• 오리마을
• /
• 제63권9호
• /
• pp.116-117
• /
• 2008

• 오리마을
• /
• 제63권9호
• /
• pp.134-136
• /
• 2008

• 오리마을
• /
• 통권85호
• /
• pp.58-63
• /
• 2010

• 오리마을
• /
• 통권189호
• /
• pp.56-63
• /
• 2019

• 오리마을
• /
• 통권209호
• /
• pp.58-63
• /
• 2020

• 오리마을
• /
• 통권208호
• /
• pp.58-63
• /
• 2020

• 한국항해항만학회:학술대회논문집
• /
• 한국항해항만학회 2014년도 춘계학술대회
• /
• pp.284-286
• /
• 2014

국제수로기구(IHO: International Hydrographic Organization)는 전자 해도의 불법 복사와 사용을 방지하기 위해 전자해도 보안 표준(IHO Data Protection Scheme, Edition 1.1.1 - April 2012, IHO Publication S-63)을 제정한 바 있으며, 국제적으로는 보안체계가 적용된 암호화된 전자 해도가 공급되고 있다. 국립해양조사원에서 국내 사용을 위해 공급하는 전자 해도에는 보안 표준을 적용하지 않다가 2014년부터 전자 해도의 비 항해 목적으로의 사용을 방지하기 위해 보안 표준 적용과 함께 비 항해용 해도 정보를 제공하고 있다. 따라서 전자해도 시스템 관점에서 전자 해도를 사용하기 위해서는 전자해도 보안 표준에 대한 높은 지식이 요구되고 있다. 이에 본 연구에서는 전자해도 보안 표준과 체계를 분석하고, 표준에서 제공하는 전자해도 보안 표준 테스트 데이터를 전자해도 시스템 관점에서 시험하였다. 이를 통해 보안 모듈과 전자해도 시스템과의 관계를 소프트웨어 구조 관점에서 제시하고 표준을 적용할 때 필요로 하는 표준 접근 절차와 방법을 연구결과로 제시 하였다.

• 대한영양사협회학술지
• /
• 제10권4호
• /
• pp.401-406
• /
• 2004

The purposes of this study was to evaluate the foodservice quality on the viewpoint of personnel in hospital foodservice. In result, S hospital's foodservice quality, that was evaluated by personnel, was comparatively high on the every variables and factors, especially on 'Q13. Meals delivery to the bed'(4.70 out of 5.0), 'Q14. Removal service of tray by foodservice personnel'(4.63), 'Q15. Kind foodservice personnel'(4.63), 'Q16. Foodservice personnel's clean and neat uniforms'(4.56) and 'factor 4. personnel attitude'(4.63). Foodservice quality factors weren't significant by general characteristics of personnel, thus all personnel were considered to hold similarly the level of foodservice quality. On the basis of these results, the gap between foodservice providers and customers need to be analyzed in the further study.

• Asian Pacific Journal of Cancer Prevention
• /
• 제16권11호
• /
• pp.4633-4639
• /
• 2015

Background: Previous studies suggested that the H63D and C282Y polymorphisms in the HFE genes were susceptible to many cancer types, nevertheless, the present results were inconclusive. Thus, the present study was aimed to evaluate the association between the HFE polymorphisms (H63D and C282Y) and cancer risk via meta-analysis. Materials and Methods: We retrieved PubMed, Google Scholar, Embase and Web of Science databases for all eligible studies up to April 1, 2015. All the statistical analysis was conducted by STATA 12.0. Results: Finally, a total of 20 publications including 24 case-control studies, comprising 6,524 cases and 31,080 controls for HFE-C282Y polymorphism and 19 publications including 21 case control studies, comprising 5,648 cases and 14,257 controls for HFE-H63D polymorphism were enrolled in our analysis. An increased risk for overall cancer risk was identified in HFE-H63D polymorphism under allele contrast (D vs H: OR=1.153; 95%CI=1.031-1.289, Pheterogeneity=0.002), homozygotes vs wide type (DD vs HH: OR=1.449; 95%CI=1.182-1.777, Pheterogeneity=0.391), dominant model (DD+HD vs HH: OR=1.145; 95%CI=1.007-1.301, Pheterogeneity=0.002) and recessive model (DD vs HD+HH: OR=1.416 ; 95%CI=1.156-1.735, Pheterogeneity=0.549), as well as HFE-C282Y under homozygotes vs wide type (YY vs CC: OR=1.428, 95%CI=1.017-2.006, Pheterogeneity=0.220). In addition, in the stratified analysis by cancer type, an increased risk was identified in hepatocellular carcinoma and breast cancer in C282Y polymorphism, as well as pancreatic cancer in H63D polymorphism, whereas a decreased risk of colorectal cancer was identified in C282Y polymorphism. Conclusions: Present study suggested that H63D and C282Y polymorphisms associated with an increased risk of overall cancer. Nevertheless, well-designed study with large sample size will be continued on this issue of interest.

• Asian Pacific Journal of Cancer Prevention
• /
• 제14권6호
• /
• pp.3751-3755
• /
• 2013

Backgroud and Aims: MicroRNA-206 has proven to be down-regulated in many human malignancies in correlation with tumour progression. Our study aimed to characterize miR-206 contributions to initiation and malignant progression of human osteosarcoma. Methods: MiR-206 expression was detected in human osteosarcoma cell 1ine MG63, human normal osteoblastic cell line hFOB 1.19, and paired osteosarcoma and normal adjacent tissues from 65 patients using quantitative RT-PCR. Relationships of miR-206 levels to clinicopathological characteristics were also investigated. Moreover, miR-206 mimics and negative control siRNA were transfected into MG63 cells to observe effects on cell viability, apoptosis, invasion and migration. Results: We found that miR-206 was down-regulated in the osteosarcoma cell line MG63 and primary tumor samples, and decreased miR-206 expression was significantly associated with advanced clinical stage, T classification, metastasis and poor histological differentiation. Additionally, transfection of miR-206 mimics could reduce MG-63 cell viability, promote cell apoptosis, and inhibit cell invasion and migration. Conclusions: These findings indicate that miR-206 may have a key role in osteosarcoma pathogenesis and development. It could serve as a useful biomarker for prediction of osteosarcoma progression, and provide a potential target for gene therapy.