• 한국약용작물학회지
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• 제20권5호
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• pp.315-319
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• 2012

This study was conducted to select plant growth regulators effective at ginseng berry set inhibition to help root growth in Korean ginseng (Panax ginseng C. A. Meyer). PGRs (ethephon, gibberellic acid, maleic hydrazide, coumarin) were applied to field grown 5-year-old Korean ginseng between one and two times, before and during bloom in 2009, 2010. The number of treatment was more effective in ginseng berry set inhibition when used two times compared with one time in GA 1,000 ppm, MH (5,000, 10,000 ppm), coumarin (5,000, 10,000 ppm) treatment. According to treatment period of plant growth regulator, ginseng berry set inhibition rate from 20days before flowering date to 5days after blooming was the highest in MH 5000 ppm showing 99.9% and the lowest in GA 100 ppm showing 32.8%. The spray treatments of Ethephon (50, 150 ppm) and MH (5,000, 10,000 ppm) from 20 days before the flowering bloom up to 5 days before, and coumarin (5,000, 10,000 ppm) from 20 days to 6 days and before blooming that induced the inhibitory effect more than 90% after 12 weeks. Considering ginseng berry set inhibition characteristics and treatment period ethephon and coumarin was important about applied period but, MH treatment appeared to effective ginseng berry set inhibition regardless of treatment period.

• 한국동물학회지
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• 제20권1호
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• pp.19-28
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• 1977

In order to evaluate the mutagenic potential in animal for these pesticides which were proved to be mutagenic in the bacterial screening system with a metabolic activation in vitro, we have studied in vivo cytogenetic effects on mouse bone marrow by means of the micronucleus test. The clastogenic activity of the chemical is evaluated as the frequency of micronuclei in polychromatic erythrocytes. We have tested six pesticides, insecticides, DDVP and trichlorfon, fungicide, TMTD, herbicides, NIP and MO and growth regula색, maleic hydrazide. It was found that among the tested pesticides only TMTD exhibited minimal activity in inducing micronuclei. Organophosphorus insecticide DDVP that is the most broadly used and economically important chemical, did not increase the micronuclei frequencies in mouse bone marrow cells as with the all other pesticides tested.

• 한국식품과학회지
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• 제35권6호
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• pp.1007-1016
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• 2003

마늘의 녹변 유도기간과 저온저장 마늘의 녹변억제 방법을 살펴 보기위하여 수확된 마늘을 4, 8, $12^{\circ}C$에서 저장하면서 녹변생성 시점을 조사한 결과 $4^{\circ}C$에서는 7일만에 녹변이 발현되었고, 8 및 $12^{\circ}C$에서 저장할 때는 약 15일후에 녹변이 발생하였다. 감마선을 조사한 마늘은 대조구에 비해 녹변이 촉진되었으며, 생장포절제인 maleic hydrazide는 마늘 녹변발생과는 무관하였다. 녹변방지를 위해 마늘에 cysteine을 첨가한 결과 녹변현상과 흰반점이 발생하여 상품으로서 부적합하였다. 마늘에 3% ascorbic acid를 첨가하여 녹변 생성을 경시적으로 관찰한 결과 대조구는 약 30분만 실온에서 녹변이 발현된데 비하여 ascorbic acid 첨가구는 6시간이 지난 후 발현되었으나 저온에 저장하였을 때는 24시간 안정하였다. 한편, 수확한 마늘을 일정기간 저온저장시 생리적 냉해(chilling injury)현상으로 정상적인 대사가 억제되어 녹변현상이 발생한다는 가설하에 온도조절에 의한 녹변억제 경향을 조사하였다. 30일 동안 저온 저장한 마늘을 20, 25, 30, 35, 40, $45^{\circ}C$에서 관찰한 결과, 대조구, 20, $25^{\circ}C$에서 온도조절한 마늘은 20일이 경과한 후에도 지속적으로 녹변이 유지되었고, $30^{\circ}C$에서 온도조절한 마늘은 20일이 되어서야 녹변이 억제되었다. 또한 35, 40, $45^{\circ}C$에서 온도조절한 마늘은 4일이 경과되었을 때 비녹변 마늘과 비슷한 L, a, b값을 나타내고 있었으며 육안으로도 녹변현상이 억제되었음을 확인할 수 있었다. 따라서 본 실험결과로부터 물리적인 방법에 의한 녹변억제는 $35^{\circ}C$에서 4일간 온도조절하였을 때 가장 양호하였다.

• 한국연초학회지
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• 제23권1호
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• pp.5-11
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• 2001

Experiments were conducted to evaluated the sucker control efficiency of several suckercides and their combinations for flue-cured tobacco in Korea. In 1999, Flumetralin(Prime+, P+), Butralin(B), Fatty alcohol(FA, n-decanol 85%), pottasium salt of maleic hydrazide(MH), tank mix of MH with P+ (MH·P+), and another tank mix of MH with FA(MH·FA) were applied after topping, respectively, then sucker number and sucker dry weight were investigated on the 30th and 50th day from treatment. Sucker number and dry weight were significantly lowered by P+, B, FA and MH·P+ applications compared with those of MH and MH·FA for both investigated periods. A significant reduction in sucker number and dry weight was observed in P+, B, and MH·P+ applications. FA treatment was not so much effective in sucker control as P+. In 1999 and 2000, several suckercides and tank mixes of their combinations were applied first at button stage and second after topping, respectively, to compare the effects of sucker control on yield, price index and chemical components of cured leaves. Compared to the standard method of FA 30x at button stage followed by MH40x application after topping(FA 30x + MH 40x), sucker number and dry weight decreased by [FA 30x (or P+ 1%] or [FA 30x(or B 1.5%) + MH 60x·B 1.5%] treatments, even at reduced MH levels. But yield, price index and chemical components of cured leaves were not differed among treatments.

