References
- Agudo A, Bonet C, Sala N, et al (2013). Hemochromatosis (HFE)gene mutations and risk of gastric cancer in the European Prospective Investigation into Cancer and Nutrition (EPIC) study. Carcinogenesis, 34, 1244-50. https://doi.org/10.1093/carcin/bgt045
- Al-Moundhri MS, Al-Kindi M, Al-Nabhani M, et al (2007).NAT2 polymorphism in Omani gastric cancer patients-risk predisposition and clinicopathological associations. World J Gastroenterol, 13, 2697-702. https://doi.org/10.3748/wjg.v13.i19.2697
- Balaji L, Krishna BS, Bhaskar LV (2012). An unlikely role for the NAT2 genotypes and haplotypes in the oral cancer of south Indians. Arch Oral Biol, 57, 513-8. https://doi.org/10.1016/j.archoralbio.2011.10.019
- Begg CB, Mazumdar M (1994). Operating characteristics of a rank correlation test for publication bias. Biometrics, 50,1088-101. https://doi.org/10.2307/2533446
- Blum M, Grant DM, McBride W, et al (1990). Human arylamineN-acetyltransferase genes: isolation, chromosomallocalization and functional expression. DNA Cell Biol, 9,193-203. https://doi.org/10.1089/dna.1990.9.193
- Borlak J, Reamon-Buettner SM (2006). N-acetyltransferase2 (NAT2) gene polymorphisms in colon and lung cancer patients. BMC Med Genet, 7, 58. https://doi.org/10.1186/1471-2350-7-58
- Cleary SP, Cotterchio M, Shi E, et al (2010). Cigarette smoking, genetic variants in carcinogen-metabolizing enzymes and colorectal cancer risk. Am J Epidemiol, 172, 1000-14. https://doi.org/10.1093/aje/kwq245
- Colditz GA, Burdick E, Mosteller F (1995). Heterogeneity in meta-analysis of data from epidemiologic studies: acommentary. Am J Epidemiol, 142, 371-82.
- Cotterchio M, Boucher BA, Manno M, et al (2008). Red meat intake, doneness, polymorphisms in genes that encodecarcinogen-metabolizing enzymes and colorectal cancerrisk. Cancer Epidemiol Biomarkers Prev, 17, 3098-107. https://doi.org/10.1158/1055-9965.EPI-08-0341
- De Stefani E, Boffetta P, Mendilaharsu M, et al (1998). Dietary nitrosamines, heterocyclic amines, and risk of gastric cancer:a case-control study in Uruguay. Nutr Cancer, 30, 158-62. https://doi.org/10.1080/01635589809514656
- Delort L, Satih S, Kwiatkowski F, et al (2010). Evaluation of breast cancer risk in a multigenic model including lowpenetrance genes involved in xenobiotic and estrogenmetabolisms. Nutr Cancer, 62, 243-51. https://doi.org/10.1080/01635580903305300
- Demokan S, Suoglu Y, Gozeler M, et al (2010). N-acetyltransferase1 and 2 gene sequence variants and risk of head and neck cancer. Mol Biol Rep, 37, 3217-26. https://doi.org/10.1007/s11033-009-9905-8
- DerSimonian R, Laird N (1986). Meta-analysis in clinical trials. Control Clin Trials, 7, 177-88. https://doi.org/10.1016/0197-2456(86)90046-2
- Egger M, Davey Smith G, Schneider M, et al (1997). Bias inmeta-analysis detected by a simple, graphical test. BMJ, 315, 629-34. https://doi.org/10.1136/bmj.315.7109.629
- Eichholzer M, Rohrmann S, Barbir A, et al (2012). Polymorphismsin heterocyclic aromatic amines metabolism-related genes are associated with colorectal adenoma risk. Int J Mol Epidemiol Genet, 3, 96-106.
- Gemignani F, Landi S, Szeszenia-Dabrowska N, et al (2007). Development of lung cancer before the age of 50: the role ofxenobiotic metabolizing genes. Carcinogenesis, 28, 1287-93. https://doi.org/10.1093/carcin/bgm021
- Hein DW (1988). Acetylator genotype and arylamine-induced carcinogenesis. Biochim Biophys Acta, 948, 37-66.
- Hein DW, Doll MA, Gray K, et al (1993). Metabolicactivation of N-hydroxy-2-aminofluorene and N-hydroxy2-acetylaminofluorene by monomorphic N-acetyltransferase(NAT1) and polymorphic N-acetyltransferase (NAT2) incolon cytosols of Syrian hamsters congenic at the NAT2 locus. Cancer Res, 53, 509-14.
