MUTYH Association with Esophageal Adenocarcinoma in a Han Chinese Population |
Kong, Feng
(Central Research Laboratory, The Second Hospital of Shandong University)
Han, Xue-Ying (Jinan Blood Station, Blood Center of Shandong Province) Luan, Yun (Central Research Laboratory, The Second Hospital of Shandong University) Qi, Tong-Gang (Central Research Laboratory, The Second Hospital of Shandong University) Sun, Chao (Central Research Laboratory, The Second Hospital of Shandong University) Wang, Jue (Central Research Laboratory, The Second Hospital of Shandong University) Hou, Hua-Ying (Oncology Department, The Second Hospital of Shandong University) Jiang, Yu-Hua (Oncology Department, The Second Hospital of Shandong University) Zhao, Jing-Jie (Molecular Biology Laboratory, The Second Hospital of Shandong University) Cheng, Guang-Hui (Central Research Laboratory, The Second Hospital of Shandong University) |
1 |
Brand R, Nielsen M, Lynch H, Infante E (2012). MUTYH-Associated Polyposis. |
2 | Cheng G, Wang H, Chen M, et al (2011). Lack of evidence to support the association of polymorphisms within the TNFSF4 gene and coronary heart disease in a Chinese Han population. Exp Ther Med, 2, 275-280. DOI |
3 | Gu H, Ding G, Zhang W, et al (2012). Replication study of PLCE1 and C20orf54 polymorphism and risk of esophageal cancer in a Chinese population. Mol Biol Rep, 39, 9105-11. DOI |
4 | Markkanen E, Dorn J, Hubscher U (2013). MUTYH DNA glycosylase: the rationale for removing undamaged bases from the DNA. Front Genet, 4, 18. |
5 | Miyaishi A, Osawa K, Osawa Y, et al (2009). MUTYH Gln324His gene polymorphism and genetic susceptibility for lung cancer in a Japanese population. J Exp Clin Cancer Res, 28, 10. DOI ScienceOn |
6 | Picelli S, Zajac P, Zhou XL, et al (2010). Common variants in human CRC genes as low-risk alleles. Eur J Cancer, 46, 1041-8. DOI ScienceOn |
7 | Przybylowska K, Kabzinski J, Sygut A, et al (2013). An association selected polymorphisms of XRCC1, OGG1 and MUTYH gene and the level of efficiency oxidative DNA damage repair with a risk of colorectal cancer. Mutat Res, 745-746, 6-15. DOI ScienceOn |
8 | Ruggieri V, Pin E, Russo MT, et al (2012). Loss of MUTYH function in human cells leads to accumulation of oxidative damage and genetic instability. Oncogene, 32, 4500-8. |
9 | Santos LS, Branco SC, Silva SN, et al (2012). Polymorphisms in base excision repair genes and thyroid cancer risk. Oncol Rep, 28, 1859-68. DOI |
10 | Sliwinski T, Markiewicz L, Rusin P, et al (2009). Polymorphisms of the DNA base excision repair gene MUTYH in head and neck cancer. Exp Oncol, 31, 57-9. |
11 | Stanczyk M, Sliwinski T, Cuchra M, et al (2011). The association of polymorphisms in DNA base excision repair genes XRCC1, OGG1 and MUTYH with the risk of childhood acute lymphoblastic leukemia. Mol Biol Rep, 38, 445-51. DOI |
12 | Torrezan GT, da Silva FC, Santos EM, et al (2013). Mutational spectrum of the APC and MUTYH genes and genotype-phenotype correlations in Brazilian FAP, AFAP, and MAP patients. Orphanet J Rare Dis, 8, 54. DOI |
13 | You SH, Wang X, Huang S, et al (2013). MYH rs3219476 and rs3219472 polymorphisms and risk of cholangiocarcinoma. Mol Med Rep, 7, 347-51 DOI |
![]() |