1 |
Kaati, G., Bygren, L. O. and Edvinsson, S. : Cardiovascular and diabetes mortality determined by nutrition during parents' and grandparents' slow growth period. European Journal of Human Genetics, 10(11), 682-688, 2002
DOI
ScienceOn
|
2 |
Cheng, R. Y., Hockman, T., Crawford, E., Anderson, L. M. and Shiao, Y. H. : Epigenetic and gene expression changes related to transgenerational carcinogenesis. Molecular Carcinogenesis, 40(1), 1-11, 2004
DOI
ScienceOn
|
3 |
Bindhumol, V., Chitra, K. C. and Mathur, P. P. : Bisphenol A induces reactive oxygen species gener ation in the liver of male rats. Toxicology, 188(2-3), 117-124, 2003
DOI
ScienceOn
|
4 |
Valinluck, V., Tsai, H. H., Rogstad, D. K., Burdzy, A., Bird, A., Sowers, L. C. : Oxidative damage to methyl-CpG sequences inhibits the binding of the methyl-CpG binding domain (MBD) of methyl-CpG binding protein 2 (MeCP2). Nucleic Acids Research, 32(14), 4100-4108, 2004
DOI
ScienceOn
|
5 |
Poirier, L. A., Vlasova, T. I. : The prospective role of abnormal methyl metabolism in cadmium toxicity. Environmental Health Perspectives, 110 Suppl 5, 793-795, 2002
DOI
ScienceOn
|
6 |
ATSDR. Toxicological profile for cadmium. Atlanta, GA: U.S. Department of Health & Human Services, 1999
|
7 |
Waalkes, M. P. : Cadmium carcinogenesis in review. Journal of Inorganic Biochemistry, 79(1-4), 241-244, 2000
DOI
ScienceOn
|
8 |
Sciandrello, G., Caradonna, F., Mauro, M. and Barbata, G. : Arsenic-induced DNA hypomethylation affects chromosomal instability in mammalian cells. Carcinogenesis, 25(3), 413-417, 2004
DOI
ScienceOn
|
9 |
Chen, W. T., Hung, W. C., Kang, W. Y., Huang, Y.C. and Chai, C. Y. : Urothelial carcinomas arising in arsenic-contaminated areas are associated with hypermethylation of the gene promoter of the deathassociated protein kinase. Histopathology, 51(6), 785-792, 2007
DOI
ScienceOn
|
10 |
Pilsner, J. R., Hu, H., Ettinger, A., Snchez, B. N.,Wright, R. O., Cantonwine, D., et al. : Inf luence o f Prenatal Lead Exposure on Genomic Methylation of Cord Blood DNA. Environmental Health Perspectives, (in press), 2009
DOI
|
11 |
Ji, W., Yang, L., Yu, L., Yuan, J., Hu, D. and Zhang, W., et al. : Epigenetic silencing of O6-methylguanine DNA methyltransferase gene in NiS-transformed cells. Carcinogenesis, 29(6), 1267-1275, 2008
DOI
ScienceOn
|
12 |
Lee, Y. W., Klein, C. B., Kargacin, B., Salnikow, K., Kitahara, J. and Dowjat. K., et al. : Carcinogenic nickel silences gene expression by chromatin condensation and DNA methylation: a new model for epigenetic carcinogens. Molecular and Cellular Biology, 15(5), 2547-2557, 1995
DOI
ScienceOn
|
13 |
Rubin, M. M. : Antenatal exposure to DES: lessons learned...future concerns. Obstetrical & Gynecological Survey, 62(8), 548-555, 2007
DOI
ScienceOn
|
14 |
Sato, K., Fukata, H., Kogo, Y., Ohgane, J., Shiota, K. and Mori, C. : Neonatal exposure to diethylstilbestrol alters the expression of DNA methyltransferases and methylation of genomic DNA in the epididymis of mice. Endocrine Journal, 53(3), 331-337, 2006
DOI
ScienceOn
|
15 |
Wu, Q., Ohsako, S., Ishimura, R., Suzuki, and J. S.,Tohyama, C. : Exposure of mouse preimplantation embryos to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) alters the methylation status o f imprinted genes H19 and Igf2. Biology of Reproduction, 70(6), 1790-1797, 2004