- Hickman D, Pope J, Patil SD, et al (1998). Expression ofarylamine N-acetyltransferase in human intestine. Gut, 42,402-9. https://doi.org/10.1136/gut.42.3.402
- Hickman D, Risch A, Camilleri JP, et al (1992). Genotyping human polymorphic arylamine N-acetyltransferase: identification of new slow allotypic variants. Pharmacogenetics, 2, 217-26. https://doi.org/10.1097/00008571-199210000-00004
- Higgins JP, Thompson SG (2002). Quantifying heterogeneity in a meta-analysis. Stat Med, 21, 1539-58. https://doi.org/10.1002/sim.1186
- Hou YY, Ou HL, Chu ST, et al (2011). NAT2 slow acetylation haplotypes are associated with the increased risk of betelquid-related oral and pharyngeal squamous cell carcinoma. Oral Surg Oral Med Oral Pathol Oral Radiol Endod, 112,484-92. https://doi.org/10.1016/j.tripleo.2011.03.036
- Jang, JH, Cotterchio M, Borgida A, et al (2012). Genetic variantsin carcinogen-metabolizing enzymes, cigarette smoking andpancreatic cancer risk. Carcinogenesis, 33, 818-27. https://doi.org/10.1093/carcin/bgs028
- Kwon HT, Ma GX, Gold RS, et al (2013). Primary care physicians'cancer screening recommendation practices and perceptions of cancer risk of Asian Americans. Asian Pac J Cancer Prev, 14, 1999-2004. https://doi.org/10.7314/APJCP.2013.14.3.1999
- Landi S, Gemignani F, Moreno V, et al (2005). A comprehensiveanalysis of phase I and phase II metabolism genepolymorphisms and risk of colorectal cancer. PharmacogenetGenomics, 15, 535-46.
- Le Marchand L, Sivaraman L, Franke AA, et al (1996). Predictors of N-acetyltransferase activity: should caffeine phenotyping and NAT2 genotyping be used interchangeablyin epidemiological studies? Cancer Epidemiol BiomarkersPrev, 5, 449-55.
- Leiro-Fernandez V, Valverde D, Vazquez-Gallardo R, etal (2010). Genetic variations of NAT2 and CYP2E1and isoniazid hepatotoxicity in a diverse population. Pharmacogenomics, 11, 1205-6. https://doi.org/10.2217/pgs.10.109
- Majumdar S, Mondal BC, Ghosh M, et al (2008). Association of cytochrome P450, glutathione S-transferase and N-acetyltransferase 2 gene polymorphisms with incidence of acute myeloid leukemia. Eur J Cancer Prev, 17, 125-32. https://doi.org/10.1097/CEJ.0b013e3282b6fd68
- Mantel N, Haenszel W (1959). Statistical aspects of the analysisof data from retrospective studies of disease. J Natl Cancer Inst, 22, 719-48.
- Muthusamy KA, Lian LH, Vairavan N, et al (2012). Genetic polymorphisms of EGF 5'-UTR and NAT2 857G/A associated with glioma in a case control study of Malaysian patients. Genet Mol Res, 11, 2939-45. https://doi.org/10.4238/2012.June.15.7
- Nikishina MV, Vavilin VA, Makarova SI, et al (2007). Analysis of associations of NAT2 gene polymorphisms with the risk of lung cancer. Bull Exp Biol Med, 143, 83-6. https://doi.org/10.1007/s10517-007-0023-0
- Osian G, Procopciuc L, Vlad L (2006). NAT2 gene polymorphismand sporadic colorectal cancer. Prevalence, tumor stage and prognosis. A preliminary study in 70 patients. J Gastrointestin Liver Dis, 15, 347-53.
- Pande JN, Pande A, Singh SP (2003). Acetylator status, drug metabolism and disease. Natl Med J India, 16, 24-6.
- Probst-Hensch NM, Haile RW, Ingles SA, et al (1995).Acetylation polymorphism and prevalence of colorectaladenomas. Cancer Res, 55, 2017-20.
- Silveira VS, Canalle R, Scrideli CA, et al (2012). CYP3A5and NAT2 gene polymorphisms: role in childhood acutelymphoblastic leukemia risk and treatment outcome. Mol Cell Biochem, 364, 217-23. https://doi.org/10.1007/s11010-011-1220-8
- Wang G, Hou J, Ma L, et al (2012). Risk factor for clear cell renal cell carcinoma in Chinese population: a case-control study. Cancer Epidemiol, 36, 177-82. https://doi.org/10.1016/j.canep.2011.09.006
- Windmill KF, Gaedigk A, Hall PM, et al (2000). Localization of N-acetyltransferases NAT1 and NAT2 in human tissues. Toxicol Sci, 54, 19-29. https://doi.org/10.1093/toxsci/54.1.19
- Yamada S, Tang M, Richardson K, et al (2009). Genetic variations of NAT2 and CYP2E1 and isoniazid hepatotoxicity in adiverse population. Pharmacogenomics, 10, 1433-45. https://doi.org/10.2217/pgs.09.66
- Zanrosso CW, Emerenciano M, Faro A, et al (2012). Genetic variability in N-acetyltransferase 2 gene determinessusceptibility to childhood lymphoid or myeloid leukemia in Brazil. Leuk Lymphoma, 53, 323-7. https://doi.org/10.3109/10428194.2011.619605
- Zgheib NK, Shamseddine AA, Geryess E, et al (2013). Genetic polymorphisms of CYP2E1, GST, and NAT2 enzymes are not associated with risk of breast cancer in a sample of Lebanese women. Mutat Res, 747-748, 40-7. https://doi.org/10.1016/j.mrfmmm.2013.04.004
Cited by
- Genetic polymorphisms of NAT2 and risk of acute myeloid leukemia vol.96, pp.42, 2017, https://doi.org/10.1097/MD.0000000000007499