Association of Cytotoxic T-lymphocyte Antigen-4 Polymorphisms with Malignant Bone Tumors Risk: A Meta-analysis |
Zhang, Chao
(Department of orthopaedics, The First Hospital of Lanzhou University)
Hou, Wei-Hua (Department of orthopaedics, The First Hospital of Lanzhou University) Ding, Xuan-Xi (Department of orthopaedics, The First Hospital of Lanzhou University) Wang, Xiong (Department of orthopaedics, The First Hospital of Lanzhou University) Zhao, Hui (Department of orthopaedics, The First Hospital of Lanzhou University) Han, Xing-Wen (Department of orthopaedics, The First Hospital of Lanzhou University) Wang, Wen-Ji (Department of orthopaedics, The First Hospital of Lanzhou University) |
1 | Lesterhuis W J, Salmons J, Nowak A K, et al (2013). Synergistic effect of CTLA-4 blockade and cancer chemotherapy in the induction of anti-tumor immunity. PloS one, 8, 61895. DOI |
2 | Liu X, Yang B, Ren H, et al (2015). Current Evidence On the Cytotoxic T-Lymphocyte Antigen 4+49G>A polymorphism and digestive system cancer risks: a meta-analysis involving 11, 923 subjects. Meta Gene, 6, 105-8. DOI |
3 | Lo K L, Mertz D, Loeb M (2014). Newcastle-Ottawa Scale: comparing reviewers' to authors' assessments. Bmc Med Res Method, 14, 45. DOI |
4 | Mackintosh C, Ordonez J L, Garcia-Dominguez D J, et al (2012). 1q gain and CDT2 overexpression underlie an aggressive and highly proliferative form of Ewing sarcoma. Oncogene, 31, 1287-98. DOI |
5 | Miller B J, Lynch C F, Buckwalter J A (2013). Conditional survival is greater than overall survival at diagnosis in patients with osteosarcoma and Ewing's sarcoma. Clin Orthop Relat R, 471, 3398-404. DOI |
6 | Postel-Vinay S, Veron A S, Tirode F, et al (2012). Common variants near TARDBP and EGR2 are associated with susceptibility to Ewing sarcoma. Nat Genet, 44, 323-7. DOI |
7 | Rachel W, Carroll R J (2014). Testing Hardy-Weinberg equilibrium with a simple root-mean-square statistic. Biostatistics, 15, 74-86. DOI |
8 | Salvi S, Fontana V, Boccardo S, et al (2012). Evaluation of CTLA-4 expression and relevance as a novel prognostic factor in patients with non-small cell lung cancer. Cancer Immunol Immun, 61, 1463-72. DOI |
9 | Silva D S B S, Sawitzki F R, De Toni E C, et al (2012). Ewing's sarcoma: analysis of single nucleotide polymorphism in the EWS gene. Gene, 509, 263-6. DOI |
10 | Sun T, Zhou Y M, Hu Z, et al (2008). Functional genetic variations in cytotoxic T-lymphocyte antigen 4 and susceptibility to multiple types of cancer. Cancer Res, 68, 7025-34. DOI |
11 | Yuan J, Ginsberg B, Page D, et al (2011). CTLA-4 blockade increases antigen-specific CD8+ T cells in prevaccinated patients with melanoma: three cases. Cancer Immunol Immun, 60, 1137-46. DOI |
12 | Wang W, Wang J, Song H, et al (2011). Cytotoxic T-lymphocyte antigen-4 +49G/A polymorphism is associated with increased risk of osteosarcoma. Genet Test Mol Biom, 15, 503-6. DOI |
13 | Yang L, Zhimin H, Dapeng F, et al (2011). Cytotoxic T-lymphocyte antigen-4 polymorphisms and susceptibility to osteosarcoma. Dna Cell Biol, 30, 1051-5. DOI |
