Induction of Apoptosis by N-methyl-N'-nitro-N-nitrosoguanidine, an Alkylating Agent, in Human Prostate Carcinoma Cells |
Park, Cheol
(Department of Biochemistry, Dong-Eui University College of Oriental Medicine, and Research Institute of Oriental Medicine, Department of Biology, Busan National University)
Choi, Byung-Tae (Department of Anatomy, Dong-Eui University College of Oriental Medicine and Research Institute of Oriental Medicine) Lee, Won-Ho (Department of Biology, Busan National University) Choi, Yung-Hyun (Department of Biochemistry, Dong-Eui University College of Oriental Medicine, and Research Institute of Oriental Medicine) |
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Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c
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DOI ScienceOn |
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Oncogenic transformation of HPV-immortalized human oral keratinocytes is associated with the genetic instability of cells
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Apopotosis by death factor
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DOI ScienceOn |
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Cadherins: crucial regulators of structure and function in reproductive tissues
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DOI ScienceOn |
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Activation of a p53-independent, sphingolipid-mediated cytolytic pathway in p53-negative mouse fibroblast cells treated with Nmethyl-N-nitro-N-nitrosoguanidine
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DOI ScienceOn |
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hMutSalpha- and hMutLalpha-dependent phosphorylation of p53 in response to DNA methylator damage
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Yama/CPP32 β, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase
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Ecadherin-catenin cell-cell adhesion complex and human cancer
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Evidence for a connection between the mismatch repair system and the G2 cell cycle checkpoint
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Necrosis and apoptosis in acute renal failure
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Apoptosis-induced cleavage of β-catenin by caspase-3 results in proteolytic fragments with reduced transactivation potential
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ATM is activated in response to N-methyl-N'-nitro-N-nitrosoguanidine-induced DNA alkylation
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An alkylation-tolerant, mutator human cell line is deficient in strand-specific mismatch repair
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Survival and proliferation of cells expressing caspase-uncleavable Poly(ADP-ribose) polymerase in response to death-inducing DNA damage by an alkylating agent
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Apoptosis signaling by death receptors
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XIAP: apoptotic brake and promising therapeutic target
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Research technics for the cell cycle study
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Cleavage of poly(ADP-ribose) polymerase by a proteinase with properties like ICE
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Regulation of cyclin D1 by calpain protease
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Bax directly induces release of cytochrome c from isolated mitochondria
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Diverse chemical carcinogens fail to induce G(1) arrest in MCF-7 cells
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Intragenomic heterogeneity of DNA damage formation and repair: a review of cellular responses to covalent drug DNA interaction
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p53 Phosphorylation at serine 15 is required for transcriptional induction of the plasminogen activator inhibitor-1 (PAI-1) gene by the alkylating agent N-methyl-N'-nitro-N-nitrosoguanidine
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DOI ScienceOn |
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The machinery of programmed cell death
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Apoptosis and melanoma: molecular mechanisms
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Apoptosis-associated cleavate of β-catenin in human colon cancer and rat hepatoma cells
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DOI ScienceOn |
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Cell cycle was disturbed in the MNNG-induced initiation stage during in vitro two-stage transformation of Balb/3T3 cells
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DOI |
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Apoptotic activity of novel bile acid derivatives in human leukemic T cells through the activation of caspases
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Current chemotherapy and future directions in research for the treatment of advanced hormone-refractory prostate cancer
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DNA damage can induce apoptosis in proliferating lymphoid cells via p53-independent mechanisms inhibitable by Bcl-2
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Role of DNA mismatch repair and p53 in signaling induction of apoptosis by alkylating agents
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SN2 DNA-alkylating agent-induced phosphorylation of p53 and activation of p21 gene expression
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The human cyclin B1 protein modulates sensitivity of DNA mismatch repair deficient prostate cancer cell lines to alkylating agents
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Mechanisms of action of, and modes of resistance to, alkylating agents used in the treatment of haematological malignancies
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Necrosis and apoptosis: distinct modes of cell death with fundamentally different significance
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IAP proteins; blocking the road to death's door
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Cell death: the significance of apoptosis
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Competency in mismatch repair prohibits clonal expansion of cancer cells treated with N-methyl-N'-nitro-N-nitrosoguanidine
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Widely used prostate carcinoma cell lines share common origins
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Specific proteolytic cleavage of poly(ADP-ribose) polymerase: an early marker of chemotherapy-induced apoptosis
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Mitochondria, oxidative stress, and antioxidant defences
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DNA damage-induced cell cycle checkpoints involve both p53-dependent and -independent pathways: role of telomere repeat binding factor 2
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The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis
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p53-independent induction of p21 (WAF1/CIP1), reduction of cyclin B1 and G2/M arrest by the isoflavone genistein in human prostate carcinoma cells
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Cell adhesion molecules in the development and progression of malignant melanoma
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Bcl-2 family proteins
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The Bcl-2 protein family
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Apoptosis and the cell cycle
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