and elimination by autophagy. EMBO 27, 433-446.
DOI
|
7 |
Vainshtein, A. and Hood, D. A. 2016. The regulation of autophagy during exercise in skeletal muscle. J. Appl. Physiol. 120, 664-673.
DOI
|
8 |
Warburton, D. E., Nicol, C. W. and Bredin, S. S. 2006. Health benefits of physical activity: the evidence. CM AJ. 174, 801-809.
|
9 |
Wright, D. C., Han, D. H., Garcia-Roves, P. M., Geiger, P. C. and Jones, E. T., et al. 2007. Exercise-induced mitochondrial biogenesis begins before the increase in muscle PGC-1alpha expression. J. Biol. Chem. 282, 194-199.
DOI
|
10 |
Xiao, B., Heath, R., Saiu, P., Leiper, F. C. and Leone, P., et al. 2007. Structural basis for AMP binding to mammalian AMP-activated protein kinase. Nature 449, 496-500.
DOI
|
11 |
Yan, Z., Lira, V. A. and Greene, N. P. 2012. Exercise training-induced regulation of mitochondrial quality. Exerc. Sport Sci. Rev. 40, 159-164.
|
12 |
Gerhart-Hines, Z., Rodgers, J. T., Bare, O., Lerin, C. and Kim, S. H., et al. 2007. Metabolic control of muscle mitochondrial function and fatty acid oxidation through SIRT1/PGC-1alpha. EMBO J. 26, 1913-1923.
DOI
|
13 |
Menzies, K. J., Singh, K., Saleem, A. and Hood, D. A. 2013. Sirtuin 1-mediated effects of exercise and resveratrol on mitochondrial biogenesis. J. Biol. Chem. 288, 6968-6979.
DOI
|
14 |
Chan, D. C. 2012. Fusion and fission: interlinked processes critical for mitochondrial health. Annu. Rev. Genet. 46, 265-287.
DOI
|
15 |
Davies, S. P., Helps, N. R., Cohen, P. T. and Hardie, D. G. 1995. 5'-AMPK inhibits dephosphorylation, as well as promoting phosphorylation, of the AMPK-activated protein kinase: studies using bacterially expressed human protein phosphatase-2C alpha and native bovine protein phosphatase-2AC. FEBS Lett. 377, 421-425.
DOI
|
16 |
Drake, J. C., Wilson, R. J. and Yan, Z. 2016. Molecular mechanisms for mitochondrial adaptation to exercise training in skeletal muscle. FASEB J. 30, 13-22.
DOI
|
17 |
Geisler, S., Holmstrom, K. M., Skujat, D., Fiesel, F. C. and Rothfuss, O. C., et al. 2010. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1. Nat. Cell Biol. 12, 119-131.
DOI
|
18 |
Klionsky, D. J. 2007. Autophagy: from phenomenology to molecular understanding in less than a decade. Nat. Rev. Mol. Cell Biol. 8, 931-937.
DOI
|
19 |
Jin, S. M., Lazarou, M., Wang, C., Kane, L. A. and Narendra, D. P., et al. 2010. Mitochondrial membrane potential regulates PINK1 import and proteolytic destabilization by PARL. J. Cell Biol. 191, 933-942.
DOI
|
20 |
Ju, J. S., Jeon, S. I., Park, J. Y., Lee, J. Y. and Lee, S. C., et al. 2016. Autophagy plays a role in skeletal muscle mitochondrial biogenesis in an endurance exercise-trained condition. J. Physiol. Sci. 66, 417-430.
DOI
|
21 |
Pilegaard, H., Saltin, B. and Neufer, P. D. 2003. Exercise induces transient transcriptional activation of the PGC-1 alpha gene in human skeletal muscle. J. Physiol. 546, 851-858.
DOI
|
22 |
Mizushima, N., Ohsumi, Y. and Yoshimori, T. 2002. Autophagosome formation in mammalian cells. Cell Struct. Funct. 27, 421-429.
DOI
|
23 |
Palikaras, K. and Tavernarakis, N. 2014. Mitochondrial homeostasis: the interplay between mitophagy and mitochondrial biogenesis. Exp. Geront. 56, 182-188.
DOI
|
24 |
Perry, C. G., Lally, J., Holloway, G. P. and Heigenhauser, G. J., et al. 2010. Repeated transient mRNA bursts precede increases in transcriptional and mitochondrial proteins during training in human skeletal muscle. J. Physiol. 588, 4795-4810.
DOI
|
25 |
Puigserver, P., Adelmant, G., Wu, Z., Fan, M. and Xu, J., et al. 1999. Activation of PPARgamma coactivator-1 through transcription factor docking. Science 286, 1368-1371.
DOI
|
26 |
Ravikumar, B., Sarkar, S., Davies, J. E., Futter, M. and Garcia-Arencibia, M., et al. 2010. Regulation of mammalian autophagy in physiology and pathophysiology. Physiol. Rev. 90, 1383-1435.
DOI
|
27 |
Safdar, A., Little, J. P., Stokl, A. J., Hettinga, B. P. and Akhtar, M., et al. 2011. Exercise increases mitochondrial PGC-1alpha content and promotes nuclear-mitochondrial cross-talk to coordinate mitochondrial biogenesis. J. Biol. Chem. 286, 10605-10617.
DOI
|
28 |
Tam, B. T., Pei, X. M., Yu, A. P., Sin, T. K. and Leung, K. K., et al. 2015. Autophagic adaptation is associated with exercise-induced fibre-type shifting in skeletal muscle. Acta. Physiol. 214, 221-236.
DOI
|
29 |
Levine, B. and Klionsky, D. J. 2004. Development by self-digestion: molecular mechanisms and biological functions of autophagy. Dev. Cell. 6, 463-477.
DOI
|
30 |
Konopka, A. R., Suer, M. K., Wolff, C. A. and Harber, M. P. 2014. Markers of human skeletal muscle mitochondrial biogenesis and quality control: effects of age and aerobic exercise training. J. Gerontol. A. Biol. Sci. Med. Sci. 69, 371-378.
DOI
|
31 |
Lira, V. A., Okutsu, M., Zhang, M., Greene, N. P. and Laker, R. C, et al. 2013. Autophagy is required for exercise training-induced skeletal muscle adaptation and improvement of physical performance. FASEB J. 27, 4184-4193.
DOI
|
32 |
Little, J. P., Safdar, A., Benton, C. R. and Wright, D.C. 2011. Skeletal muscle and beyond: the role of exercise as mediator of systemic mitochondrial biogenesis. Appl. Physiol. Nutr. Metab. 36, 598-607.
DOI
|
33 |
Masiero, E., Agatea, L., Mammucari, C., Blaauw, B. and Loro, E., et al. 2009. Autophagy is required to maintain muscle mass. Cell Metab. 10, 507-515.
DOI
|
34 |
McConell, G. K., Ng, G. P., Phillips, M., Ruan, Z. and Macaulay, S. L., et al. 2010. Central role of nitric oxide synthase in AICAR and caffeine-induced mitochondrial biogenesis in L6 myocytes. J. App.l Physiol. 108, 589-595.
|