1 |
Webster C, Silberstein L, Hays AP, et al. Fast muscle fibers are preferentially affected in Duchenne muscular dystrophy. Cell. 1988;52(4):503-13.
DOI
|
2 |
Widegren U, Ryder JW, Zierath JR. Mitogen-activated protein kinase signal transduction in skeletal muscle: effects of exercise and muscle contraction. Acta Physiol Scand. 2001;172(3):227-38.
DOI
|
3 |
Wilson JM, Marin PJ, Rhea MR, et al. Concurrent training: a meta-analysis examining interference of aerobic and resistance exercises. J Strength Cond Res. 2012;26(8):2293-307.
DOI
|
4 |
Reidy PT, Konopka AR, Hinkley JM, et al. The effect of feeding during recovery from aerobic exercise on skeletal muscle intracellular signaling. Int J Sport Nutr Exerc Metab. 2014;24(1):70-8.
DOI
|
5 |
Ahmad S, Bhatia K, Kannan A, et al. Molecular mechanisms of neurodegeneration in spinal muscular atrophy. J Exp Neurosci. 2016;10:39-49.
|
6 |
Baar K. Training for endurance and strength: lessons from cell signaling. Med Sci Sports Exerc. 2006;38(11): 1939-44.
DOI
|
7 |
Bassel-Duby R, Olson EN. Signaling pathways in skeletal muscle remodeling. Annu Rev Biochem. 2006;75: 19-37.
DOI
|
8 |
Beyfuss K, Hood DA. A systematic review of p53 regulation of oxidative stress in skeletal muscle. Redox Rep. 2018;23(1):100-17.
DOI
|
9 |
Biondi O, Grondard C, Lecolle S, et al. Exercise-induced activation of NMDA receptor promotes motor unit development and survival in a type 2 spinal muscular atrophy model mouse. J Neurosci. 2008;28(4):953-62.
DOI
|
10 |
Bujak AL, Crane JD, Lally JS, et al. AMPK activation of muscle autophagy prevents fasting-induced hypoglycemia and myopathy during aging. Cell Metab. 2015;21(6):883-90.
DOI
|
11 |
Christie-Brown V, Mitchell J, Talbot K. The SMA Trust: the role of a disease-focused research charity in developing treatments for SMA. Gene Ther. 2017; 24(9):544-6.
DOI
|
12 |
Bushby K, Finkel R, Birnkrant DJ, et al. Diagnosis and management of Duchenne muscular dystrophy, part 1: diagnosis, and pharmacological and psychosocial management. Lancet Neurol. 2010;9(1):77-93.
DOI
|
13 |
Camera DM, Smiles WJ, Hawley JA. Exercise-induced skeletal muscle signaling pathways and human athletic performance. Free Radic Biol Med. 2016;98:131-43.
DOI
|
14 |
Cao S, Li B, Yi X, et al. Effects of exercise on AMPK signaling and downstream components to PI3K in rat with type 2 diabetes. PLoS One. 2012;7(12):e51709.
DOI
|
15 |
Chau A, Kalsotra A. Developmental insights into the pathology of and therapeutic strategies for DM1: Back to the basics. Dev Dyn. 2015;244(3):377-90.
DOI
|
16 |
Cho DH, Tapscott SJ. Myotonic dystrophy: emerging mechanisms for DM1 and DM2. Biochim Biophys Acta. 2007;1772(2):195-204.
DOI
|
17 |
Chtara M, Chaouachi A, Levin GT, et al. Effect of concurrent endurance and circuit resistance training sequence on muscular strength and power development. J Strength Cond Res. 2008;22(4):1037-45.
DOI
|
18 |
Coffey VG, Hawley JA. The molecular bases of training adaptation. Sports Med. 2007;37(9):737-63.
DOI
|
19 |
Coffey VG,Hawley JA. Concurrent exercise training: do opposites distract? J Physiol. 2017;595(9):2883-96.
DOI
|
20 |
Deshmukh AS, Hawley JA, Zierath JR. Exercise-induced phospho-proteins in skeletal muscle. Int J Obes (Lond). 2008;32(Suppl 4):S18-23.
DOI
|
21 |
Dial AG, Ng SY, Manta A, et al. The Role of AMPK in Neuromuscular Biology and Disease. Trends Endocrinol Metab. 2018;29(5):300-12.
