• 제목/요약/키워드: indirect defenses

검색결과 3건 처리시간 0.014초

간접접근전략으로 본 6·25전쟁기 북한군의 호남 방면 대우회기동 실패 요인 분석 -'교란'을 저지한 해군·해병대의 통영상륙작전을 중심으로- (Analysis of the factors of the failure of the North Korean Army's Great Bypass Maneuver to Honam during the Korea War from an indirect approach strategy: Focusing on the Naval and Marine Corps' Tongyeong Amphibious Operation to Stop the "Dislocation")

  • 최호재
    • 해양안보
    • /
    • 제6권1호
    • /
    • pp.109-135
    • /
    • 2023
  • 북한군의 호남방면 대우회 기동은 6.25전쟁 기간 동안 북한군이 보여준 가장 빠른 기동이었으며, 국군과 연합군의 낙동강 방어선을 전면 조정하게 할 만큼 위협적인 공격이었다. 하지만 본 연구가 간접접근전략 측면에서 북한군의 호남방면 대우회기동을 분석한 결과, 이 기동에는 실패할 수밖에 없었던 여러 요인이 존재했다. 간접접근전략 수행 측면에 있어서 북한군은 전투력 분산, 작전선 변경의 융통성 부족, 지상군 위주의 기동, 심리적 견제 결여 등 여러 실패 요인을 노정했다. 하지만 북한군은 마지막 '기습'을 준비였는데, 그것은 북한군 6사단을 후속하던 7사단이 재차 우회를 감행하여 통영 방면으로 공격을 시도한 것이었다. 이를 통해 북한군은 낙동강 방어선을 돌파하고, 부산으로 향하고자 했다. 그러나 북한군의 공격은 한국 해군·해병대의 통영상륙작전으로 인해 최종 좌절되었다. 바다를 기동공간으로 한 신속한 기동으로 해군·해병대는 주요 요충지를 먼저 점령했고, 유리한 상황을 조성한 가운데 북한군 7사단의 공격을 막아냈다. 해군·해병대의 통영상륙작전은 북한군의 호남방면 대우회기동을 최종 저지함으로써 낙동강 방어선이 유지되도록 하였다.

  • PDF

Training Molecularly Enabled Field Biologists to Understand Organism-Level Gene Function

  • Kang, Jin-Ho;Baldwin, Ian T.
    • Molecules and Cells
    • /
    • 제26권1호
    • /
    • pp.1-4
    • /
    • 2008
  • A gene's influence on an organism's Darwinian fitness ultimately determines whether it will be lost, maintained or modified by natural selection, yet biologists have few gene expression systems in which to measure whole-organism gene function. In the Department of Molecular Ecology at the Max Planck Institute for Chemical Ecology we are training "molecularly enabled field biologists" to use transformed plants silenced in the expression of environmentally regulated genes and the plant's native habitats as "laboratories." Research done in these natural laboratories will, we hope, increase our understanding of the function of genes at the level of the organism. Examples of the role of threonine deaminase and RNA-directed RNA polymerases illustrate the process.

Role of RIN4 in Regulating PAMP-Triggered Immunity and Effector-Triggered Immunity: Current Status and Future Perspectives

  • Ray, Sujit Kumar;Macoy, Donah Mary;Kim, Woe-Yeon;Lee, Sang Yeol;Kim, Min Gab
    • Molecules and Cells
    • /
    • 제42권7호
    • /
    • pp.503-511
    • /
    • 2019
  • As sessile organisms, plants have developed sophisticated system to defend themselves against microbial attack. Since plants do not have specialized immune cells, all plant cells appear to have the innate ability to recognize pathogens and turn on an appropriate defense response. The plant innate immune system has two major branches: PAMPs (pathogen associated molecular patterns)-triggered immunity (PTI) and effector-triggered immunity (ETI). The ability to discriminate between self and non-self is a fundamental feature of living organisms, and it is a prerequisite for the activation of plant defenses specific to microbial infection. Arabidopsis cells express receptors that detect extracellular molecules or structures of the microbes, which are called collectively PAMPs and activate PTI. However, nucleotidebinding site leucine-rich repeats (NB-LRR) proteins mediated ETI is induced by direct or indirect recognition of effector molecules encoded by avr genes. In Arabidopsis, plasmamembrane localized multifunctional protein RIN4 (RPM1-interacting protein 4) plays important role in both PTI and ETI. Previous studies have suggested that RIN4 functions as a negative regulator of PTI. In addition, many different bacterial effector proteins modify RIN4 to destabilize plant immunity and several NB-LRR proteins, including RPM1 (resistance to Pseudomonas syringae pv. maculicola 1), RPS2 (resistance to P. syringae 2) guard RIN4. This review summarizes the current studies that have described signaling mechanism of RIN4 function, modification of RIN4 by bacterial effectors and different interacting partner of RIN4 in defense related pathway. In addition, the emerging role of the RIN4 in plant physiology and intercellular signaling as it presents in exosomes will be discussed.