1 |
Goldring MB (2000) The role of the chondrocyte in osteoarthritis. Arthritis Rheum 43, 1916-1926
DOI
|
2 |
Goldring MB and Goldring SR (2007) Osteoarthritis. J Cell Physiol 213, 626-634
DOI
|
3 |
Goldring SR and Goldring MB (2004) The role of cytokines in cartilage matrix degeneration in osteoarthritis. Clin Orthop Relat Res, S27-36
|
4 |
Rosenberg JH, Rai V, Dilisio MF and Agrawal DK (2017) Damage-associated molecular patterns in the pathogenesis of osteoarthritis: potentially novel therapeutic targets. Mol Cell Biochem 434, 171-179
DOI
|
5 |
Rosenberg JH, Rai V, Dilisio MF, Sekundiak TD and Agrawal DK (2017) Increased expression of damageassociated molecular patterns (DAMPs) in osteoarthritis of human knee joint compared to hip joint. Mol Cell Biochem 436, 59-69
DOI
|
6 |
Hwang HS, Park SJ, Cheon EJ, Lee MH and Kim HA (2015) Fibronectin fragment-induced expression of matrix metalloproteinases is mediated by MyD88-dependent TLR-2 signaling pathway in human chondrocytes. Arthritis Res Ther 17, 320
DOI
|
7 |
Hwang HS, Lee MH and Kim HA (2019) Fibronectin fragment inhibits xylosyltransferase-1 expression by regulating Sp1/Sp3-dependent transcription in articular chondrocytes. Osteoarthritis Cartilage 27, 833-843
DOI
|
8 |
Kim HA, Cho ML, Choi HY et al (2006) The catabolic pathway mediated by Toll-like receptors in human osteoarthritic chondrocytes. Arthritis Rheum 54, 2152-2163
DOI
|
9 |
Xiao TS and Fitzgerald KA (2013) The cGAS-STING pathway for DNA sensing. Mol Cell 51, 135-139
DOI
|
10 |
Bobacz K, Sunk IG, Hofstaetter JG et al (2007) Toll-like receptors and chondrocytes: the lipopolysaccharideinduced decrease in cartilage matrix synthesis is dependent on the presence of toll-like receptor 4 and antagonized by bone morphogenetic protein 7. Arthritis Rheum 56, 1880-1893
DOI
|
11 |
Bai J, Cervantes C, Liu J et al (2017) DsbA-L prevents obesity-induced inflammation and insulin resistance by suppressing the mtDNA release-activated cGAS-cGAMPSTING pathway. Proc Natl Acad Sci U S A 114, 12196-12201
DOI
|
12 |
Li T and Chen ZJ (2018) The cGAS-cGAMP-STING pathway connects DNA damage to inflammation, senescence, and cancer. J Exp Med 215, 1287-1299
DOI
|
13 |
Dou Z, Ghosh K, Vizioli MG et al (2017) Cytoplasmic chromatin triggers inflammation in senescence and cancer. Nature 550, 402-406
DOI
|
14 |
Barber GN (2015) STING: infection, inflammation and cancer. Nat Rev Immunol 15, 760-770
DOI
|
15 |
Kawai T and Akira S (2011) Toll-like receptors and their crosstalk with other innate receptors in infection and immunity. Immunity 34, 637-650
DOI
|
16 |
Chen AF, Davies CM, De Lin M and Fermor B (2008) Oxidative DNA damage in osteoarthritic porcine articular cartilage. J Cell Physiol 217, 828-833
DOI
|
17 |
Grishko VI, Ho R, Wilson GL and Pearsall AWt (2009) Diminished mitochondrial DNA integrity and repair capacity in OA chondrocytes. Osteoarthritis Cartilage 17, 107-113
DOI
|
18 |
Ma Z and Damania B (2016) The cGAS-STING Defense Pathway and Its Counteraction by Viruses. Cell Host Microbe 19, 150-158
DOI
|
19 |
Medzhitov R (2007) TLR-mediated innate immune recognition. Semin Immunol 19, 1-2
DOI
|
20 |
Brubaker SW, Bonham KS, Zanoni I and Kagan JC (2015) Innate immune pattern recognition: a cell biological perspective. Annu Rev Immunol 33, 257-290
DOI
|
21 |
Heinhuis B, Koenders MI, van de Loo FA et al (2010) IL-32gamma and Streptococcus pyogenes cell wall fragments synergise for IL-1-dependent destructive arthritis via upregulation of TLR-2 and NOD2. Ann Rheum Dis 69, 1866-1872
DOI
|
22 |
Hwang HS, Lee MH, Choi MH and Kim HA (2019) NOD2 signaling pathway is involved in fibronectin fragment-induced pro-catabolic factor expressions in human articular chondrocytes. BMB Rep [Epub ahead of print]
|
23 |
Crow YJ (2015) Type I interferonopathies: mendelian type I interferon up-regulation. Curr Opin Immunol 32, 7-12
DOI
|
24 |
Joosten LA, Heinhuis B, Abdollahi-Roodsaz S et al (2008) Differential function of the NACHT-LRR (NLR) members Nod1 and Nod2 in arthritis. Proc Natl Acad Sci U S A 105, 9017-9022
