Situation of HPV16 E2 Gene Status During Radiotherapy Treatment of Cervical Carcinoma |
Kahla, Saloua
(Unit of Immunology Microbiology Environmental and Carcinogenesis (IMEC), Science Faculty of Bizerte, University of Carthage)
Kochbati, Lotfi (Radio-Oncology Department, Salah Azaiez Institute) Maalej, Mongi (Radio-Oncology Department, Salah Azaiez Institute) Oueslati, Ridha (Unit of Immunology Microbiology Environmental and Carcinogenesis (IMEC), Science Faculty of Bizerte, University of Carthage) |
1 | Wang CC, Lai CH, Huang HJ, et al (2010). Clinical effect of human papillomavirus genotypes in patients with cervical cancer undergoing primary radiotherapy. Int J Radiat Oncol Biol Phys, 78, 1111-20. DOI ScienceOn |
2 | Thierry F, Demeret C (2008). Direct activation of caspase 8 by the proapoptotic E2 protein of HPV18 independent of adaptor proteins. Cell Death Differ, 15, 1356-63. DOI ScienceOn |
3 | Thierry F (2009). Transcriptional regulation of the papillomavirus oncogenes by cellular and viral transcription factors in cervical carcinoma. Virology, 384, 375-9. DOI ScienceOn |
4 | Vozenin MC, Lord HK, Hartl D, Deutsch E (2010). Unravelling the biology of human papillomavirus (HPV) related tumours to enhance their radiosensitivity. Cancer Treat Rev, 36, 629-36. DOI ScienceOn |
5 | Wang L, Dai SZ, Chu HJ, Cui HF, Xu XY (2013). Integration sites and genotype distributions of human papillomavirus in cervical intraepithelial neoplasia. Asian Pac J Cancer Prev, 14, 3837-41. DOI ScienceOn |
6 | Yang YY, Koh LW, Tsai JH, et al (2004). Correlation of viral factors with cervical cancer in Taiwan. J Microbiol Immunol Infect, 37, 282-7. |
7 | Lai CH, Chou HH, Chang CJ, et al (2013). Clinical implications of human papillomavirus genotype in cervical adenoadenosquamous carcinoma. Eur J Cancer, 49, 633-41. DOI ScienceOn |
8 | Gammoh N, Isaacson E, Tomaic V, et al (2009). Inhibition of HPV-16 E7 oncogenic activity by HPV-16 E2. Oncogene, 28, 2299-304. DOI ScienceOn |
9 | Gillison ML, Koch WM, Capone RB, et al (2000). Evidence for a causal association between human papillomavirus and a subset of head and neck cancers. J Natl Cancer Inst, 92, 709-20. DOI ScienceOn |
10 | Harima Y, Sawada S, Nagata K, Sougawa M, Ohnishi T (2001). Chromosome 6p21.2, 18q21.2 and human papilloma virus (HPV) DNA can predict prognosis of cervical cancer after radiotherapy. Int J Cancer, 96, 286-96. DOI ScienceOn |
11 | Le Tinier F, Reynaert N, Castelain B, et al (2012). Is adaptive intensity-modulated radiotherapy for uterine cervix carcinoma necessary? Cancer Radiother, 16, 681-7. DOI ScienceOn |
12 | Lindel K, Burri P, Studer HU, et al (2005). Human papillomavirus status in advanced cervical cancer: predictive and prognostic significance for curative radiation treatment. Int J Gynecol Cancer, 15, 278-84. DOI |
13 | Li H, Yang Y, Zhang R, et al (2013). Preferential sites for the integration and disruption of human papillomavirus 16 in cervical lesions. J Clin Virol, 56, 342-7. DOI ScienceOn |
14 | Lindel K, Beer KT, Laissue J, Greiner RH, Aebersold DM (2001). Human papillomavirus positive squamous cell carcinoma of the oropharynx: a radiosensitive subgroup of head and neck carcinoma. Cancer, 92, 805-13. DOI |
