Protective Effect of Puerariae radix Against Ethanol-induced Apoptosis on Human Neuroblastoma Cell Line SK-N-MC |
Koo Gyo Sung
(Oriental Medicine Research Institute, Kyungwon University)
Cho Son Hae (Oriental Medicine Research Institute, Kyungwon University) Jang Mi Hyean (Department of Physiology. College of Medicine, Kyunghee University) Kim Chang Ju (Department of Physiology. College of Medicine, Kyunghee University) Kim Ee Hwa (Department of Meridinology, College of Oriental. Medicine, Semyung University) Lee Choong Yeol (Oriental Medicine Research Institute, Kyungwon University) |
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Ethanol increases apoptotic cell death of thymocytes in vitro
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Staurosporine-induced apoptosis of cultured rat hippocampal neurons involves caspase-1-like proteases as upstream initiators and increased production of superoxide as a main downstream effector
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Caspases: the executioners of apoptosis
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Protective effects of Puerariae flos against ethanol-induced apoptosis on human neuroblastoma cell line SK-N-MC
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Indomethacin induces poptosis and inhibits proliferation in chronic myeloid leukemia cells
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Expression and activation of caspase-3/CPP32 in CD34+ cord blood cells is linked to apotosis after growth factor withdrawal
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Potentiation of the bioavailability of daidzin by an extract of Radix puerariae
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Daidzin: A potent, selective inhibitor of human mitochondrial aldehyde dehydrogenase
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Analysis of Puerariae radixand its medicinal preparations by capillary electrophoresis
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Daidzin and its antidipsotropic analogs inhibit serotonin anddopamine metabolism in isolated mitochondria
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Daidzin inhibits mitochondrial aldehyde dehydrogenase and suppresses ethanol intake of Syrian golden hamsters
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Differential neuronal loss following early postnatal alcohol exposure
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Ethanol-induced apoptotic neurodegeneration and fetal alcohol syndrome
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Ethanol-induced cell death in cultured rat astroglia
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Glial-derived neurotrophic factor (GDNF) prevents ethanol-induced apoptosis and JUN kinase phosphorylation
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Chronic ethanol feeding increases apoptosis and cell proliferation in rat liver
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Bcl-2gene family and the regulation of programmed cell death
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Activation of CPP32 during apoptosis of neurons and astrocytes
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Aberrant expression of bcl-2 gene family in Down's syndrome brains
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Bcl-2 heterodimerizes in vivo with a conserved homolog, bax, that accelerates programmed cell death
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The origins of DNA breaks: a consequence of DNA damage, DNA repair or apoptosis?
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Enhanced caspase activity during ethanol-induced apoptosis in rat cerebellar granule cells
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Bax-dependent caspase-3 activation is a key determinant in p53-induced apoptosis in neurons
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Double identity for proteins of the bcl-2 family
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Prevention of programmed cell death of sympathetic neurons by the bcl-2 proto-oncogene
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Bcl-2 overexpression protects the neonatal cerebellum from ethanol neurotoxicity
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Fetal alcohol syndrome: the vulnerability of the developing brain and possible mechanisms of damage
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Bax deletion further orders the cell death pathway in cerebellar granule cells and suggests a caspase-independent pathway to cell death
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Essential contribution of caspase-3/CPP32 to apoptosis and its associated nuclear changes
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Modulation of caspase-3 activity and Fas ligand mRNA expression in rat liver cells in vivo by alcohol and lipopolysccharide
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