DOI
ScienceOn
|
16 |
Tang, W. Y., Newbold, R., Mardilovich, K., Jefferson, W., Cheng, R. Y. and Medvedovic, M., et al. :Persistent hypomethylation in the promoter of nucleosomal binding protein 1 (Nsbp1) correlates with overexpression of Nsbp1 in mouse uteri neonatally exposed to diethylstilbestrol or genistein. Endocrinology, 149(12), 5922-5931, 2008
DOI
ScienceOn
|
17 |
Rusiecki, J. A., Baccarelli, A., Bollati, V., Tarantini, L., Moore, L. E. and Bonefeld-Jorgensen, E. C. :Global DNA hypomethylation is associated with high serum-persistent organic pollutants in Greenlandic Inuit. Environmental Health Perspectives, 116(11), 1547-1552, 2008
DOI
ScienceOn
|
18 |
Okey, A. B. : An aryl hydrocarbon receptor odyssey to the shores of toxicology: the Deichmann Lecture, International Congress of Toxicology-XI. Toxicological Science, 98(1), 5-38, 2007
DOI
ScienceOn
|
19 |
Schwartz, J. : Air pollution and children's health. Pediatrics, 113(4 Suppl), 1037-1043, 2004
|
20 |
Cobiac, L. : Epigenomics and nutrition. Forum of Nutrition, 60, 31-41, 2007
DOI
|
21 |
Gallou-Kabani, C., Vige, A., Gross, M. S. and Junien, C. : Nutri-epigenomics: lifelong remodelling of our epigenomes by nutritional and metabolic factors and beyond. Clinical Chemistry and Laboratory Medicine, 45(3), 321-327, 2007
DOI
ScienceOn
|
22 |
Holliday, R. : Epigenetics: a historical overview. Epigenetics, 1(2), 76-80, 2006
DOI
ScienceOn
|
23 |
Szyf, M., Weaver, I., Meaney, M. : Maternal care, the epigenome and phenotypic differences in behavior. Reproductive Toxicology, 24(1), 9-19, 2007
DOI
ScienceOn
|
24 |
Barker, D. J. : In utero programming of chronic disease. Clinical Science (Lond), 95(2), 115-128, 1998
DOI
ScienceOn
|
25 |
Bolin, C. M., Basha, R, Cox, D., Zawia, N. H., Maloney,B., Lahiri, D. K., et al. : Exposure to lead and the developmental origin of oxidative DNA damage in the aging brain. FASEB Journal, 20(6), 788-790, 2006
DOI
ScienceOn
|
26 |
Gluckman, P. D., Hanson, M. A., Cooper, C., Thornburg, K. L. : Effect of in utero and early-life conditions on adult health and disease. New England Journal of Medicine, 359(1), 61-73, 2008
DOI
ScienceOn
|
27 |
Waterland, R. A. and Michels, K. B. : Epigenetic epidemiology of the developmental origins hypothesis. Annual Review of Nutrition, 27, 363-388, 2007
DOI
ScienceOn
|
28 |
Yoo, C. B. and Jones, P. A. : Epigenetic therapy of cancer: past, present and future. Nature Reviews Drug Discovery, 5(1), 37-50, 2006
DOI
ScienceOn
|
29 |
Steinke, J. W., Rich, S. S. and Borish, L. : 5. Genetics of allergic disease. Journal of Allergy and Clinical Immunology, 121(2 Suppl), S384-7; quiz S416, 2008
DOI
ScienceOn
|
30 |
Allis, C. D., Jenuwein, T., Reinberg, D. Overview and Concepts. In: Allis, C. D., Jenuwein, T., Reinberg, D., Caparros, M.-L, eds. Epigenetics. New York: Cold Spring Harbor Laboratory Press, 2007
|
31 |
Slotkin, R. K., Martienssen R. : Transposable elements and the epigenetic regulation of the genome. Nature Reviews Genetics, 8(4), 272-285, 2007
DOI
ScienceOn
|
32 |
Waterland, R. A, Jirtle, R. L. : Transposable elements:targets for early nutritional effects on epigenetic gene regulation. Molecular and Cellular Biology, 23(15), 5293-5300, 2003
DOI
ScienceOn
|
33 |
