• 제목/요약/키워드: Puccinia graminis f. sp. tritici

검색결과 2건 처리시간 0.016초

한국에 분포한 맥류 줄기녹병균(Puccinia graminis f. sp. tritici)의 생리적 품종 (Physiologic Races of Puccinia graminis f. tritici in Korea)

  • 정봉구;이지영
    • 한국응용곤충학회지
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    • 제12권2호
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    • pp.79-82
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    • 1973
  • 1. 맥류 줄기 녹병균에(p. graminis f. sp. tritici) 대한 저항성 품종 육종에 기초자료를 얻고자 1971-1972년 2개년간에 한국에 분포한 생리적 품종을 조사하였다. 2. 2개년간 총 55개의 균주를 채점하여 Stakman씨 방법에 따라 접종한 결과 27개 균주가 동정되었는데 그 성적은 다음과 같다. Race 11이 2균주, Race 17과 21은 각각 8균주 Race 34의 4균주 그리고 Race 37과 56이 각각 한 균주였다. 그 나머지 3균주는 Stakman씨의 색인표와 부합치 않으므로 가칭 $K_3K_4$$K_5$라고 명명하였다. 3. 그러므로 맥류줄기녹병균에 대한 생리적 품종의 조사는 저항성 품종 육종과 병행하여 계속 실시되어 야 할 것이다.

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Insights into Tan Spot and Stem Rust Resistance and Susceptibility by Studying the Pre-Green Revolution Global Collection of Wheat

  • Abdullah, Sidrat;Sehgal, Sunish Kumar;Jin, Yue;Turnipseed, Brent;Ali, Shaukat
    • The Plant Pathology Journal
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    • 제33권2호
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    • pp.125-132
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    • 2017
  • Tan spot (TS), caused by the fungus Pyrenophora tritici-repentis (Died) Drechs, is an important foliar disease of wheat and has become a threat to world wheat production since the 1970s. In this study a globally diverse pre-1940s collection of 247 wheat genotypes was evaluated against Ptr ToxA, P. tritici-repentis race 1, and stem rust to determine if; (i) acquisition of Ptr ToxA by the P. tritici-repentis from Stagonospora nodorum led to increased pathogen virulence or (ii) incorporation of TS susceptibility during development stem rust resistant cultivars led to an increase in TS epidemics globally. Most genotypes were susceptible to stem rust; however, a range of reactions to TS and Ptr ToxA were observed. Four combinations of diseasetoxin reactions were observed among the genotypes; TS susceptible-Ptr ToxA sensitive, TS susceptible-Ptr ToxA insensitive, TS resistant-Ptr ToxA insensitive, and TS resistant-Ptr ToxA toxin sensitive. A weak correlation (r = 0.14 for bread wheat and -0.082 for durum) was observed between stem rust susceptibility and TS resistance. Even though there were no reported epidemics in the pre-1940s, TS sensitive genotypes were widely grown in that period, suggesting that Ptr ToxA may not be an important factor responsible for enhanced prevalence of TS.