1 |
Cuzzocrea, S., McDonald, M.C., Mazzon, E., Siriwardena, D., Costantino, G., Fulia, F., Cucinotta, G., Gitto, E., Cordaro, S., Barberi, I., et al. (2000). Effects of tempol, a membrane-permeable radical scavenger, in a gerbil model of brain injury. Brain Res.875, 96-106
DOI
ScienceOn
|
2 |
Han, S.S., Kim, K., Hahm, E.R., Park, C.H., Kimler, B.F., Lee, S.J., Lee, S.H., Kim, W.S., Jung, C.W., Park, K., et alK (2005). Arsenic trioxide represses constitutive activation of NF-kappaB and COX-2 expression in human acute myeloid leukemia, HL-60. J. Cell. Biochem. 94, 695-707
DOI
ScienceOn
|
3 |
Li, J.J., Tang, Q., Li, Y., Hu, B.R., Ming, Z.Y., Fu, Q., Qian, J.Q., and Xiang, J.Z. (2006). Role of oxidative stress in the apoptosis of hepatocellular carcinoma induced by combination of arsenic trioxide and ascorbic acid. Acta. Pharmacol. Sin.27, 1078-1084
DOI
ScienceOn
|
4 |
Park, W.H., Jung, C.W., Park, J.O., Kim, K., Kim, W.S., Im, Y.H., Lee, M.H., Kang, W.K., and Park, K. (2003). Trichostatin inhibits the growth of ACHN renal cell carcinoma cells via cell cycle arrest in association with p27, or apoptosis. Int. J. Oncol. 22, 1129-1134
|
5 |
Poot, M., Teubert, H., Rabinovitch, P.S., and Kavanagh, T.J. (1995). De novo synthesis of glutathione is required for both entry into and progression through the cell cycle. J. Cell. Physiol. 163, 555-560
DOI
ScienceOn
|
6 |
Tikhaze, A.K., Lankin, V.Z., Zharova, E.A., and Kolycheva, S.V. (2000). Trimetazidine as indirect antioxidant. Bull. Exp. Biol. Med. 130, 951-953
|
7 |
Wallach-Dayan, S.B., Izbicki, G., Cohen, P.Y., Gerstl-Golan, R., Fine, A., and Breuer, R. (2006). Bleomycin initiates apoptosis of lung epithelial cells by ROS but not by Fas/FasL pathway. Am. J. Physiol. Lung Cell Mol. Physiol. 290, L790-L796
DOI
ScienceOn
|
8 |
Woo, S.H., Park, I.C., Park, M.J., Lee, H.C., Lee, S.J., Chun, Y.J., Lee, S.H., Hong, S.I., and Rhee, C.H. (2002). Arsenic trioxide induces apoptosis through a reactive oxygen species-dependent pathway and loss of mitochondrial membrane potential in HeLa cells. Int. J. Oncol. 21, 57-63
|
9 |
Wu, X.X., Ogawa, O., and Kakehi, Y. (2004). Enhancement of arsenic trioxide-induced apoptosis in renal cell carcinoma cells by L-buthionine sulfoximine. Int. J. Oncol.24, 1489-1497
|
10 |
Zhang, W., Ohnishi, K., Shigeno, K., Fujisawa, S., Naito, K., Nakamura, S., Takeshita, K., Takeshita, A., and Ohno, R. (1998). The induction of apoptosis and cell cycle arrest by arsenic trioxide in lymphoid neoplasms. Leukemia 12, 1383-1391
DOI
|
11 |
Zorov, D.B., Juhaszova, M., and Sollott, S.J. (2006). Mitochondrial ROS-induced ROS release: An update and review. Biochem. Biophys. Acta. 1757, 509-517
DOI
ScienceOn
|
12 |
Han, Y.H., Kim, S.Z., Kim, S.H., and Park, W.H. (2007). Arsenic trioxide inhibits growth of As4.1 juxtaglomerular cells via cell cycle arrest and caspase-independent apoptosis. Am. J. Physiol. Renal. Physiol. 293, F511-520
DOI
ScienceOn
|
13 |
Bubici, C., Papa, S., Pham, C.G., Zazzeroni, F., and Franzoso, G. (2006). The NF-kappaB-mediated control of ROS and JNK signaling. Histol. Histopathol.21, 69-80
|