Kaneko-Ishino, T., Kohda, T., Ono, R. and Ishino, F. : hypothesis: the necessity of a monoallelic gene expression mechanism in mammalian development. Cytogenetic and Genome Research, 113(1-4), 24-30, 2006
DOI
ScienceOn
|
34 |
Welshons, W. V., Nagel, S. C. and vom Saal, F. S. :Large effects from small exposures. III. Endocrine mechanisms mediating effects of bisphenol A at levels of human exposure. Endocrinology, 147(6 Suppl), S56-69, 2006
DOI
ScienceOn
|
35 |
Esteller, M. : Epigenetics in cancer. New England Journal of Medicine, 358(11), 1148-1159, 2008
DOI
ScienceOn
|
36 |
Sutherland, J. E., Costa, M. : Epigenetics and the environment. Annals of the New York Academy of Sciences, 983, 151-160, 2003
DOI
ScienceOn
|
37 |
Newbold, R. R., Padilla-Banks, E. and Jefferson, W.N. : Adverse effects of the model environmental estrogen diethylstilbestrol are transmitted to subsequent generations. Endocrinology, 147(6 Suppl), S11-7, 2006
DOI
ScienceOn
|
38 |
Franco, R., Schoneveld, O., Georgakilas, A. G. and Panayiotidis, M. I. Oxidative stress, DNA methylation and carcinogenesis. Cancer Letters, 266(1), 6-11, 2008
DOI
ScienceOn
|
39 |
Basha, M. R., Wei, W., Bakheet, S. A., Benitez, N.,Siddiqi, H. K. and Ge, Y. W., et al. : The fetal basis of amyloidogenesis: exposure to lead and latent overexpression of amyloid precursor protein and beta-amyloid in the aging brain. Journal of Neuroscience, 25(4), 823-829, 2005
DOI
ScienceOn
|
40 |
Maffini, M. V., Rubin, B. S., Sonnenschein, C. and Soto, A. M. : Endocrine disruptors and reproductive health: the case of bisphenol-A. Molecular and Cellular Endocrinology, 254-255, 179-186, 2006
DOI
ScienceOn
|
41 |
WHO/FAO. Diet, nutrition and the prevention of chronic diseases. World Health Organ Tech Rep Ser, 916, i-viii, 1-149, backcover, 2003
|
42 |
Lopez, J., Percharde, M., Coley, H. M., Webb, A. and Crook, T. : The context and potential of epigenetics in oncology. British Journal of Cancer, 100(4), 571- 577, 2009
DOI
ScienceOn
|
43 |
Benbrahim-Tallaa, L., Waterland, R. A., Styblo, M.,Achanzar, W. E., Webber, M. M. and Waalkes, M.P. : Molecular events associated with arsenicinduced malignant transformation of human prostatic epithelial cells: aberrant genomic DNA methylation and K-ras oncogene activation. Toxicology and Applied Pharmacology, 206(3), 288-298, 2005
DOI
ScienceOn
|
44 |
Schnekenburger, M., Talaska, G. and Puga, A. : Chromium cross-links histone deacetylase 1-DNA methyltransferase 1 complexes to chromatin, inhibiting histone-remodeling marks critical for transcriptional activation. Molecular and Cellular Biology, 27(20), 7089-7101, 2007
DOI
ScienceOn
|
45 |
Baccarelli, A., Wright, R. O., Bollati, V., Tarantini, L., Litonjua, A. A. and Suh, H. H., et al. : Rapid DNA methylation changes after exposure to traffic particles. American Journal of Respiratory and Critical Care Medicine, 179(7), 572-578, 2009
DOI
ScienceOn
|
46 |
Perera, F., Tang, W. Y., Herbstman, J., Tang, D.,Levin, L. and Miller, R., et al. : R elation of DNA methylation of 5'-CpG island of ACSL3 to transplacental exposure to airborne polycyclic aromatic hydrocarbons and childhood asthma. Public Library of Science ONE, 4(2), e4488, 2009
DOI
|
47 |
Zeisel, S. H. : Importance of methyl donors during reproduction. American Journal of Clinical Nutrition, 89(2), 673S-677S, 2009