• 한국작물학회지
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• 제42권5호
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• pp.609-614
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• 1997

땅콩의 무효개화를 유제하여 정입율이 높은 재배법을 강구하기 위하여 C-MH를 개화후 24, 28, 31일에 0.2％액, 0.1％액, 0.067％액으로 달리하여 처리한 시험 결과는 다음과 같다. 1. 주경장은 무처리에 비하여 38∼44％ 감소되었으나 분지수는 처리시기 및 처리농도에 관계없이 변화가 거의 없었다. 2. 처리후 28일의 개화유제율은 0.2∼0.1％액 처리시 29∼38％, 0.067％액 처리시 22％였다. 3. C-MH처리에 따른 완숙입율(=정입율)은 무처리구(56％)에 비하여 개화후 28일 처리시 55∼59％이고, 이 중 0.1％액 처리구에서 59％로서 3％가 향상되었다. 4. 개화후 28일에 0.1∼0.067％액처리구와 개화후 31일에 0.1％액처리구의 수량이 무처리구와 대등하였다.

• Current Research on Agriculture and Life Sciences
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• 제1권
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• pp.35-40
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• 1983

식물생장조절제(植物生長調節劑)에 의(依)한 담배의 액아발생(腋芽發生)에 관(關)한 기초자료(基礎資料)를 얻고자 조생종(早生種)인 hicks와 만생종(晩生種)인 kusaga mammoth에 MH, GA, BA의 농도를(濃度)를 달리해서 단독(單獨) 또는 혼용처리(混用處理) 하였을 시 액아발생(腋芽發生)에 미치는 영향(影響)을 구명(究明)한바 그 결과(結果)를 요약(要約)하면 다음과 같다. 적심(摘心) 직후(直後) MH900g(a.i.)/ha 처리(處理)는 액아발생(腋芽發生)을 완전히(完全)히 억제(抑制)시켰지만 농도(濃度)가 감소(減少)함에 따라 품종(品種)에 관계(關係)없이 액아(腋芽)가 발생(發生)하였다. 무적심(無摘心) 개체(個體)에서도 비슷한 경향(傾向)을 나타내었다. 적심(摘心)한 개체(個體)에서는 GA와 BA의 여느 농도(濃度)에서 혼용(混用)을 하여도 MH가 첨가(添加)되면 액아(腋芽)가 발생(發生)하지 않지만 무적심(無摘心) 개체(個體)에서는 MH 900 g(a. i.)/ha에 BA와 GA의 농도비(濃度比)가 10:1로 혼용(混用)될 때는 액아(腋芽)가 발생(發生)하였다. 무적심(無摘心) 개체(個體)에 GA와 BA 처리(處理)는 액아발생(腋芽發生)에 아무런 영향(影響)을 미치지 않았다. 적심(摘心)한 개체(個體)에 GA와 BA의 단독처리(單獨處理)는 농도(濃度)가 높아짐에 따라 액아발생(腋芽發生)을 증가(增加)시키며 혼용(混用)하였을 때에는 antagonistio한 경향(傾向)을 나타내었다.

• 한국식품위생안전성학회지
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• 제14권2호
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• pp.201-215
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• 1999

Limited information is available on the acceptability of Korean MRLs(maximum residue limits) and the health risk based on the pesticide exposure by food intake. The aim of this study was to evaluate TMDI(theoretical maximum daily intake) and EDI(estimated daily intake) for Korean by using MRLs, food intake, residue data, and correction factors, and compare with ADI(acceptable daily intake) in order to estimate the health risk based on the pesticide exposure. The study was performed in three steps. In the frist step, the residual pesticides in each category of food were investigated using the pesticide residue analytical data(1995-96) from officially approved organizations and the analytical data for poultry was adopted from Korean food code method. In the second step, TMDI was estimated from MRLs and food factors, and was compared with ADI. In the third step, the effectiveness of each culinary treatment (washing, peeling, steaming, boiling, and salting) was evaluated and EDI was calculated using pesticide residue data, food factor, and correction factor by treatment. TMDI obtained from MRLs and food intake, and food intake was summed as 1,100.99 g, which was 79.1% of total consumption. The percent ratio of TMDI to ADI for 156 pesticides was mostly below 80% and only 30 pesticides exceeded the ADI. In particular, non-treated EDI from pesticide residue data and food intake was summed up to about 43 $\mu\textrm{g}$/day/capita, and the rank was procymidone(8.6 $\mu\textrm{g}$) > maleic hydrazide(8.2 $\mu\textrm{g}$) > EPN(3.7 $\mu\textrm{g}$) > deltamethrin(3.5 $\mu\textrm{g}$) > cypermethrin(3.0 $\mu\textrm{g}$). The treated EDI calculated from pesticide residue data, food intake, and correction factor by culinary treatment was summed up to 13.7 $\mu\textrm{g}$/day/captia. The percentage of ADI was TMDI(79.74%) > non-treated EDI (0.17%) > treated EDI (0.04%), and the exposure level of Korean population to whole pesticides was below the level to produce health risk. Oncogenic risk of five pesticides used in Korea whose oncogenic potency(Q*) was known were assessed from TMDI and treated EDI. Dietary oncogenic risk for Korean was estimated to be 2.0$\times$10-3 on the basis of TMDI, 8.3$\times$10-7 on the basis of treated EDI. The oncogenic risk from TMDI exceeded the risk level(1$\times$10-6) of EPA, whereas the oncogenic risk from treated EDI and real exposure level lower than that of EPA.