DOI
|
25 |
Vieira SM, Cunha TM, Franca RF et al (2012) Joint NOD2/RIPK2 signaling regulates IL-17 axis and contributes to the development of experimental arthritis. J Immunol 188, 5116-5122
DOI
|
26 |
Sun L, Wu J, Du F, Chen X and Chen ZJ (2013) Cyclic GMP-AMP synthase is a cytosolic DNA sensor that activates the type I interferon pathway. Science 339, 786-791
DOI
|
27 |
Kawane K, Ohtani M, Miwa K et al (2006) Chronic polyarthritis caused by mammalian DNA that escapes from degradation in macrophages. Nature 443, 998-1002
DOI
|
28 |
Lan YY, Londono D, Bouley R, Rooney MS and Hacohen N (2014) Dnase2a deficiency uncovers lysosomal clearance of damaged nuclear DNA via autophagy. Cell Rep 9, 180-192
DOI
|
29 |
Ahn J, Gutman D, Saijo S and Barber GN (2012) STING manifests self DNA-dependent inflammatory disease. Proc Natl Acad Sci U S A 109, 19386-19391
DOI
|
30 |
Kim HA, Lee YJ, Seong SC, Choe KW and Song YW (2000) Apoptotic chondrocyte death in human osteoarthritis. J Rheumatol 27, 455-462
|
31 |
Kim J, Xu M, Xo R et al (2010) Mitochondrial DNA damage is involved in apoptosis caused by proinflammatory cytokines in human OA chondrocytes. Osteoarthritis Cartilage 18, 424-432
DOI
|
32 |
Campbell IK, Ianches G and Hamilton JA (1993) Production of macrophage colony-stimulating factor (M-CSF) by human articular cartilage and chondrocytes. Modulation by interleukin-1 and tumor necrosis factor alpha. Biochim Biophys Acta 1182, 57-63
DOI
|
33 |
Leizer T, Cebon J, Layton JE and Hamilton JA (1990) Cytokine regulation of colony-stimulating factor production in cultured human synovial fibroblasts: I. Induction of GM-CSF and G-CSF production by interleukin-1 and tumor necrosis factor. Blood 76, 1989-1996
DOI
|
34 |
Hamilton JA, Filonzi EL and Ianches G (1993) Regulation of macrophage colony-stimulating factor (M-CSF) production in cultured human synovial fibroblasts. Growth Factors 9, 157-165
DOI
|
35 |
Campbell IK, Novak U, Cebon J, Layton JE and Hamilton JA (1991) Human articular cartilage and chondrocytes produce hemopoietic colony-stimulating factors in culture in response to IL-1. J Immunol 147, 1238-1246
|
36 |
Achuthan A, Cook AD, Lee MC et al (2016) Granulocyte macrophage colony-stimulating factor induces CCL17 production via IRF4 to mediate inflammation. J Clin Invest 126, 3453-3466
DOI
|
37 |
Lee MC, Saleh R, Achuthan A et al (2018) CCL17 blockade as a therapy for osteoarthritis pain and disease. Arthritis Res Ther 20, 62
DOI
|
38 |
Cook AD, Pobjoy J, Steidl S et al (2012) Granulocytemacrophage colony-stimulating factor is a key mediator in experimental osteoarthritis pain and disease development. Arthritis Res Ther 14, R199
DOI
|
39 |
Yang YH and Hamilton JA (2001) Dependence of interleukin-1-induced arthritis on granulocyte-macrophage colony-stimulating factor. Arthritis Rheum 44, 111-119
DOI
|
40 |
Cook AD, Pobjoy J, Sarros S et al (2013) Granulocytemacrophage colony-stimulating factor is a key mediator in inflammatory and arthritic pain. Ann Rheum Dis 72, 265-270
DOI
|
41 |
Miller JC, Ma Y, Bian J et al (2008) A critical role for type I IFN in arthritis development following Borrelia burgdorferi infection of mice. J Immunol 181, 8492-8503
DOI
|
42 |
Roelofs MF, Wenink MH, Brentano F et al (2009) Type I interferons might form the link between Toll-like receptor (TLR) 3/7 and TLR4-mediated synovial inflammation in rheumatoid arthritis (RA). Ann Rheum Dis 68, 1486-1493
DOI
|
43 |
Triantaphyllopoulos KA, Williams RO, Tailor H and Chernajovsky Y (1999) Amelioration of collagen-induced arthritis and suppression of interferon-gamma, interleukin-12, and tumor necrosis factor alpha production by interferon-beta gene therapy. Arthritis Rheum 42, 90-99
DOI
|
44 |
Wong T, Majchrzak B, Bogoch E, Keystone EC and Fish EN (2003) Therapeutic implications for interferon-alpha in arthritis: a pilot study. J Rheumatol 30, 934-940
|
45 |
Burdette DL and Vance RE (2013) STING and the innate immune response to nucleic acids in the cytosol. Nat Immunol 14, 19-26
DOI
|