1 |
Chun MJ, Korbet SM, Schwartz MM, Leweis EJ. Focal segmental glomerulosclerosis in nephrotic adults : presentation, prognosis, and response to therapy of the histologic variants. J Am Soc Nephrol 2004;15:2169-77
DOI
ScienceOn
|
2 |
Vincenti F, Ghiggerri GM. New insights into the pathogenesis and the therapy of recurrent focal glomerulosclerosis. Am J Transplant 2005;5:1179-85
DOI
ScienceOn
|
3 |
Ruf RG, Lichtenberger A, Karle Sm, Haas JP, Anacleto FE, Schultheiss M, et al. Patients with mutations in NPHS2(podocin) do not respond to standard steroid treatment of nephrotic syndrome. J Am Sco Nephrol 2004;15:722-32
DOI
ScienceOn
|
4 |
White RH. The familial nephrotic syndrome. 1. A European survey. Clin Nephrol 1973;1:215-9
|
5 |
Reise J, Gersdorff GV, Loos M, Oh M, Asanuma K, Giardino L, et al. Induction of B7-1 in podocytes is associated with nephrotic syndrome. J Clin Invest 2004;113:1390-7
|
6 |
Ruf RG, Fuchshuber A, Karle SM, Lemainque A, Huck K, Wienker T, et al. Identification of the first gene locus(SSNS1) for steroid sensitive nephrotic syndrome on chromosome 2p. J Am So Nephrol 2003;14:1897-900
DOI
ScienceOn
|
7 |
Schmid H, Henger A, Cohen CD, Frach K, Grone HJ, Schlondorff D, et al. Gene expression profiles of podocyte-associated molecules as diagnostic markers in acquired proteinuric disease. J Am Soc Nephrol 2003;14:2958-66.
DOI
ScienceOn
|
8 |
Caridi G, Bertelli R, Di Duca M, Dagnino M, Emma F, Onetti Muda A, et al. Broadening the spectrum of disease related to podocin mutations. J Am Soc Nephrol 2003;14:1278-86
DOI
ScienceOn
|
9 |
Boute N, Gribouval O, Roselli S, Benessy F, Lee H, Fuchshuber A, et al. NPHS2, encoding the glomerular protein podocin is mutated in autosomal recessive steroid-resistant nephrotic syndrome. Nat Genet 2000;24:349-354
DOI
ScienceOn
|
10 |
Ruth EM. Children with steroid-sensitive nephrotic syndrome come of age: long-term outcome. J Pediatr 2005;147:202-7
DOI
ScienceOn
|
11 |
Lewis MA, Baildom EM, Davis N, Houston IB, Postlethwaite RJ. Nephrotic syndrome : from toddlers to twenties. Lancet 1989;8632:255-9
|
12 |
Punnonen J, Aversa G, Cocks BG, McKenzie ANJ, Menon S, Zurawski G, et al. Interleukin-13 induces interleukin-4 independent IgG4 and IgE synthesis and CD23 expression by human B cells. Proc Natl Acad Sci USA 1993;90:3730-4
DOI
ScienceOn
|
13 |
Levy F, Kristofic C, Heusser C, Brinkmann V. Role of IL-13 in CD4 T cell-dependent IgE production in atopy. Int Arch Allergy Immunol 1997:112:49-58
DOI
ScienceOn
|
14 |
Trompeter RS, Lloyd BW, Hicks J, White RH, Cameron JS. Longterm outcome for children with minimal change nephrotic syndrome. Lancet 1985;8425:368-70
|
15 |
Koskimies O, Vilska J, Rapola J, Hallman N. Longterm outcome of primary nephrotic syndrome. Arch Dis Child 1982;7;544-8
|
16 |
Cheung W, Wei CL, Seah CC, Yap HK. Atopy, serum IgE, and interleukin-13 in steroid-responsive nephrotic syndrome. Pediatr Nephrol 2004;19:627-32
DOI
ScienceOn
|
17 |
Glassock RJ. Circulating permeability factors in the nephrotic syndrome : a fresh look at an old problem. J Am Soc Nephrol 2003;14:541-3
DOI
ScienceOn
|
18 |
Pereira AC, Pereira AB, Mota GF, Cunha RS, Herkenhoff FL, Pollak MR, et al. NPHS2 R229Q functional variant is associated with microalbuminuria in the general population. Kidney Int 2004;65:1026-30.
DOI
ScienceOn
|
19 |
Bacharier LB, Jabara H, Geha RS. Molecular mechanism of immunoglobulin E regulation. Int Arch Allergy Immunol 1998;115:257-69
DOI
ScienceOn
|
20 |
Lahdenkari AT, Suvanto M, Kaskimies O, Kestila M, Jalanko H. Clinical features and outcome of childhood minimal change nephrotic syndrome : is genetics involved? Pediatr Nephrol 2005;20;1073-80
DOI
ScienceOn
|
21 |
Bassi C, Petrini C, Rizza V, Napodano P, Pararella M, Arrigo G, et al. Fractional excretion of IgG predicts renal outcome and response to therapy in primary focal segmental glomerulosclerosis : a pilot study. Am J Kidney Dis 2003;41:328-35
DOI
ScienceOn
|
22 |
Kobayashi Y, Arakawa H, Suzuki M, Takizawa T, Tokuyama K, Morikawa A. Polymorphism of IL-4 related genes in Japanese children with minimal change nephrotic syndrome. Am J Kidney Dis 2003;42:271-6
DOI
ScienceOn
|
23 |
Kaplan JM, Kim SH, North KN, Rennke H, Correia LA, Tong HQ, et al. Mutations in ACTN4, encoding alphaactinin-4, cause familial focal segmental glomerulosclerosis. Nat Genet 2000;24:251-6
DOI
ScienceOn
|
24 |
Ehrich JH, Brohdehl J. Long versus standard prednisone therapy for initial treatment of idopathic nephrotic syndrome in children. Arbeitsgemeinschaft fur Padiatrische Nephrologie. Eur J Pediatr 1993;152:357-61
DOI
ScienceOn
|
25 |
Akhtar M, Al Mana h. Molecular basis of proteinuria. Adv Anat Pathol 2004;11:304-9
DOI
ScienceOn
|
26 |
Kestila M, Lenkkeri U, Mannikko M, Lamerdin J, Mc-Cready P, Putaala H, et al. Positionally cloned gene for a novel glomerular protein-nephritis mutated in congenital nephrotic syndrome. Mol Cell 1998;4:575-82
|
27 |
Fakhouri F, Bocquet N, Taupin P, Presne C, Gagnadous MF, Landais P, et al. Steroid sensitive nephrotic syndrome : from childhood to adulthood. Am J Kidney Dis 2003;3:550-7
|
28 |
Winn MP, Conlon PJ, Lynn KL, Howell DN, Slotterbeck BD, Smith AH, et al. Linkage of a gene causing familial focal glomerulosclerosis to chromosome 11 and further evidence of genetic heterogeneity. Genomics 1999;58:113-20
DOI
ScienceOn
|
29 |
Bakr A, Shokeir M, El-Chenawi F, El-Husseni F, Abdel-Rahman A, El-Ashry R. Tumor necrosis factor-alpha production from mononuclear cells in nephrotic syndrome. Pediatr Nephrol 2003;18:516-20
|
30 |
Carlotti AP, Franco PB, Elias LL, Facincani I, Costa EL, Foss N, et al. Glucocorticoid receptors, in vitro steroid sensitivity, and cytokine secretion in idiopathic nephrotic syndrome. Kidney Int 2004;65:403-8
DOI
ScienceOn
|