DOI
ScienceOn
|
48 |
Wu, J., Basha, M. R., Brock, B., Cox, D. P., Cardozo-Pelaez, F. and McPherson, C. A, et al. : Alzheimer's disease (AD)-like pathology in aged monkeys after infantile exposure to environmental metal lead (Pb): evidence for a developmental origin and environmentallink for AD. Journal of Neuroscience, 28(1), 3-9, 2008
DOI
ScienceOn
|
49 |
Ke, Q., Davidson, T., Chen, H., Kluz, T. and Costa, M. : Alterations of histone modifications and transgene silencing by nickel chloride. Carcinogenesis, 27(7), 1481-1488, 2006
DOI
ScienceOn
|
50 |
Weaver, I. C., Cervoni, N., Champagne, F. A., D'Alessio, A. C., Sharma, S. and Seckl, J. R., et al. :Epigenetic programming by maternal behavior. Nature Neuroscience, 7(8), 847-854, 2004
DOI
ScienceOn
|
51 |
Kondo, K., Takahashi, Y., Hirose, Y., Nagao, T.,Tsuyuguchi, M. and Hashimoto, M., et al. : The reduced expression and aberrant methylation of p16(INK4a) in chromate workers with lung cancer. Lung Cancer, 53(3), 295-302, 2006
DOI
ScienceOn
|
52 |
Anway, M. D., Leathers, C. and Skinner, M. K. :Endocrine disruptor vinclozolin induced epigenetic transgenerational adult-onset disease. Endocrinology, 147(12), 5515-5523, 2006
DOI
ScienceOn
|
53 |
Bird, A. : Perceptions of epigenetics. Nature, 447(7143), 396-398, 2007
DOI
ScienceOn
|
54 |
Chanda, S., Dasgupta, U. B., Guhamazumder, D.,Gupta, M., Chaudhuri, U. and Lahiri, S., et al. :DNA hypermethylation of promoter of gene p53 and p16 in arsenic-exposed people with and without malignancy. Toxicological Science, 89(2), 431-437, 2006
DOI
ScienceOn
|
55 |
Chen, H., Liu, J., Zhao, C. Q,, Diwan, B. A., Merrick,B. A. and Waalkes, M. P. : Association of cmyc overexpression and hyperproliferation with arsenite- induced malignant transformation. Toxicology and Applied Pharmacology, 175(3), 260-268, 2001
DOI
ScienceOn
|
56 |
Ramirez, T., Brocher, J., Stopper, H. and Hock, R. :Sodium arsenite modulates histone acetylation, histone deacetylase activity and HMGN protein dynamics in human cells. Chromosoma, 117(2), 147-157, 2008
DOI
ScienceOn
|
57 |
Park, S. K., O'Neill, M. S., Vokonas, P. S., Sparrow, D., Spiro, A., 3rd and Tucker, K. L., et al. :Traffic-related particles are associated with elevated homocysteine: the VA normative aging study. American Journal of Respiratory and Critical Care Medicine, 178(3), 283-289, 2008
DOI
ScienceOn
|
58 |
Rossiello, M. R., Aresta, A. M., Prisco, M. and Kanduc, D. : DNA hypomethylation during liver cell proliferation induced by a single dose of lead nitrate. Bollettino Societa Italiana Biologia Sperimentale, 67(12), 993-997, 1991
|
59 |
Dolinoy, D. C., Huang, D. and Jirtle, R. L. : Maternal nutrient supplementation counteracts bisphenol A-induced DNA hypomethylation in early development. Proceedings of the National Academy of Sciences of the United States of America, 104(32), 13056-13061, 2007
DOI
ScienceOn
|
60 |
Chai, C. Y., Huang, Y. C., Hung, W. C., Kang, W.Y. and Chen, W. T. : Arsenic salts induced autophagic cell death and hypermethylation of DAPK promoter in SV-40 immortalized human uroepithelial cells. Toxicology Letters, 173(1), 48-56, 2007
DOI
ScienceOn
|
61 |
