• Annals of Clinical Neurophysiology
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• v.19 no.1
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• pp.50-53
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• 2017

An infection is less likely to elicit chronic inflammatory demyelinating polyneuropathy (CIDP) than Guillain-$Barr{\acute{e}}$ syndrome. We here report a case of acute-onset CIDP following hepatitis A virus infection and briefly comment on the potential mechanisms regarding the induction and chronicity of autoimmunity after a viral infection.

• 대한두경부종양학회:학술대회논문집
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• 2002.11a
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• pp.254-254
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• 2002

• 대한핵의학회:학술대회논문집
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• 2001.11a
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• pp.44.1-44.1
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• 2001

• 대한세포병리학회:학술대회논문집
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• 2008.05a
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• pp.13-13
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• 2008

• 대한예방의학회:학술대회논문집
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• 1998.10b
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• pp.189-191
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• 1998

• 대한예방의학회:학술대회논문집
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• 2000.10a
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• pp.11-11
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• 2000

• 대한예방의학회:학술대회논문집
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• 1997.10b
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• pp.199-201
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• 1997

• Annals of Laboratory Medicine
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• v.38 no.6
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• pp.613-615
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• 2018

• Proceedings of the Korean Nutrition Society Conference
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• 2002.05a
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• pp.102-102
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• 2002

• Proceedings of the Korean Biophysical Society Conference
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• 2003.06a
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• pp.31-31
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• 2003

Cardiac atrium is now well-known as an endocrine organ which secretes atrial natriuretic peptide (AMP), participating in the regulation of body fluid and blood pressure. ANP is released mainly from cardiac muscle cells in response to various physiological and pathological conditions to induce atrial stretch. Ca$\^$2+/ may be one of the most important factors affecting ANP secretion even though controversy still persists. The aim of the present study is to investigate the effect of lysophosphatidylcholines (LPCs) and moxonidine on atrial hemodynamics and ANP secretion in hypertrophied atria. LPC is an endogenous phospholipid released from cell membrane during ischemia, and moxonidine is a imidazoline 1 (Il) receptor agonoist.