• 한국체육학회지인문사회과학편
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• v.55 no.4
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• pp.85-107
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• 2016

By examining the constitutive characteristics of Yon-Ko-Jeon and analyzing the lived meanings and experiencing essence of students, this study details the students' awareness, responses, and strategies with regards to the phenomenon of women's alienation from Yon-Ko-Jeon. Students from the two colleges were divided into two groups- one which was aware of women's alienation from Yon-Ko-Jeon, and another group that was unaware of such a phenomenon. Students without awareness tended to perceive the alienation of women from Yon-Ko-Jeon as a natural phenomenon. On the other hand, the members who had awareness tended to see it as a phenomenon that was either justified, unequal, or in need of change. In the latter group, those who saw women's alienation as a justified phenomenon responded with tolerance and adopted a bystander strategy. Conversely, those who saw it as an unequal phenomenon responded with rejection and instead chose to adopt anti-/alternative Yon-Ko-Jeon strategies. The last group, which perceived women's alienation as a phenomenon in need of change, adopted correcting responses as well as intercollegiate solidarity and cooperation strategies. A generalization of the findings reveals that the students' experiencing essence of the phenomenon extends from critical thinking to awareness, response, and strategy in a step-by-step and sequential process. Through this study, it was found that the students' experiencing essence all follow a continuous thinking pattern, as illustrated in the diagram provided.

• 한국체육학회지인문사회과학편
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• v.55 no.5
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• pp.447-457
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• 2016

The purpose of this study is to check awareness of 'the phenomenon of women's alienation in Yon-Ko-Jeon' to women students, and improve the gender equality for Yon-Ko-Jeon through IPA(Importance Performance Analysis). For this study, 322 women undergraduates at Yonsei University and Korea University, from sophomore to senior students, are selected and asked to be surveyed. At first, a majority of women students from two universities didn't recognize the lack of women sports and athletes in Yon-Ko-Jeon. Moreover, they didn't think about women's alienation in Yon-Ko-Jeon as well as improvement for the issue seriously. It shows the life style of gender in the sport. Second, as a result of IPA, improvement plan of Yon-Ko-Jeon on gender equality shows it needs to reflect member's opinion and introduce women sports clubs first. Recently, the improvement for the Yon-Ko-Joen is the top priority because the event doesn't reflect member's opinion. Furthermore, it shows that Yon-Ko-Jeon needs to introduce the competition of women's club sports. These are what the event has to improve.

• The Korean Journal of Physiology and Pharmacology
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• v.23 no.3
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• pp.191-201
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• 2019

The transient receptor potential canonical (TRPC) 5 channel, known as a nonselective cation channel, has a crucial role in calcium influx. TRPC5 has been reported to be activated by muscarinic receptor activation and extracellular pH change and inhibited by the protein kinase C pathway. Recent studies have also suggested that TRPC5 is extracellularly activated by englerin A (EA), but the mechanism remains unclear. The purpose of this study is to identify the EA-interaction sites in TRPC5 and thereby clarify the mechanism of TRPC5 activation. TRPC5 channels are over-expressed in human embryonic kidney (HEK293) cells. TRPC5 mutants were generated by site-directed mutagenesis. The whole-cell patch-clamp configuration was used to record TRPC5 currents. Western analysis was also performed to observe the expression of TRPC5 mutants. To identify the EA-interaction site in TRPC5, we first generated pore mutants. When screening the mutants with EA, we observed the EA-induced current increases of TRPC5 abolished in K554N, H594N, and E598Q mutants. The current increases of other mutants were reduced in different levels. We also examined the functional intactness of the mutants that had no effect by EA with TRPC5 agonists, such as carbachol or $GTP{\gamma}S$. Our results suggest that the three residues, Lys-554, His-594, and Glu-598, in TRPC5 might be responsible for direct interaction with EA, inducing the channel activation. We also suggest that although other pore residues are not critical, they could partly contribute to the EA-induced channel activation.