14 |
Hyun Park, W., Hee Cho, Y., Won Jung, C., Oh Park, J., Kim, K., Hyuck Im, Y., Lee, M.H., Ki Kang, W., and Park, K. (2003). Arsenic trioxide inhibits the growth of A498 renal cell carcinoma cells via cell cycle arrest or apoptosis. Biochem. Biophys. Res. Commun. 300, 230-235
DOI
ScienceOn
|
15 |
Khan, N.S., Ahmad, A., and Hadi, S.M. (2000). Anti-oxidant, prooxidant properties of tannic acid and its binding to DNA. Chem. Biol. Interact. 125, 177-189
DOI
ScienceOn
|
16 |
Shim, H.Y., Park, J.H., Paik, H.D., Nah, S.Y., Kim, D.S., and Han, Y.S. (2007). Acacetin-induced apoptosis of human breast cancer MCF-7 cells involves caspase cascade, mitochondriamediated death signaling and SAPK/JNK1/2-c-Jun activation. Mol. Cells 24, 95-104
|
17 |
Yamada, J., Yoshimura, S., Yamakawa, H., Sawada, M., Nakagawa, M., Hara, S., Kaku, Y., Iwama, T., Naganawa, T., Banno, Y. et alK (2003). Cell permeable ROS scavengers, Tiron and Tempol, rescue PC12 cell death caused by pyrogallol or hypoxia/ reoxygenation. Neurosci. Res.45, 1-8
DOI
ScienceOn
|
18 |
Han, Y.H., Kim, S.Z., Kim, S.H., and Park, W.H. (2008b). Intracellular GSH level is a factor in As4.1 juxtaglomerular cell death by arsenic trioxide. J. Cell. Biochem. 104, 995-1009
DOI
ScienceOn
|
19 |
Kito, M., Akao, Y., Ohishi, N., Yagi, K., and Nozawa, Y. (2002). Arsenic trioxide-induced apoptosis and its enhancement by buthionine sulfoximine in hepatocellular carcinoma cell lines. Biochem. Biophys. Res. Commun. 291, 861-867
DOI
ScienceOn
|
20 |
Dai, J., Weinberg, R.S., Waxman, S., and Jing, Y. (1999). Malignant cells can be sensitized to undergo growth inhibition and apoptosis by arsenic trioxide through modulation of the glutathione redox system. Blood 93, 268-277
|
21 |
Han, Y.H., Kim, S.H., Kim, S.Z., and Park, W.H. (2008a). Apoptosis in arsenic trioxide-treated Calu-6 lung cells is correlated with the depletion of GSH levels rather than the changes of ROS levels. J. Cell. Biochem. 104, 862-878
DOI
ScienceOn
|
22 |
Macho, A., Hirsch, T., Marzo, I., Marchetti, P., Dallaporta, B., Susin, S.A., Zamzami, N., and Kroemer, G. (1997). Glutathione depletion is an early and calcium elevation is a late event of thymocyte apoptosis. J. Immunol.158, 4612-4619
|
23 |
Scott, N., Hatlelid, K.M., MacKenzie, N.E., and Carter, D.E. (1993). Reactions of arsenic(III) and arsenic(V) species with glutathione. Chem. Res. Toxicol. 6, 102-106
DOI
ScienceOn
|
24 |
Zhang, T.C., Cao, E.H., Li, J.F., Ma, W., and Qin, J.F. (1999). Induction of apoptosis and inhibition of human gastric cancer MGC-803 cell growth by arsenic trioxide. Eur. J. Cancer 35, 1258-1263
DOI
ScienceOn
|
25 |
Baj, G., Arnulfo, A., Deaglio, S., Mallone, R., Vigone, A., De Cesaris, M.G., Surico, N., Malavasi, F., and Ferrero, E. (2002). Arsenic trioxide and breast cancer: analysis of the apoptotic, differentiative and immunomodulatory effects. Breast Cancer Res. Treat. 73, 61-73
DOI
ScienceOn
|
26 |
Dasmahapatra, G., Rahmani, M., Dent, P., and Grant, S. (2006). The tyrphostin adaphostin interacts synergistically with proteasome inhibitors to induce apoptosis in human leukemia cells through a reactive oxygen species (ROS)-dependent mechanism. Blood 107, 232-240