DOI
|
22 |
Gibala M. Molecular responses to high-intensity interval exercise. Appl Physiol Nutr Metab. 2009;34(3): 428-32.
DOI
|
23 |
Dudley GA, Djamil R. Incompatibility of endurance- and strength-training modes of exercise. J Appl Physiol (1985). 1985;59(5):1446-51.
DOI
|
24 |
d'Ydewalle C, Sumner CJ. Spinal Muscular Atrophy Therapeutics: Where do we Stand? Neurotherapeutics. 2015;12(2):303-16.
DOI
|
25 |
Fry AC. The role of resistance exercise intensity on muscle fibre adaptations. Sports Med. 2004;34(10):663-79.
DOI
|
26 |
Gomes AS, Ramos H, Soares J, et al. p53 and glucose metabolism: an orchestra to be directed in cancer therapy. Pharmacol Res. 2018;131:75-86.
DOI
|
27 |
Goulet BB, Kothary R, Parks RJ. At the “junction” of spinal muscular atrophy pathogenesis: the role of neuromuscular junction dysfunction in SMA disease progression. Curr Mol Med. 2013;13(7):1160-74.
DOI
|
28 |
Gowans GJ, Hawley SA, Ross FA, et al. AMP is a true physiological regulator of AMP-activated protein kinase by both allosteric activation and enhancing net phosphorylation. Cell Metab. 2013;18(4):556-66.
DOI
|
29 |
Hamilton G, Gillingwater TH. Spinal muscular atrophy: going beyond the motor neuron. Trends Mol Med. 2013;19(1):40-50.
DOI
|
30 |
Hardie DG. AMP-activated protein kinase: a cellular energy sensor with a key role in metabolic disorders and in cancer. Biochem Soc Trans. 2011a;39(1):1-13.
DOI
|
31 |
Hardie DG. Sensing of energy and nutrients by AMP-activated protein kinase. Am J Clin Nutr. 2011b;93(4):891S-6.
DOI
|
32 |
Scoto M, Finkel RS, Mercuri E, et al. Therapeutic approaches for spinal muscular atrophy (SMA). Gene Ther. 2017;24(9):514-9.
DOI
|
33 |
Hawley JA, Hargreaves M, Joyner MJ, et al. Integrative biology of exercise. Cell. 2014;159(4):738-49.
DOI
|
34 |
Sakamoto K, Goodyear LJ. Invited review: intracellular signaling in contracting skeletal muscle. J Appl Physiol (1985). 2002;93(1):369-83.
DOI
|
35 |
Sandri M, Coletto L, Grumati P, et al. Misregulation of autophagy and protein degradation systems in myopathies and muscular dystrophies. J Cell Sci. 2013;126(23): 5325-33.
DOI
|
36 |
Stein SC, Woods A, Jones NA, et al. The regulation of AMP-activated protein kinase by phosphorylation. Biochem J. 2000;345(3):437-43.
DOI
|
37 |
Tesch PA. Skeletal muscle adaptations consequent to long-term heavy resistance exercise. Med Sci Sports Exerc. 1988;20(5 Suppl):132-4.
DOI
|
38 |
Vainshtein A, Hood DA. The regulation of autophagy during exercise in skeletal muscle. J Appl Physiol (1985). 2016;120(6):664-73.
DOI
|
39 |
Vander Heiden MG, Cantley LC, Thompson CB. Understanding the Warburg effect: the metabolic requirements of cell proliferation. Science. 2009;324(5930):1029-33.
DOI
|
40 |
Vousden KH, Ryan KM. p53 and metabolism. Nat Rev Cancer. 2009;9(10):691-700.
DOI
|
41 |
Wang L, Mascher H, Psilander N, et al. Resistance exercise enhances the molecular signaling of mitochondrial biogenesis induced by endurance exercise in human skeletal muscle. J Appl Physiol (1985). 2011;111(5): 1335-44.
DOI
|
42 |
Richter EA, Ruderman NB. AMPK and the biochemistry of exercise: implications for human health and disease. Biochem J. 2009;418(2):261-75.
DOI
|
43 |
Hawley JA. Molecular responses to strength and endurance training: are they incompatible? Appl Physiol Nutr Metab. 2009;34(3):355-61.
DOI
|
44 |
Warburg O. Notiz uber den Stoffwechsel der Tumoren. Biochem. Zeitschr. 1930;228 (1/3):257-8.
|
45 |
Warburg O. On the origin of cancer cells. Science. 1956;123(3191):309-14.
