• Title/Summary/Keyword: Vestibulospinal reflex

Search Result 5, Processing Time 0.021 seconds

Vestibular Evoked Myogenic Potential (전정유발근육전위)

  • Kim, Sung Hun;Nam, Eui Cheol
    • Annals of Clinical Neurophysiology
    • /
    • v.7 no.1
    • /
    • pp.1-6
    • /
    • 2005
  • Loud click or tone burst sound can activate vestibular receptor and evoke reflex changes in tonic electromyographic activity within the stenocleidomastoid muscles. This reflex is assumed to originate in the saccule, the afferent pathways being the inferior vestibular nerve, and the efferent pathways the vestibulospinal tract. Averaging these muscular responses allows vestibular evoked myogenic potentials (VEMP) to be obtained. The earliest response ipsilateral to a loud click, p13n23, is dependent upon vestibular activation, specifically saccular afferents. These new techniques are beginning to be applied clinically in the patient of vestibular neuritis, Meniere's disease, acoustic neuromas, Tullio phenomenon, etc. VEMP recording will provide both a straightforward non-invasive exploration of each vestibule independently and an attractive method by which to explore otolithic receptors and vestibulospinal pathways.

  • PDF

The Change of H Reflex by Galvanic Vestibular Stimulation (평류전정자극에 의한 H 반사의 변화)

  • Hwang, Tae-Yeun;Kim, Tae-Youl;Park, Jang-Sung
    • Journal of the Korean Academy of Clinical Electrophysiology
    • /
    • v.2 no.3
    • /
    • pp.65-73
    • /
    • 2004
  • In experimental method, this study was that galvanic vestibular stimulation in vestibular system influenced the excitability of spinal neuron through. H-reflex was measured by galvanic vestibular stimulation of binaural(right-negative pole and left-positive pole) at left head turning and prone position in sixteen normal subjects in their twenties age were selected. The summary of the comparison results were obtained below. 1. In the change of H reflex according to galvanic vestibular stimulation(GVS), Hmax amplitude(p<.05) increased significantly after stimulation. 2. In the change of H reflex according to galvanic vestibular stimulation(GVS), Hmax/Mmax ratio(p<.05) increased significantly after stimulation. In the conclusion, galvanic vestibular stimulation influenced the excitability of vestibulospinal tract and spinal neuron.

  • PDF

A Method for Slow Component Velocity Measurement of Nystagmus Eye Movements using RLSM (RLSM을 이용한 안구운동의 저속도 측정방법에 대한 연구)

  • Kim Gyu-Gyeom;Ko Jong-Sun;Park Byung-Rim
    • Proceedings of the KIPE Conference
    • /
    • 2002.07a
    • /
    • pp.455-458
    • /
    • 2002
  • A control of the body posture and movement is maintained by the vestibular system, vision, and proprioceptors. Especially, vestibular system has a very important function that controls the eye movement through vestibuloocular reflex and contraction of skeletal muscles through vestibulospinal reflex. However, postural disturbance caused by loss of vestibular function results in nausea, vomiting, vertigo and loss of craving for life. Lose of vestibular function leads to abnormal reflex of eye movements named nystagmus. Analysis of the nystagmus is needed to diagnose the vertigo, which is performed by means of electronystagmography (ENG). The purpose of this study is to develop a computerized system for data processing and an algorithm for the automatic evaluation of the slow component velocity (SCV) of nystagmus Induced by optokinetic(OKN) stimulation system. A new algorithm using recursive least square method (RLSM) to detect SCV of nystagmus is suggested in this paper. This method allows a fast and precise evaluation of the nystagmus, through artifact rejection techniques. The results are depicted in this paper.

  • PDF

A Method for Slow Component Velocity Measurement of Nystagmus Eye Movements using RLSM (RLSM을 이용한 안구운동의 저속도 측정방법에 대한 연구)

  • 김규겸;고종선;박병림
    • The Transactions of the Korean Institute of Power Electronics
    • /
    • v.7 no.6
    • /
    • pp.546-553
    • /
    • 2002
  • A control of the body posture and movement is maintained by the vestibular system, vision, and proprioceptors. Especially, vestibular system has a very important function that controls the eye movement through vestibuloocular reflex and contraction of skeletal muscles through vestibulospinal reflex. However, postural disturbance caused by loss of vestibular function results in nausea, vomiting, vertigo and loss of craving for life. Lose of vestibular function leads to abnormal reflex of eye movements named nystagmus. Analysis of the nystagmus is needed to diagnose the vertigo, which is performed by means of electrooculography(EOG). The purpose of this study is to develop a computerized system for data processing and an algorithm for the automatic evaluation of the slow component velocity(SCV) of nystagmus induced by optokinetic(OKN) stimulation system. A new algorithm using recursive least square method(RLSM) to detect SCV of nystagmus is suggested in this paper. This method allows a fast and precise evaluation of the nystagmus, through artifact rejection techniques. The results are depicted in this paper.

Electrical Stimulation Can Facilitate Vestibular Compensation Following Unilateral Labyrinthectomy in Rats

  • Park, Byung-Rim;Kim, Jin-Ok;Kim, Min-Sun;Lee, Moon-Young;Kim, Jae-Hyo;Jin, Yuan-Zhe;Choi, Myung-Ae
    • The Korean Journal of Physiology and Pharmacology
    • /
    • v.5 no.2
    • /
    • pp.123-131
    • /
    • 2001
  • To investigate the effects of electrical stimulation on vestibular compensation, which is the recovery of vestibular symptoms following unilateral labyrinthectomy (UL), intermittent electrical stimulation was applied to the injured vestibular portion in Sprague-Dawley rats. Vestibuloocular and vestibulospinal reflexes, electrical activity and expression of c-Fos protein in medial vestibular nuclei (MVN) were measured with time following UL. Spontaneous nystagmus occurred with frequency of $2.9{\pm}0.2$ beats/sec at 2 hours after UL and disappeared after 72 hours. Electrical stimulation decreased the frequency of nystagmus significantly till 24 hours after UL. Roll head deviation was $107{\pm}9.7^{\circ}$ at 2 hours after UL and the deviation was maintained till 72 hours, but electrical stimulation decreased the deviation significantly 6 hours after UL. Resting activity of type I neurons in ipsilateral MVN to the injured vestibular side decreased significantly compared with control at 6 and 24 hours after UL, but the activity of type I neurons was recovered to control level by electrical stimulation at 24 hours after UL. Gain of type I neurons induced by sinusoidal rotation of 0.1 Hz decreased significantly till 24 hours after UL, but electrical stimulation restored the activity at 24 hours. The gain of type II neurons decreased significantly at 6 hours after UL, but electrical stimulation restored the activity. Expression of c-Fos protein was asymmetric between bilateral MVN till 24 hours after UL, but the asymmetry disappeared by electrical stimulation 6 hours after UL. These results suggest that electrical stimulation to the injured vestibular portion facilitates vestibular compensation following UL by restoration of symmetry of neuronal activity between bilateral vestibular nuclei resulting from increased activity in ipsilateral vestibular nuclei to the injured side.

  • PDF