DOI
ScienceOn
|
27 |
Kim, H.R., Kim, E.J., Yang, S.H., Jeong, E.T., Park, C., Kim, S.J., Youn, M.J., So, H.S., and Park, R. (2006). Combination treatment with arsenic trioxide and sulindac augments their apoptotic potential in lung cancer cells through activation of caspase cascade and mitochondrial dysfunction. Int. J. Oncol. 28, 1401-1408
|
28 |
Soignet, S.L., Maslak, P., Wang, Z.G., Jhanwar, S., Calleja, E., Dardashti, L.J., Corso, D., DeBlasio, A., Gabrilove, J., Scheinberg, D.A.I et al. (1998). Complete remission after treatment of acute promyelocytic leukemia with arsenic trioxide. N. Engl. J. Med. 339, 1341-1348
DOI
ScienceOn
|
29 |
Li, M., Cai, J.F., and Chiu, J.F. (2002). Arsenic induces oxidative stress and activates stress gene expressions in cultured lung epithelial cells. J. Cell. Biochem. 87, 29-38
DOI
ScienceOn
|
30 |
Nakagawa, Y., Akao, Y., Morikawa, H., Hirata, I., Katsu, K., Naoe, T., Ohishi, N., and Yagi, K. (2002). Arsenic trioxide-induced apoptosis through oxidative stress in cells of colon cancer cell lines. Life Sci. 70, 2253-2269
DOI
ScienceOn
|
31 |
Simon, H.U., Haj-Yehia, A., and Levi-Schaffer, F. (2000). Role of reactive oxygen species (ROS) in apoptosis induction. Apoptosis 5, 415-418
DOI
ScienceOn
|
32 |
Uslu, R., Sanli, U.A., Sezgin, C., Karabulut, B., Terzioglu, E., Omay, S.B., and Goker, E. (2000). Arsenic trioxide-mediated cytotoxicity and apoptosis in prostate and ovarian carcinoma cell lines. Clin. Cancer Res.6, 4957-4964
|
33 |
Oketani, M., Kohara, K., Tuvdendorj, D., Ishitsuka, K., Komorizono, Y., Ishibashi, K., and Arima, T. (2002). Inhibition by arsenic trioxide of human hepatoma cell growth. Cancer Lett. 183, 147-153
DOI
ScienceOn
|
34 |
Baran, C.P., Zeigler, M.M., Tridandapani, S., and Marsh, C.B. (2004). The role of ROS and RNS in regulating life and death of blood monocytes. Curr. Pharm. Des.10, 855-866
DOI
ScienceOn
|
35 |
Chen, T.J., Jeng, J.Y., Lin, C.W., Wu, C.Y., and Chen, Y.C. (2006). Quercetin inhibition of ROS-dependent and -independent apoptosis in rat glioma C6 cells. Toxicology 223, 113-126
DOI
ScienceOn
|
36 |
Jing, Y., Dai, J., Chalmers-Redman, R.M., Tatton, W.G., and Waxman, S. (1999). Arsenic trioxide selectively induces acute promyelocytic leukemia cell apoptosis via a hydrogen peroxidedependent pathway. Blood 94, 2102-2111
|
37 |
Park, W.H., Seol, J.G., Kim, E.S., Hyun, J.M., Jung, C.W., Lee, C.C., Kim, B.K., and Lee, Y.Y. (2000). Arsenic trioxide-mediated growth inhibition in MC/CAR myeloma cells via cell cycle arrest in association with induction of cyclin-dependent kinase inhibitor, p21, and apoptosis. Cancer Res. 60, 3065-3071
|
38 |
Maeda, H., Hori, S., Ohizumi, H., Segawa, T., Kakehi, Y., Ogawa, O., and Kakizuka, A. (2004). Effective treatment of advanced solid tumors by the combination of arsenic trioxide and Lbuthionine- sulfoximine. Cell Death Differ.11, 737-746
DOI
ScienceOn
|
39 |
Lauterburg, B.H. (2002). Analgesics and glutathione. Am. J. Ther. 9, 225-233
DOI
ScienceOn
|
40 |