14 | Yang M, Sun T, Zhou Y, et al (2012). The functional cytotoxic T lymphocyte–associated Protein 4 49G-to-A genetic variant and risk of pancreatic cancer. Cancer, 118, 4681-86. DOI |
15 | Yang S, Wang C, Zhou Y, et al (2012). Cytotoxic T-lymphocyte antigen-4 polymorphisms and susceptibility to Ewing's sarcoma. Genet Test Mol Biom, 16, 1236-40. DOI |
16 | Yu F, Miao J (2013). Significant association between cytotoxic T lymphocyte antigen 4 +49G>A polymorphism and risk of malignant bone tumors. Tumour Biol, 34, 3371-5. DOI |
17 | Zhang Y, Zhang J, Yao D, et al (2011). Polymorphisms in the cytotoxic T-lymphocyte antigen 4 gene and cancer risk: a meta-analysis. Cancer, 117, 4312-24. DOI |
18 | Benhatchi K, Jochmanova I, Habalova V, et al (2011). CTLA4 exon1 A49G polymorphism in Slovak patients with rheumatoid arthritis and Hashimoto thyroiditis-results and the review of the literature. Clinical Rheumatol, 30, 1319-24. DOI |
19 | Alfadhli S, Almutawa Q, Abbas J M K, et al (2013). Association of Hashimoto's thyroiditis with cytotoxic T lymphocyteassociated antigen-4 (CTLA-4) and inducible co-stimulator (ICOS) genes in a Kuwaiti population. Endocrine, 43, 666-77. DOI |
20 | Balamuth N J, Womer R B (2010). Ewing's sarcoma. Lancet Oncol, 11, 184-192. DOI |
21 | Gil M A, Maside C, Cuello C, et al (2015). Osteosarcoma of the Spine: Prognostic Variables for Local Recurrence and Overall Survival, A Multicenter Ambispective Study. Mol Reprod Dev, 5, 651-63. |
22 | Bian Z, He Q, Wang X, et al (2014). Association of genetic polymorphisms with osteosarcoma risk: a meta-analysis. Int J Clin Exp Med, 8, 8317-28. |
23 | Bielack S, Carrle D, Casali P G (2009). Osteosarcoma: ESMO clinical recommendations for diagnosis, treatment and follow-up. Ann Oncol, 20, 137-9. DOI |
24 | Feng D, Yang X, Li S, et al (2016). Cytotoxic T-lymphocyte antigen-4 genetic variants and risk of Ewing's sarcoma. Genet Test Mol Biom, 17, 458-63. |
25 | Hao Q, Weifeng T, Pengfei Y, et al (2013). Cytotoxic T-lymphocyte associated antigen 4 polymorphism and hashimoto's thyroiditis susceptibility: a meta-analysis. Endocrine, 45, 198-205. |
26 | He L, Deng T, Luo H S (2014). Association between cytotoxic T-lymphocyte antigen-4 +49A/G polymorphism and colorectal cancer risk: a meta-analysis. Int J Clin Exp Med, 8, 3752-60. |
27 | Hironori U, Howson J M M, Laura E, et al (2003). Association of the T-cell regulatory gene CTLA4 with susceptibility to autoimmune disease. Nature, 423, 506-11. DOI |
28 | Lang C, Chen L, Li S (2012). Cytotoxic T-lymphocyte antigen-4+ 49G/A polymorphism and susceptibility to pancreatic cancer[J]. Dna Cell Biol, 31, 683-7. DOI |
29 | Huang H J, Angelo L S, Rodon J, et al (2011). R1507, an antiinsulin- like growth factor-1 receptor (IGF-1R) antibody, and EWS/FLI-1 siRNA in Ewing's sarcoma: convergence at the IGF/IGFR/Akt axis. PloS one, 6, 26060. DOI |
30 | Kager L, Zoubek A, Pötschger U, et al (2003). Primary metastatic osteosarcoma: presentation and outcome of patients treated on neoadjuvant Cooperative Osteosarcoma Study Group protocols. J Clin Oncol, 21, 2011-8. DOI |