Stimulation of eNOS-Ser617 Phosphorylation by Fluid Shear Stress in Endothelial Cells |
Boo, Yong-Chool (Department of Molecular Medicine, Kyungpook National University School of Medicine) |
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Endothelial NO synthase phosphorylated at SER635 produces NO without requiring intracellular calcium increase.
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Identification of regulatory sites of phosphorylation of the bovine endothelial nitric-oxide synthase at serine 617 and serine 635
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Akt mediates cytoprotection of endothelial cells by vascular endothelial growth factor in an anchorage-dependent manner
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Mutations in the catalytic subunit of cAMP-dependent protein kinase result in unregulated biological activity
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Regulation of endothelium-derived nitric oxide production by the protein kinase Akt
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Flow Shear Stress Stimulates Gab1 Tyrosine Phosphorylation to Mediate Protein Kinase B and Endothelial Nitric-oxide Synthase Activation in Endothelial Cells
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Mechanism of activation and function of protein kinase B
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A mechanosensory complex that mediates the endothelial cell response to fluid shear stress
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DOI ScienceOn |
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Shear stress stimulates phosphorylation of endothelial nitric-oxide synthase at Ser1179 by Akt-independent mechanisms: role of protein kinase A
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Shear stress stimulates phosphorylation of eNOS at Ser(635) by a protein kinase Adependent mechanism
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Endothelial nitric oxide synthase activity is linked to its presence at cell-cell contacts
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PECAM-1 interacts with nitric oxide synthase in human endothelial cells: implication for flow-induced nitric oxide synthase activation
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Role of PECAM-1 in the shear-stress-induced activation of Akt and the endothelial nitric oxide synthase (eNOS) in endothelial cells
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DOI ScienceOn |
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Colocalization of eNOS and the catalytic subunit of PKA in endothelial cell junctions: a clue for regulated NO production
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Phosphorylation of Thr(495) regulates Ca(2+)/calmodulin-dependent endothelial nitric oxide synthase activity
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Dephosphorylation of endothelial nitric-oxide synthase by vascular endothelial growth factor. Implications for the vascular responses to cyclosporin A
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Compensatory phosphorylation and protein-protein interactions revealed by loss
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Fluid shear stress and the vascular endothelium: for better and for worse
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Atherosclerosis
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DOI ScienceOn |
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Carotid bifurcation atherosclerosis. Quantitative correlation of plaque localization with flow velocity profiles and wall shear stress
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DOI ScienceOn |
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The role of shear stress in the pathogenesis of atherosclerosis
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Intracellular pH and tyrosine phosphorylation but not calcium determine shear stress-induced nitric oxide production in native endothelial cells
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DOI ScienceOn |
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Cyclic strain increases endothelial nitric oxide synthase activity
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Activation of nitric oxide synthase in endothelial cells by Akt- dependent phosphorylation
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Shear stress regulates endothelial nitric oxide synthase expression through c-Src by divergent signaling pathways
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DOI ScienceOn |
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Flow-dependent regulation of endothelial nitric oxide synthase: role of protein kinases
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eNOS at a glance
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