• Structural Engineering and Mechanics
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• 제64권5호
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• pp.621-640
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• 2017

This paper reports the numerical investigation conducted to study the influence of Local-Distortional (L-D) interaction mode buckling on post buckling strength erosion in fixed ended lipped channel cold formed steel columns. This investigation comprises of 81 column sections with various geometries and yield stresses that are carefully chosen to cover wide range of strength related parametric ratios like (i) distortional to local critical buckling stress ratio ($0.91{\leq}F_{CRD}/F_{CRL}{\leq}4.05$) (ii) non dimensional local slenderness ratio ($0.88{\leq}{\lambda}_L{\leq}3.54$) (iii) non-dimensional distortional slenderness ratio ($0.68{\leq}{\lambda}_D{\leq}3.23$) and (iv) yield to non-critical buckling stress ratio (0.45 to 10.4). The numerical investigation is carried out by conducting linear and non-linear shell finite element analysis (SFEA) using ABAQUS software. The non-linear SFEA includes both geometry and material non-linearity. The numerical results obtained are deeply analysed to understand the post buckling mechanics, failure modes and ultimate strength that are influenced by L-D interaction with respect to strength related parametric ratios. The ultimate strength data obtained from numerical analysis are compared with (i) the experimental tests data concerning L-D interaction mode buckling reported by other researchers (ii) column strength predicted by Direct Strength Method (DSM) column strength curves for local and distortional buckling specified in AISI S-100 (iii) strength predicted by available DSM based approaches that includes L-D interaction mode failure. The role of flange width to web depth ratio on post buckling strength erosion is reported. Then the paper concludes with merits and limitations of codified DSM and available DSM based approaches on accurate failure strength prediction.

• Journal of Chest Surgery
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• 제13권1호
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• pp.77-81
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• 1980

We present one case of 26-year-old male having saddle block combined with mitral valvular disease [NYHA Class IV] with auricular fibrillation. The most common cause of emboli is atrial fibrillation. The clinical manifestations of saddle emboli are relatively slow due to development of collateral circulation and large size of lumen of the aorta. The 5month duration of saddle emboli in this case led to severe atrophic changes, coldness, peripheral cyanosis on the both lower extremities, and flexion deformity on the knee and ankle joint of the left lower extremity. We planned staged operation for the saddle block and for mitral stenoinsufficiency and tricuspid insufficiency, because of poor general condition of the patient. The thromboembolectomy of aortic bifurcation was performed through the transabdominal approach without trial of Fogarthy catheter embolectomy, because of expectation of the secondary inflammatory changes of the vessel wall and thrombi which was 3 cm X 1 cm X 0.5 cm in size with irregular surfaced solid in consistency. 1 month later, after thromboembolectomy, mitral valve replacement and tricuspid annuloplasty were performed, with successful early operative result. During operation organized thrombi [1 cm X 0.5 cm] in the left auricle was removed. We wonder if simple management using Fogarthy catheter might be possible to remove the thromboemboli instead of thromboembolectomy by aortotomy in this case.

• 한국항해항만학회:학술대회논문집
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• 한국항해항만학회 2003년도 춘계공동학술대회논문집
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• pp.21-26
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• 2003

지금까지의 탄성설계 개념에서 진전하여 소성을 고려한 탄소성 설계 개념의 설계적용이 필요하다 본 연구에서는 선체의 대표적인 구조물인 판을 대상으로 일반적인 하중형증분법 및 변위형증분법과 같은 비선형해석 기법과 호장증분법과 Newton-Raphson법을 병용한 증분형해법을 비교하였다. 특히. 초기처짐의 형태에 따른 해석을 통하여 초지처짐의 형상에 따른 판의 탄소성거동을 규명하였다 압축하중을 받는 박판구조물은 초기좌굴후 거동과 2차좌굴후 거동이 구조물 전체에 미치는 영향이 매우 크며 이러한 복잡한 거동을 구하기 위해서 해석방법으로는 범용 유한요소해석 프로그램인 ANSYS를 이용하였고 특히 호장증분법은 좌굴점에서의 복잡한 비선형 거동을 추정하기 위하여 적용하였다.

• The Korean Journal of Legal Medicine
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• 제40권2호
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• pp.61-64
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• 2016

Ehlers-Danlos syndrome type IV (EDS IV) is a hereditary disorder of the connective tissue, characterized by easy bruising, thin skin with visible veins, and spontaneous rupture of the large arteries, uterus, or bowel. EDS IV is caused by mutations of the gene for type III procollagen (COL3A1), resulting in insufficient collagen production or a defect in the structure of collagen. EDS IV can have fatal complications such as the rupture of great vessels or organs, which can cause hemorrhaging and sudden unexpected death. Here, we report a case of a 43-year-old female who collapsed after a struggle with a neighbor. In this patient, the bifurcation of the bilateral common iliac artery ruptured, with no evidence of trauma, inflammation, or atherosclerosis. Genetic analysis of COL3A1 showed the presence of a c.2771G>A (p.Gly924Arg) mutation, which may be associated with EDS IV. The forensic pathologist should consider the possibility that the spontaneous visceral or arterial rupture was caused by EDS IV. Genetic analysis is not currently a routine procedure during autopsy. However, in this case, we suggest that the patient possibly had an underlying EDS IV condition, and we recommended family members of the deceased to seek genetic analysis and counseling.