Takiguchi, M., Achanzar, W. E, Qu, W., Li, G. and Waalkes, M. P. : Effects of cadmium on DNA-(Cytosine-5) methyltransferase activity and DNA methylation status during cadmium-induced cellular transformation. Experimental Cell Research, 286(2), 355-365, 2003
DOI
ScienceOn
|
62 |
Jiang, G., Xu, L., Song, S., Zhu, C., Wu, Q. and Zhang. L., et al. : Effects of long-term low-dose cadmium exposure on genomic DNA methylation in human embryo lung fibroblast cells. Toxicology, 244(1), 49-55, 2008
DOI
ScienceOn
|
63 |
NTP, CERHR. NTP-CERHR Monograph on the potential human reproductive and developmental effects of bisphenol A. National Institutes of Health 2008
|
64 |
Bunger, M. K., Glover, E., Moran, S. M., Walisser, J. A., Lahvis, G. P. and Hsu, E. L., et al. : Abnormal liver development and resistance to 2,3,7,8-tetrachlorodibenzo-p-dioxin toxicity in mice carrying a mutation in the DNA-binding domain of the aryl hydrocarbon receptor. Toxicological Science, 106(1), 83-92, 2008
DOI
ScienceOn
|
65 |
Pilsner, J. R., Liu, X., Ahsan, H., Ilievski, V., Slavkovich, V. and Levy, D., et al. : Genomic methylation of peripheral blood leukocyte DNA: influences of arsenic and folate in Bangladeshi adults. American Journal of Clinical Nutrition, 86(4), 1179-1186, 2007
DOI
ScienceOn
|
66 |
Schanen, N. C. : Epigenetics of autism spectrum disorders. Human Molecular Genetics, 15 Spec No 2, R138-50, 2006
DOI
ScienceOn
|
67 |
Santos, F., Hendrich, B., Reik, W., Dean, W. : Dynamic reprogramming of DNA methylation in the early mouse embryo. Developmental Biology, 241(1), 172-182, 2002
DOI
ScienceOn
|
68 |
Cui, X., Wakai, T., Shirai, Y., Hatakeyama, K. and Hirano, S. : Chronic oral exposure to inorganic arsenate interferes with methylation status of p16INK4a and RASSF1A and induces lung cancer in A/J mice. Toxicological Science, 91(2), 372-381, 2006
DOI
ScienceOn
|
69 |
Barker, D. J. : Fetal origins of coronary heart disease. British Medical Journal, 311(6998), 171-174, 1995
DOI
ScienceOn
|
70 |
Ercal, N., Gurer-Orhan, H., Aykin-Burns, N. : Toxic metals and oxidative stress part I: mechanisms involved in metal-induced oxidative damage. Current Topics in Medicinal Chemistry, 1(6), 529-539, 2001
DOI
ScienceOn
|
71 |
Campion, J., Milagro, F. I. and Martinez. J. A. :Individuality and epigenetics in obesity. Obes Rev, 10(4), 383-392, 2009
DOI
ScienceOn
|
72 |
Fish, E. W., Shahrokh, D., Bagot, R., Caldji, C.,Bredy, T. and Szyf, M., et al. : Epigenetic programming of stress responses through variations in maternal care. Annals of the New York Academy of Sciences, 1036, 167-180, 2004
DOI
ScienceOn
|
73 |
Barker, D. J., Osmond, C., Golding, J., Kuh, D.,Wadsworth, M. E. : Growth in utero, blood pressure in childhood and adult life, and mortality from cardiovascular disease. British Medical Journal, 298(6673), 564-567, 1989
DOI
ScienceOn
|
74 |
Zhang, A. H., Bin, H. H., Pan, X. L., Xi and X. G.. :Analysis of p16 gene mutation, deletion and methylation in patients with arseniasis produced by indoor unventilated-stove coal usage in Guizhou, China. Journal of Toxicology and Environmental Health, 70(11), 970-975, 2007
DOI
ScienceOn
|
75 |
Pilsner, J. R., Liu, X., Ahsan, H., Ilievski, V., Slavkovich, V. and Levy, D., et al. : Folate deficiency, hyperhomocysteinemia, low urinary creatinine, and hypomethylation of leukocyte DNA are risk factors for arsenic-induced skin lesions. Environmental Health Perspectives, 117(2), 254-260, 2009