Wilcox, C.S. (2002). Reactive oxygen species: roles in blood pressure and kidney function. Curr. Hypertens Rep.4, 160-166
DOI
ScienceOn
|
41 |
Schnelldorfer, T., Gansauge, S., Gansauge, F., Schlosser, S., Beger, H.G., and Nussler, A.K. (2000). Glutathione depletion causes cell growth inhibition and enhanced apoptosis in pancreatic cancer cells. Cancer 89, 1440-1447
DOI
ScienceOn
|
42 |
Shen, Z.Y., Shen, W.Y., Chen, M.H., Shen, J., and Zeng, Y. (2003). Reactive oxygen species and antioxidants in apoptosis of esophageal cancer cells induced by As2O3. Int. J. Mol. Med.11, 479-484
|
43 |
Greenstock, C.L., and Miller, R.W. (1975). The oxidation of tiron by superoxide anion. Kinetics of the reaction in aqueous solution in chloroplasts. Biochim. Biophys. Acta. 396, 11-16
DOI
ScienceOn
|
44 |
Kitamura, K., Minami, Y., Yamamoto, K., Akao, Y., Kiyoi, H., Saito, H., and Naoe, T. (2000). Involvement of CD95-independent caspase 8 activation in arsenic trioxide-induced apoptosis. Leukemia 14, 1743-1750
DOI
|
45 |
Seol, J.G., Park, W.H., Kim, E.S., Jung, C.W., Hyun, J.M., Kim, B.K., and Lee, Y.Y. (1999). Effect of arsenic trioxide on cell cycle arrest in head and neck cancer cell line PCI-1. Biochem. Biophys. Res. Commun. 256, 400-404
|
46 |
Gonzalez, C., Sanz-Alfayate, G., Agapito, M.T., Gomez-Nino, A., Rocher, A., and Obeso, A. (2002). Significance of ROS in oxygen sensing in cell systems with sensitivity to physiological hypoxia. Respir. Physiol. Neurobiol. 132, 17-41
DOI
ScienceOn
|
47 |
Hedley, D.W., and Chow, S. (1994). Evaluation of methods for measuring cellular glutathione content using flow cytometry. Cytometry 15, 349-358
DOI
ScienceOn
|
48 |
Haga, N., Fujita, N., and Tsuruo, T. (2005). Involvement of mitochondrial aggregation in arsenic trioxide (As2O3)-induced apoptosis in human glioblastoma cells. Cancer science 96, 825-833
DOI
ScienceOn
|
49 |
Wang, Z.G., Rivi, R., Delva, L., Konig, A., Scheinberg, D.A., Gambacorti- Passerini, C., Gabrilove, J.L., Warrell, R.P., Jr., and Pandolfi, P.P. (1998). Arsenic trioxide and melarsoprol induce programmed cell death in myeloid leukemia cell lines and function in a PML and PML-RARalpha independent manner. Blood 92, 1497-1504
|
50 |
Miller, W.H., Jr., Schipper, H.M., Lee, J.S., Singer, J., and Waxman, S. (2002). Mechanisms of action of arsenic trioxide. Cancer Res. 62, 3893-3903
|
51 |
Stanley, W.C., and Marzilli, M. (2003). Metabolic therapy in the treatment of ischaemic heart disease: the pharmacology of trimetazidine. Fundam. Clin. Pharmacol. 17, 133-145
DOI
ScienceOn
|
52 |
Pu, Y.S., Hour, T.C., Chen, J., Huang, C.Y., Guan, J.Y., and Lu, S.H. (2002). Cytotoxicity of arsenic trioxide to transitional carcinoma cells. Urology 60, 346-350
DOI
ScienceOn
|
53 |
Kang, Y.H., Yi, M.J., Kim, M.J., Park, M.T., Bae, S., Kang, C.M., Cho, C.K., Park, I.C., Park, M.J., Rhee, C.H.I et alK (2004). Caspase- independent cell death by arsenic trioxide in human cervical cancer cells: reactive oxygen species-mediated poly(ADPribose polymerase-1 activation signals apoptosis-inducing factor release from mitochondria. Cancer Res. 64, 8960-8967
DOI
ScienceOn
|