• Speech Sciences
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• v.13 no.3
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• pp.185-195
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• 2006

This study aims to clarify disfluency loci in a neurogenic stuttering group and to examine how the characteristics are different from a developmental stuttering group. For the study, spoken language samples were collected from 11 adults with developmental stuttering and 11 adults with neurogenic stuttering in the course of speaking tasks including reading, monologue and conversation. Using the collected samples, disfluency characteristics of the two groups were to be investigated by analyzing adaptation effect, consistency effect and frequency of disfluency occurrence according to word position, which are related to the occurrence loci of disfluency. Results of this study were as follows: First, while the neurogenic stuttering group did not show any adaptation effect, the developmental stuttering group showed the adaptation effect that the percent of disfluency word reducing as they read the same materials repeatedly. Second, there was no meaningful difference of consistency effect between the two stuttering groups. Third, the neurogenic stuttering group showed more disfluency frequency in final sounds among the word position compared to the developmental stuttering group.

• Journal of Genetic Medicine
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• v.18 no.2
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• pp.75-82
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• 2021

Speech and language functions are highly cognitive and human-specific features. The underlying causes of normal speech and language function are believed to reside in the human brain. Developmental persistent stuttering, a speech and language disorder, has been regarded as the most challenging disorder in determining genetic causes because of the high percentage of spontaneous recovery in stutters. This mysterious characteristic hinders speech pathologists from discriminating recovered stutters from completely normal individuals. Over the last several decades, several genetic approaches have been used to identify the genetic causes of stuttering, and remarkable progress has been made in genome-wide linkage analysis followed by gene sequencing. So far, four genes, namely GNPTAB, GNPTG, NAGPA, and AP4E1, are known to cause stuttering. Furthermore, thegeneration of mouse models of stuttering and morphometry analysis has created new ways for researchers to identify brain regions that participate in human speech function and to understand the neuropathology of stuttering. In this review, we aimed to investigate previous progress, challenges, and future perspectives in understanding the genetics and neuropathology underlying persistent developmental stuttering.

• The Journal of the Korea Contents Association
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• v.13 no.11
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• pp.269-276
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• 2013

This study was aimed of the effects of the Parent's Gestalt Speech Intervention for stuttering development of Fluency disorder Child. The Parent's Gestalt Speech Intervention was made up of a program understand phase, an awareness phase, a change phase and, finally, an arrangement and termination phase. The subjects 6 (female 2, male 4) of this research were developed a stuttering behavior without any apparent neurological damage or other speech or developmental impediments. To access their stuttering behaviors, I used methods for observing levels of behavioral in each phase. The results of the study are as follows: In regard to stuttering behavior, word repetition frequency decreased in the interim assessments, showing that the learning of fluent speech was acquired early in the therapy process. In conclusion, the results of the study show that Parent's Gestalt Speech Intervention for stuttering development of Fluency disorder Child.

• Speech Sciences
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• v.15 no.4
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• pp.189-197
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• 2008

In the process of speech therapy, fluency is acquired and speech rate increases in the process when the behavioral modification strategy, inducing speech fluency by making speech rate slower intentionally in an early stage, is applied. Therefore, the purpose of this study was to investigate the pause characteristics in slowed speech intentionally of treated stutterer. In this study, 10 developmental stutterers who had well established fluency in speech were involved. We had collected each 200 syllables sample of intentionally much slowed speech and a little slowed one in reading task. To measure the features of pause, total frequency of pauses, total durations of pauses, average duration of pauses and proportions of pause were investigated. The findings were as follows: Both the total durations and total frequency of pauses of much slowed speech were higher than that of a little slowed one. However, both the average duration and proportions of pauses of much slowed speech were not significantly higher than that of a little slowed one.

• Journal of the Korean Academy of Child and Adolescent Psychiatry
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• v.4 no.1
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• pp.124-132
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• 1993

We studied the clinical characteristics of 45 children with tic symptoms, and a comparison was made according to their birth order patterns. The results were as follows. Sex ration was 14:1 higher for boys. The eldest children were 46.7%, single children, 15.6%, the youngest children, 33.3%, and twins 4.4% of all Organicity was suggested in 37.8%, early developmental problems in 71.1%, and family problems were in 89.9%. Among the co-existing problems, ADHD 46.7%, OCD 17.7%, Separation anxiety disorder 24.4%, GAD or anxiety dreams 17.8%, somatization disorder and enuresis 13.3% each, stuttering 8.9%, and other conditions. Overall, 84.4% of the patients have one or more co-existing conditions other than tic symptoms. When compared according to birth order patterns, the most significant difference was the time of onset The youngests have more incidences around the entrance period for elementary school(p<0.01). Among the twins, the lower birth-weight child was the patient. Summing up these findings, we concluded there were significant environmental factors working on the manifestation of tic and tourette disorders.