1 |
Hiramatsu M, Sasaki M, Nabeshima T, et al. Effects of dynorphin A (1-13) on carbon monoxide-induced delayed amnesia in mice. Pharmacol Biochem Behav. 1997;56(1):73-9.
DOI
|
2 |
Akyol S, Erdogan S, Idiz N, et al. The role of reactive oxygen species and oxidative stress in carbon monoxide toxicity: an indepth analysis. Redox Rep. 2014;19(5):180-9.
DOI
|
3 |
Aldini G, Altomare A, Baron G, et al. N-Acetylcysteine as an antioxidant and disulphide breaking agent: the reasons why. Free Radic Res. 2018;52(7):751-62.
DOI
|
4 |
Raub JA, Mathieu-Nolf M, Hampson NB, et al. Carbon monoxide poisoning--a public health perspective. Toxicology. 2000;145(1):1-14.
DOI
|
5 |
Rose JJ, Wang L, Xu Q, et al. Carbon Monoxide Poisoning: Pathogenesis, Management, and Future Directions of Therapy. Am J Respir Crit Care Med. 2017;195(5):596-606.
DOI
|
6 |
Kim YJ, Sohn CH, Oh BJ, et al. Carbon monoxide poisoning during camping in Korea. Inhal Toxicol. 2016;28(14):719-23
DOI
|
7 |
Weaver LK. Carbon Monoxide Poisoning. N Engl J Med. 2009;360(12):1217-25.
DOI
|
8 |
Hendrickson RG. What is the most appropriate dose of N-acetylcysteine after massive acetaminophen overdose?. Clin Toxicol (Phila). 2019;57(8):686-91
DOI
|
9 |
Hu H, Gan J, Jonas P. Interneurons. Fast-spiking, parvalbumin (+) GABAergic interneurons: from cellular design to microcircuit function. Science. 2014;345:1255-63.
DOI
|
10 |
Kim MJ, Choi SJ, Lim ST, et al. Ferulic acid supplementation prevents trimethyltin-induced cognitive deficits in mice. Biosci Biotechnol Biochem. 2007;71(4):1063-8.
DOI
|
11 |
Xue L, Wang WL, Li Y, et al. Effects of hyperbaric oxygen on hippocampal neuronal apoptosis in rats with acute carbon monoxide poisoning. Undersea Hyperb Med. 2017;44(2):121-31.
DOI
|
12 |
Gusel'nikova VV, Korzhevskiy DE. NeuN As a Neuronal Nuclear Antigen and Neuron Differentiation Marker. Acta naturae. 2015;7(2):42-7.
DOI
|
13 |
Hiramatsu M, Inoue K. Des-tyrosine(1) dynorphin A-(2-13) improves carbon monoxide-induced impairment of learning and memory in mice. Brain Res. 2000;859(2):303-10.
DOI
|
14 |
Fan DF, Hu HJ, Sun Q, et al. Neuroprotective effects of exogenous methane in a rat model of acute carbon monoxide poisoning. Brain Res. 2016;1633:62-72.
DOI
|
15 |
Cobley JN, Fiorello ML, Bailey DM. 13 reasons why the brain is susceptible to oxidative stress. Redox Biol. 2018;15:490-503.
DOI
|
16 |
Ruden JB, Dugan LL, Konradi C. Parvalbumin interneuron vulnerability and brain disorders. Neuropsychopharmacology. 2021;46(2):279-87.
DOI
|
17 |
Lee MS, Yang SB, Heo JH. Application of Thallium Autometallography for Observation of Changes in Excitability of Rodent Brain following Acute Carbon Monoxide Intoxication. J Korean Soc Clin Toxicol. 2019;17(2):66-78.
DOI
|
18 |
Miettinen R, Sirvio J, Riekkinen P Sr, et al. Neocortical, hippocampal and septal parvalbumin- and somatostatin-containing neurons in young and aged rats: correlation with passive avoidance and water maze performance. Neuroscience. 1993;53(2):367-78.
DOI
|
19 |
Freund TF, Buzsaki G. Interneurons of the hippocampus. Hippocampus. 1996;6(4):347-470.
DOI
|
20 |
Oh SH, Choi SC. Acute carbon monoxide poisoning and delayed neurological sequelae: a potential neuroprotection bundle therapy. Neural Regen Res. 2015;10(1):36-8.
DOI
|
21 |
Gustafsson B, Wigstrom H. Long-term potentiation in the hippocampal CA1 region: its induction and early temporal development. Prog Brain Res. 1990;83:223-32.
DOI
|
22 |
Mereu G, Cammalleri M, Fa M, et al. Prenatal exposure to a low concentration of carbon monoxide disrupts hippocampal long-term potentiation in rat offspring. J Pharmacol Exp Ther. 2000;294(2):728-34.
|
23 |
Katz M, Won SJ, Park Y, et al. Cerebrospinal fluid concentrations of N-acetylcysteine after oral administration in Parkinson's disease. Parkinsonism Relat Disord. 2015;21(5):500-3.
DOI
|
24 |
Mannaioni PF, Vannacci A, Masini E. Carbon monoxide: the bad and the good side of the coin, from neuronal death to antiinflammatory activity. Inflamm Res. 2006;55(7):261-73.
DOI
|
25 |
Kekec Z, Seydaoglul G, Sever H, et al. The effect of antioxidants (N-acetylcysteine and melatonin) on hypoxia due to carbonmonoxide poisoning. Bratisl Lek Listy. 2010;111(4):189-93.
|
26 |
Sabetghadam M, Mazdeh M, Abolfathi P, et al. Evidence for a Beneficial Effect of Oral N-acetylcysteine on Functional Outcomes and Inflammatory Biomarkers in Patients with Acute Ischemic Stroke. Neuropsychiatr Dis Treat. 2020;16:1265-78.
DOI
|
27 |
Johansen FF. Interneurons in rat hippocampus after cerebral ischemia. Morphometric, functional, and therapeutic investigations. Acta Neurol Scand Suppl. 1993;150:1-32.
|
28 |
Deepmala, Slattery J, Kumar N, et al. Clinical trials of N-acetylcysteine in psychiatry and neurology: A systematic review. Neurosci Biobehav Rev. 2015;55:294-321.
DOI
|
29 |
Bavarsad Shahripour R, Harrigan MR, Alexandrov AV. N-acetylcysteine (NAC) in neurological disorders: mechanisms of action and therapeutic opportunities. Brain Behav. 2014;4(2):108-22.
DOI
|
30 |
Millea PJ. N-acetylcysteine: multiple clinical applications. Am Fam Physician. 2009;80(3):265-9.
|