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Molecular Mechanisms of Neutrophil Activation in Acute Lung Injury
급성 폐손상에서 호중구 활성화의 분자학적 기전
Yum, Ho-Kee
(Department of Internal Medicine, School of Medicine, Inje University)
Publication Information
Tuberculosis and Respiratory Diseases
/
v.53, no.6
, 2002 , pp. 595-611
More about this Journal
More about this Journal
Frequency : Quarterly
Publisher : The Korean Academy of Tuberculosis and Respiratory Diseases
KISTI Subject :
SCOPUS Subject :
Medicine (miscellaneous)
,
Pulmonary and Respiratory Medicine
Web of Science Subject :
Critical Care Medicine
,
Respiratory System
Abstract
Akt/PKB protein kinase B, ALI acute lung injury, ARDS acute respiratory distress syndrome, CREB C-AMP response element binding protein, ERK extracelluar signal-related kinase, fMLP fMet-Leu-Phe, G-CSF granulocyte colony-stimulating factor, IL interleukin, ILK integrin-linked kinase, JNK Jun N-terminal kinase, LPS lipopolysaccharide, MAP mitogen-activated protein, MEK MAP/ERK kinase, MIP-2 macrophage inflammatory protein-2, MMP matrix metalloproteinase, MPO myeloperoxidase, NADPH nicotinamide adenine dinucleotide phosphate, NE neutrophil elastase, NF-kB nuclear factor-kappa B, NOS nitric oxide synthase, p38 MAPK p38 mitogen activated protein kinase, PAF platelet activating factor, PAKs P21-activated kinases, PMN polymorphonuclear leukocytes, PI3-K phosphatidylinositol 3-kinase, PyK proline-rich tyrosine kinase, ROS reactive oxygen species, TNF-
tumor necrosis factor-a.
Keywords
Acute lung injury; Neutrophil; Signal transduction;
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