DOI
ScienceOn
|
76 |
Robertson, K. D., Wolffe, A. P. : DNA methylation in health and disease. Nature Reviews Genetics, 1(1), 11-19, 2000
DOI
ScienceOn
|
77 |
Reichard, J. F., Schnekenburger, M. and Puga, A. : Long term low-dose arsenic exposure induces loss of DNA methylation. Biochemical and Biophysical Research Communications, 352(1), 188-192, 2007
DOI
ScienceOn
|
78 |
Huang, D., Zhang, Y., Qi, Y., Chen, C. and Ji, W. :Global DNA hypomethylation, rather than reactive oxygen species (ROS), a potential facilitator of cadmium- stimulated K562 cell proliferation. Toxicology Letters, 179(1), 43-47, 2008
DOI
ScienceOn
|
79 |
Kanduc, D., Rossiello, M. R., Aresta, A., Cavazza, C., Quagliariello, E. and Farber, E. : Transitory DNA hypomethylation during liver cell proliferation induced by a single dose of lead nitrate. Archives of Biochemistry and Biophysics, 286(1), 212-216, 1991
DOI
ScienceOn
|
80 |
Tarantini, L., Bonzini, M., Apostoli, P., Pegoraro, V., Bollati, V. and Marinelli. B, et al. : E f f ects o f particulate matter on genomic DNA methylation content and iNOS promoter methylation. Environmental Environmental Health Perspectives, 117(2), 217-222, 2009
DOI
|
81 |
Zawia, N. H., Lahiri, D. K., Cardozo-Pelaez, F. : Epigenetics, oxidative stress, and Alzheimer disease. Free Radical Biology & Medicine, 46(9), 1241-1249, 2009
DOI
ScienceOn
|
82 |
Brook, R. D., Franklin, B., Cascio, W., Hong, Y.,Howard, G., Lipsett, M., et al. : Air pollution and cardiovascular disease: a statement for healthcare professionals from the Expert Panel on Population and Prevention Science of the American Heart Association. Circulation, 109(21), 2655-2671, 2004
DOI
ScienceOn
|
83 |
Goering, P. L., Aposhian, H. V., Mass, M. J., Cebrian, M., Beck, B. D.,Waalkes, M. P. : The enigma of arsenic carcinogenesis: role of metabolism. Toxicological Science, 49(1), 5-14, 1999
DOI
ScienceOn
|
84 |
Krause, E. T., Honarmand, M., Wetzel, J. and Naguib, M. : Early fasting is long lasting: differences in early nutritional conditions reappear under stressful conditions in adult female zebra finches. Public Library of Science ONE, 4(3), e5015-0000, 2009
|
85 |
Barker, D. J., Eriksson, J. G., Forsen, T. and Osmond, C. : Fetal origins of adult disease: s trength of effects and biological basis. International Journal of Epidemiology, 31(6), 1235-1239, 2002
DOI
ScienceOn
|
86 |
Foley, D. L., Craig, J. M., Morley, R., Olsson, C. A.,Dwyer, T. and Smith, K., et al. : Prospects for epigenetic epigenetic epidemiology. American Journal of Epidemiology, 169(4), 389-400, 2009
DOI
ScienceOn
|
87 |
Vickers, M. H., Gluckman, P. D., Coveny, A. H.,Hofman, P. L., Cutfield, W. S., Gertler, A., et al. :Neonatal leptin treatment reverses developmental programming. Endocrinology, 146(10), 4211-4216, 2005
DOI
ScienceOn
|
88 |
Simmons, R. A. : Developmental origins of beta-cell failure in type 2 diabetes: the role of epigenetic mechanisms. Pediatric Research, 61(5 Pt 2), 64R-67R, 2007
DOI
ScienceOn
|
89 |
Tang, W. Y. and Ho, S. M. : Epigenetic reprogramming and imprinting in origins of disease. Reviews in Endocrine & Metabolic Disorders, 8(2), 173-182, 2007
DOI
ScienceOn
|
90 |