DOI
|
46 |
Hawley JA, Hargreaves M, Zierath JR. Signalling mechanisms in skeletal muscle: role in substrate selection and muscle adaptation. Essays Biochem. 2006;42:1-12.
DOI
|
47 |
Hickson RC. Interference of strength development by simultaneously training for strength and endurance. Eur J Appl Physiol Occup Physiol. 1980;45(2-3): 255-63.
DOI
|
48 |
Holloszy JO, Coyle EF. Adaptations of skeletal muscle to endurance exercise and their metabolic consequences. J Appl Physiol Respir Environ Exerc Physiol. 1984;56(4):831-8.
|
49 |
Holloszy JO. Biochemical adaptations in muscle. Effects of exercise on mitochondrial oxygen uptake and respiratory enzyme activity in skeletal muscle. J Biol Chem. 1967;242(9):2278-82.
|
50 |
Jensen TE, Rose AJ, Jorgensen SB, et al. Possible CaMKK- dependent regulation of AMPK phosphorylation and glucose uptake at the onset of mild tetanic skeletal muscle contraction. Am J Physiol Endocrinol Metab. 2007;292(5):1308-17.
DOI
|
51 |
Jensen TE, Wojtaszewski JF, Richter EA. AMP-activated protein kinase in contraction regulation of skeletal muscle metabolism: necessary and/or sufficient? Acta Physiol (Oxf). 2009;196(1):155-74.
DOI
|
52 |
Jones TW, Howatson G, Russell M, et al. Performance and neuromuscular adaptations following differing ratios of concurrent strength and endurance training. J Strength Cond Res. 2013;27(12):3342-51.
DOI
|
53 |
Morales-Alamo D, Calbet JAL. AMPK signaling in skeletal muscle during exercise: Role of reactive oxygen and nitrogen species. Free Radic Biol Med. 2016;98:68-77.
DOI
|
54 |
Kjobsted R, Hingst JR, Fentz J, et al. AMPK in skeletal muscle function and metabolism. FASEB J. 2018;32(4): 1741-77.
DOI
|
55 |
Kwon I, Jang Y, Cho JY, et al. Long-term resistance exercise-induced muscular hypertrophy is associated with autophagy modulation in rats. J Physiol Sci. 2018;68(3):269-80.
DOI
|
56 |
Lefebvre S, Burglen L, Reboullet S, et al. Identification and characterization of a spinal muscular atrophy- determining gene. Cell. 1995;80(1):155-65.
DOI
|
57 |
Lim KR, Maruyama R, Yokota T. Eteplirsen in the treatment of Duchenne muscular dystrophy. Drug Des Devel Ther. 2017;11:533-45.
DOI
|
58 |
McDonald CM, Campbell C, Torricelli RE, et al. Ataluren in patients with nonsense mutation Duchenne muscular dystrophy (ACT DMD): a multicentre, randomised, double-blind, placebo-controlled, phase 3 trial. Lancet. 2017;390(10101):1489-98.
DOI
|
59 |
Mounier R, Theret M, Lantier L, et al. Expanding roles for AMPK in skeletal muscle plasticity. Trends Endocrinol Metab. 2015;26(6):275-86.
DOI
|
60 |
Murach KA, Bagley JR. Skeletal Muscle Hypertrophy with Concurrent Exercise Training: Contrary Evidence for an Interference Effect. Sports Med. 2016;46(8): 1029-39.
DOI
|
61 |
Murlasits Z, Kneffel Z, Thalib L. The physiological effects of concurrent strength and endurance training sequence: A systematic review and meta-analysis. J Sports Sci. 2018;36(11):1212-9.
DOI
|
62 |
O'Neill HM. AMPK and Exercise: Glucose Uptake and Insulin Sensitivity. Diabetes Metab J. 2013;37(1):1-21.
DOI
|
63 |
Preston RR, Wilson TE. Physiology. Wolters Kluwer Health/Lippincott Williams & Wilkins. 2013.
|
64 |
Perez-Schindler J, Hamilton DL, Moore DR, et al. Nutritional strategies to support concurrent training. Eur J Sport Sci. 2015;15(1):41-52.
DOI
|
65 |
Pilgram GS, Potikanond S, Baines RA, et al. The roles of the dystrophin-associated glycoprotein complex at the synapse. Mol Neurobiol. 2010;41(1):1-21.
DOI
|