• Journal of the Korea Institute of Information and Communication Engineering
• /
• v.6 no.7
• /
• pp.1064-1068
• /
• 2002

In this paper, we introduce a hyper-chaos synchronization method using State-Controlled Cellular Neural Network(SC-CNN). We make a hyper-chaos circuit using SC-CNN with the n-double scroll. A hyper-chaos circuit is created by applying identical n-double scrolls with weak coupled method, to each cell. Hyper-chaos synchronization was achieved using drive response synchronization between the transmitter and receiver about each state in the SC-CNN. From the result of the recovery signal through the demodulation method in the receiver, We shown that recovery quality of state variable $$\chi$_3$ is superior to that of ${$\chi$_2}, {$\chi$_1}$ in secure communication.

• Journal of information and communication convergence engineering
• /
• v.1 no.4
• /
• pp.223-228
• /
• 2003

In this paper, we introduce a hyper-chaos secure communication method using hyper-chaos circuit onsist of State-Controlled Cellular Neural Network SC-CNN). We make a hyper-chaos circuit using SC-CNN with the n-double scroll or Chua's oscillator. A hyper-chaos circuit is created by applying identical n-double scroll or non-identical n-double scroll and Chua's oscillator with weak coupled method to each cell. Hyper-chaos ynchronization was achieved using GS (Generalized Synchronization) method between the transmitter and receiver about each state variable in the SC-CNN. In order to secure communication, we have synthesizing the desired information with a hyper-chaos circuit by adding the information signal to the hyper-chaos signal using the SC-CNN in the transmitter. And then, transmitting the synthesized signal to the ideal channel, we confirm secure communication by separating the information signal and the hyper-chaos signal in the receiver.

• The Korean Journal of Physiology and Pharmacology
• /
• v.2 no.3
• /
• pp.297-305
• /
• 1998

This study used in vivo intracellular recording in rat hippocampus to evaluate the effect of lidocaine and MK-801 on the membrane properties and the synaptic responses of individual neurons to electrical stimulation of the commissural pathway. Cells in control group typically fired in a tonic discharge mode with an average firing frequency of $2.4{\pm}0.9$ Hz. Neuron in MK-801 treated group (0.2 mg/kg, i.p.) had an average input resistance of $3.28{\pm}5.7\;M{\Omega}$ and a membrane time constant of $7.4{\pm}1.8$ ms. These neurons exhibited $2.4{\pm}0.2$ ms spike durations, which were similar to the average spike duration recorded in the neurons of the control group. Slightly less than half of these neurons were firing spontaneously with an average discharge rate of $2.4{\pm}1.1$ Hz. The average peak amplitude of the AHP following the spikes in these groups was $7.4{\pm}0.6$ mV with respect to the resting membrane potential. Cells in MK-801 and lidocaine treated group (5 mg/kg, i.c.v.) had an average input resistance of $3.45{\pm}6.0\;M{\Omega}$ and an average time constant of $8.0{\pm}1.4$ ms. The cells were firing spontaneously at an average discharge rate of $0.6{\pm}0.4$ Hz. Upon depolarization of the membrane by 0.8 nA for 400 ms, all of the tested cells exhibited accommodation of spike discharge. The most common synaptic response contained an EPSP followed by early-IPSP and late-IPSP. Analysis of the voltage dependence revealed that the early-IPSP and late-IPSP were putative $Cl^--and\;K^+-dependent$, respectively. Systemic injection of the NMDA receptor blocker, MK-801, did not block synaptic responses to the stimulation of the commissural pathway. No significant modifications of EPSP, early-IPSP, or late-IPSP components were detected in the MK-801 and/or lidocaine treated group. These results suggest that MK-801 and lidocaine manifest their CNS effects through firing pattern of hippocampal pyramidal cells and neural network pattern by changing the synaptic efficacy and cellular membrane properties.