1 |
Sugisaki H, Yamanaka K, Kakeda M, Kitagawa H, Tanaka K, Watanabe K, et al. 2009. Increased interferon-g, interleukin- 12p40 and IL-8 production in Propionibacterium acnes-treated peripheral blood mononuclear cells from patient with acne vulgaris: host response but not bacterial species is the determinant factor of the disease. J. Dermatol. Sci. 55: 47-52.
DOI
|
2 |
Wang YY, Ryu AR, Jin S, Jeon YM, Lee MY. 2017. Chlorin e6-mediated photodynamic therapy suppresses P. acnesinduced inflammatory response via NF and MAPKs signaling pathway. PLoS One 12: e0170599.
DOI
|
3 |
Molina M, Cid VJ, Martin H. 2010. Fine regulation of Saccharomyces cerevisiae MAPK pathways by post-translational modifications. Yeast 27: 503-511.
DOI
|
4 |
Lee AY, Lee S, Kim HY, Lee S, Cho EJ. 2016. Anti-inflammatory effects of luteolin and luteoloside from Taraxacum coreanum in RAW264.7 macrophage cells. Appl. Biol. Chem. 59: 747-754.
DOI
|
5 |
Bonizzi G, Karin M. 2004. The two NF- activation pathways and their role in innate and adaptive immunity. Trends Immunol. 25: 280-288.
DOI
|
6 |
Huang WC, Tsai TH, Chuang LT, Li YY, Zouboulis CC, Tsai PJ. 2014. Anti-bacterial and anti-inflammatory properties of capric acid against Propionibacterium acnes: a comparative study with lauric acid. J. Dermatol. Sci. 73: 232-240.
DOI
|
7 |
Wu J, Zhang H, Hu B, Yang L, Wang P, Wang F, et al. 2016. Coptisine from Coptis chinensis inhibits production of inflammatory mediators in lipopolysaccharide-stimulated RAW 264.7 murine macrophage cells. Eur. J. Pharmacol. 780: 106-114.
DOI
|
8 |
Turner MD, Nedjai B, Hurst T, Pennington DJ. 2014. Cytokines and chemokines: at the crossroads of cell signalling and inflammatory disease. Biochim. Biophys. Acta 1843: 2563- 2582.
DOI
|
9 |
Nagy I, Pivarcsi A, Koreck A, Szell M, Urban E, Kemeny L. 2005. Distinct strains of Propionibacterium acnes induce selective human -defensin-2 and interleukin-8 expression in human keratinocytes through Toll-like receptors. J. Invest. Dermatol. 124: 931-938.
DOI
|
10 |
Lee WR, Kim KH, An HJ, Kim JY, Chang YC, Chung H, et al. 2014. The protective effects of melittin on Propionibacterium acnes-induced inflammatory responses in vitro and in vivo. J. Invest. Dermatol. 134: 1922-1930.
DOI
|
11 |
Viatour P, Merville MP, Bours V, Chariot A. 2005. Phosphorylation of NF- and proteins: implications in cancer and inflammation. Trends Biochem. Sci. 30: 43-52.
DOI
|
12 |
Enk R, Ehehalt R, Graham JE, Bierhaus A, Remppis A, Greten HJ. 2007. Differential effect of Rhizoma coptidis and its main alkaloid compound berberine on TNF- induced NF translocation in human keratinocytes. J. Ethnopharmacol. 109: 170-175.
DOI
|
13 |
Li JY, Wang XB, Luo JG, Kong LY. 2015. Seasonal variation of alkaloid contents and anti-inflammatory activity of Rhizoma coptidis based on fingerprints combined with chemometrics methods. J. Chromatogr. Sci. 53: 1131-1139.
DOI
|
14 |
Grange PA, Raingeaud J, Calvez V, Dupin N. 2009. Nicotinamide inhibits Propionibacterium acnes-induced IL-8 production in keratinocytes through the NF- and MAPK pathways. J. Dermatol. Sci. 56: 106-112.
DOI
|
15 |
Kim JM, Jung HA, Choi JS, Lee NG. 2010. Identification of anti-inflammatory target genes of Rhizoma coptidis extract in lipopolysaccharide-stimulated RAW264.7 murine macrophagelike cells. J. Ethnopharmacol. 130: 354-362.
DOI
|
16 |
Teng H, Choi YH. 2014. Optimization of ultrasonic-assisted extraction of bioactive alkaloid compounds from rhizoma coptidis (Coptis chinensis Franch.) using response surface methodology. Food Chem. 142: 299-305.
DOI
|
17 |
Yan D, Jin C, Xiao XH, Dong XP. 2008. Antimicrobial properties of berberines alkaloids in Coptis chinensis Franch by microcalorimetry. J. Biochem. Biophys. Methods 70: 845-849.
DOI
|
18 |
Zou K, Li Z, Zhang Y, Zhang HY, Li B, Zhu WL, et al. 2017. Advances in the study of berberine and its derivatives: a focus on anti-inflammatory and anti-tumor effects in the digestive system. Acta Pharmacol. Sin. 38: 157-167.
DOI
|
19 |
Jabbarzadeh Kaboli P, Rahmat A, Ismail P, Ling KH. 2014. Targets and mechanisms of berberine, a natural drug with potential to treat cancer with special focus on breast cancer. Eur. J. Pharmacol. 740: 584-595.
DOI
|
20 |
Roux PP, Blenis J. 2004. ERK and p38 MAPK-activated protein kinases: a family of protein kinases with diverse biological functions. Microbiol. Mol. Biol. Rev. 68: 320-344.
DOI
|
21 |
Remppis A, Bea F, Greten HJ, Buttler A, Wang H, Zhou Q, et al. 2010. Rhizoma coptidis inhibits LPS-induced MCP-1/ CCL2 production in murine macrophages via an AP-1 and NF- -dependent pathway. Mediators Inflamm. 2010: 194896.
|
22 |
Omer H, McDowell A, Alexeyev OA. 2017. Understanding the role of Propionibacterium acnes in acne vulgaris: the critical importance of skin sampling methodologies. Clin. Dermatol. 35: 118-129.
DOI
|
23 |
Dessinioti C, Katsambas AD. 2010. The role of Propionibacterium acnes in acne pathogenesis: facts and controversies. Clin. Dermatol. 28: 2-7.
DOI
|
24 |
Kim J, Ochoa MT, Krutzik SR, Takeuchi O, Uematsu S, Legaspi AJ, et al. 2002. Activation of Toll-like receptor 2 in acne triggers inflammatory cytokine responses. J. Immunol. 169: 1535-1541.
DOI
|
25 |
Kollisch G, Kalali BN, Voelcker V, Wallich R, Behrendt H, Ring J, et al. 2005. Various members of the Toll-like receptor family contribute to the innate immune response of human epidermal keratinocytes. Immunology 114: 531-541.
DOI
|
26 |
Farrar MD, Ingham E. 2004. Acne: inflammation. Clin. Dermatol. 22: 380-384.
DOI
|