15 | Lindel K, de Villiers EM, Burri P, et al (2006). Impact of viral E2-gene status on outcome after radiotherapy for patients with human papillomavirus 16-positive cancer of the uterine cervix. Int J Radiat Oncol Biol Phys, 65, 760-5. DOI |
16 | Lindel K, Rieken S, Daffinger S, et al (2012). The transcriptional regulator gene E2 of the Human Papillomavirus (HPV) 16 influences the radiosensitivity of cervical keratinocytes. Radiat Oncol, 7, 187. DOI |
17 | Nagai Y, Maehama T, Asato T, Kanazawa K (2000). Persistence of human papillomavirus infection after therapeutic conization for CIN 3: is it an alarm for disease recurrence? Gynecol Oncol, 79, 294-9. DOI ScienceOn |
18 | Parkin DM, Bray F, Ferlay J, Pisani P (2005). Global cancer statistics, 2002. CA Cancer J Clin, 55, 74-108. DOI ScienceOn |
19 | Santin AD, Hermonat PL, Ravaggi A, et al (1998). Radiationenhanced expression of E6/E7 transforming oncogenes of human papillomavirus-16 in human cervical carcinoma. Cancer, 83, 2346-52. DOI |
20 | Schmitz M, Driesch C, Beer-Grondke K, et al (2012). Loss of gene function as a consequence of human papillomavirus DNA integration. Int J Cancer, 131, 593-602. DOI |
21 | Castellsague X, Munoz N (2003). Chapter 3: Cofactors in human papillomavirus carcinogenesis-role of parity, oral contraceptives, and tobacco smoking. J Natl Cancer Inst Monogr, 3, 20-8. |
22 | Bellanger S, Tan CL, Xue YZ, Teissier S, Thierry F (2011). Tumor suppressor or oncogene? A critical role of the human papillomavirus (HPV) E2 protein in cervical cancer progression. Am J Cancer Res, 1, 373-89. |
23 | Bhattacharjee B, Sengupta S (2006). HPV16 E2 gene disruption and polymorphisms of E2 and LCR: Some significant associations with cervical cancer in Indian women. Gynecol Oncol, 100, 372-8. DOI |
24 | But-Hadzic J, Jenko K, Poljak M, et al (2011). Sinonasal inverted papilloma associated with squamous cell carcinoma. Radiol Oncol, 45, 267-72. |
25 | Castle PE, Lorincz AT, Mielzynska-Lohnas I, et al (2002). Results of human papillomavirus DNA testing with the hybrid capture 2 assay are reproducible. J Clin Microbiol, 40, 1088-90. DOI ScienceOn |
26 | Cricca M, Venturoli S, Leo E, et al (2009). Molecular analysis of HPV 16 E6I/E6II spliced mRNAs and correlation with the viral physical state and the grade of the cervical lesion. J Med Virol, 81, 1276-82. DOI |
27 | Delgado FG, Martinez E, Cespedes MA, et al (2009). Increase of human papillomavirus-16 E7-specific T helper type 1 response in peripheral blood of cervical cancer patients after radiotherapy. Immunology, 126, 523-34. DOI |
28 | Desaintes C, Goyat S, Garbay S, Yaniv M, Thierry F (1999). Papillomavirus E2 induces p53-independent apoptosis in HeLa cells. Oncogene, 18, 4538-45. DOI |
29 | Alazawi W, Pett M, Strauss S, et al (2004). Genomic imbalances in 70 snap-frozen cervical squamous intraepithelial lesions: associations with lesion grade, state of the HPV16 E2 gene and clinical outcome. Br J Cancer, 91, 2063-70. DOI |
30 | Gammoh N, Grm HS, Massimi P, Banks L (2006). Regulation of human papillomavirus type 16 E7 activity through direct protein interaction with the E2 transcriptional activator. J Virol, 80, 1787-97. DOI |
31 | Micalessi IM, Boulet GA, Bogers JJ, Benoy IH, Depuydt CE (2011). High-throughput detection, genotyping and quantification of the human papillomavirus using real-time PCR. Clin Chem Lab Med, 50, 655-61. |