84Li, S., Hansman, R., Newbold, R., Davis, B., McLachlan, J. A. and Barrett, J. C. : Neonatal diethylstilbestrol exposure induces persistent elevation of c-fos expression and hypomethylation in its exon-4 in mouse uterus. Molecular Carcinogenesis, 38(2), 78-84, 2003
DOI
ScienceOn
|
91 |
Waggoner, D. : Mechanisms of disease: epigenesis. Seminars in Pediatric Neurology, 14(1), 7-14, 2007
DOI
ScienceOn
|
92 |
Ho, S. M., Tang, W. Y., Belmonte, de Frausto, J. and Prins, G. S. : Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4. Cancer Research, 66(11), 5624-5632, 2006
DOI
ScienceOn
|
93 |
Weaver, I. C., Champagne, F. A., Brown, S. E., Dymov, S., Sharma, S. and Meaney, M. J., et al.:Reversal o f maternal programming o f stress responses in adult offspring through methyl supplementation: altering epigenetic marking later in life. Journal of Neuroscience, 25(47), 11045-11054, 2005
DOI
ScienceOn
|
94 |
Hales, C. N., Barker, D. J., Clark, P. M., Cox, L. J.,Fall, C. and Osmond, C., et al. : Fetal and infant growth and impaired glucose tolerance at age 64. British Medical Journal, 303(6809), 1019-1022, 1991
DOI
ScienceOn
|
95 |
Xu, Y., Wang, Y., Zheng, Q., Li, X., Li, B., Jin, Y.,et al. : Association of oxidative stress with arsenic methylation in chronic arsenic-exposed children and adults. Toxicology and Applied Pharmacology, 232(1), 142-149, 2008
DOI
ScienceOn
|
96 |
Crews, D., Gore, A. C., Hsu, T. S., Dangleben, N.L., Spinetta, M. and Schallert, T., et al. : Transgenerational epigenetic imprints on mate preference. Proceedings of the National Academy of Sciences of the United States of America, 104(14), 5942-5946, 2007
DOI
ScienceOn
|
97 |
Li, S., Washburn, K. A., Moore, R., Uno, T., Teng, C. and Newbold, R. R., et al. : Developmental exposure to diethylstilbestrol elicits demethylation of estrogen-responsive lactoferrin gene in mouse uterus. Cancer Research, 57(19), 4356-4359, 1997
ScienceOn
|
98 |
Baccarelli, A., Cassano, P. A., Litonjua, A., Park, S.K., Suh, H. and Sparrow, D., et al. : Cardiac autonomic dysfunction: effects from particulate air pollution and protection by dietary methyl nutrients and metabolic polymorphisms. Circulation, 117(14), 1802-1809, 2008
DOI
ScienceOn
|
99 |
Anway, M. D., Cupp, A. S., Uzumcu, M. and Skinner, M. K. : Epigenetic transgenerational actions of endocrine disruptors and male fertility. Science, 308(5727), 1466-1469, 2005
DOI
ScienceOn
|
100 |
Belinsky, S. A., Snow, S. S., Nikula, K. J., Finch, G.L., Tellez, C. S. and Palmisano, W. A. : Aberrant CpG island methylation of the p16(INK4a) and estrogen receptor genes in rat lung tumors induced by particulate carcinogens. Carcinogenesis, 23(2), 335-339, 2002
DOI
ScienceOn
|
101 |
Prins, G. S., Tang, W. Y., Belmonte, J., Ho and S.M. Perinatal exposure to oestradiol and bisphenol A alters the prostate epigenome and increases susceptibility to carcinogenesis. Basic Clin Pharmacol Toxicol, 102(2), 134-138, 2008
DOI
ScienceOn
|
102 |
Benbrahim-Tallaa, L., Waterland, R. A., Dill, A. L.,Webber, M. M. and Waalkes, M. P. : Tumor suppressor gene inactivation during cadmium-induced malignant transformation of human prostate cells correlates with overexpression of de novo DNA methyltransferase. Environmental Health Perspectives, 115(10), 1454-1459, 2007
ScienceOn
|