Anti-Cancer Effects and Apoptosis by Korean Medicinal Herbs |
Ko Seong Gyu
(Department of Internal Medicine, Sangji University Hospital of Oriental Medicine, Department of Tumor Biology, Cancer Research Institute College of Medicine, Seoul National University)
Jun Chan Yong (Department of Internal Medicine, Kyungwon University Hospital of Oriental Medicine) Park Chong Hyeong (Department of Internal Medicine, Kyungwon University Hospital of Oriental Medicine) Bae Hyun Su (Department of Pathology, Kyunghee University School of Medicine) |
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Specific repression of TATA-mediated but not initiator-mediated transcription by wild-type p53.
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Growth suppression induced by wild-type p53 protein is accompanied by selective down-regulation of proliferating-cell nuclear antigen expression.
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Identification of a p53-dependent negative response element in the Bcl-2 gene.
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Flt3 ligand stimulates proliferation and inhibits apoptosis of acute myeloid leukemia cells: Regulation of Bcl-2 and Bax.
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Programmed cell death: concept, mechanism and control.
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Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-2
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Wild-type p53 binds to the TATA-binding protein and represses Transcription.
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BAX enhances paclitaxel-induced apoptosis through a p53-independent pathway.
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Upregulated Expression of Bcl-2 in multiple myeloma cells induced by exposure to Doxorubicin, Etoposide, and Hydrogen peroxide.
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control of fibroblast senescence and activation of programmed cell death.
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The MCK enhancer contains a p53 responsive element.
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Molecular regulation of apoptosis: genetic controls on cell death.
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Apoptosis.
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Plantagoside, a novel alpha-mannosidase inhibitor isolated from the seeds of Plantago asiatica, suppresses immune response.
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Bax suppresses tumorigenesis and stimulates apoptosis in vivo.
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Wild-type p53 mediates positive regulation of gene expression through a specific DNA sequence element.
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The p53 tumor suppressor gene and product.
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Negative growth regulation in a glioblastoma tumor cell line that conditionally expresses human wild-type p53.
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Modulation of activity of the promoter of the human MDR1 gene by ras and p53.
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The control of apoptosis in mammalian cells.
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Waf1/CIP1 is induced in p53-mediated G1 arrest and apoptosis.
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Expression and regulation of Bcl-2, Bcl-xL, Bax correlate with p53 status and sensitivity to apoptosis in childhood acute lymphoblastic leukemia.
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Cdk-interacting protein 1 directly binds with proliferating cell nuclear antigen and inhibits DNA replication catalyzed by the DNA polymerase holoenzyme.
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Wild-type p53 can down-modulate the activity of various promoters.
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The p21 cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases.
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Effect of bcl-2 on Fas antigen-mediated cell death.
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Genetic basis of multidrug resistance of tumor cells.
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A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasia.
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Oncogenic forms of p53 inhibit p53-regulated gene expression.
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Study on trends of cancer study in TKM and its research strategy in future.
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p53, guardian of the genome.
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Wild-type p53 induces apoptosis of myeloid leukemic cells that is inhibited by interleukin-6.
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Mdm-2 expression is induced by wild-type p53 activity.
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Induction of Sp1-p53 binding heterocomplexes during granulocyte macrophage colony-stimulating factor-dependent proliferation in human erythroleukemia cell line TF-1.
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Antitumor activity of Taraxacum mongolicum.
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